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Paper · 41594656

BDH1 Mediates Aerobic Exercise-Induced Improvement in Skeletal Muscle Metabolic Remodeling in Type 2 Diabetes Mellitus.

Wu M and 6 others. Biomolecules, 2026. Cited by 1 paper, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. BDH1 Mediates Aerobic Exercise-Induced Improvement in Skeletal Muscle Metabolic Remodeling in Type 2 Diabetes Mellitus. Papers, record 41594656. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/41594656

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
BDH1 Mediates Aerobic Exercise-Induced Improvement in Skeletal Muscle Metabolic Remodeling in Type 2 Diabetes Mellitus.
PubMed
Journal
Biomolecules
PubMed
Year
2026
PubMed
Publication types
Journal Article
PubMed
Citations, by iCite
1
iCite
Revised in PubMed
2026-07-01
PubMed

Authors7

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Wu MMingyu
2Ma XXiaotong
3Dai WWei
4Li KKe
5Gao HHaoyang
6Guo YYifan
7Xiao WWeihua

Source PubMed

Topics12

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Muscle, SkeletalMajor topic
Diabetes Mellitus, Type 2Major topic
Physical Conditioning, AnimalMajor topic
Hydroxybutyrate DehydrogenaseMajor topic
Diabetes Mellitus, ExperimentalMajor topic
Animals
Male
Mice
Mice, Knockout
Mice, Inbred C57BL
Oxidative Stress
Energy Metabolism

Source PubMed

Grants5

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes itAgencyCountryNIH projectStated by
32371185National Natural Science Foundation of ChinaPubMed
23010504200Shanghai Science and Technology Plan ProjectPubMed
2025KF002Key Lab of Exercise and Health Sciences of Ministry of Education (Shanghai University of Sport)PubMed
NoneShanghai Oriental Talents Program (Youth Project)PubMed
NO. 11DZ2261100Shanghai Key Lab of Human Performance (Shanghai University of Sport)PubMed

Source PubMed

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References67

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
137,164,310Mitochondria-SR interaction and mitochondrial fusion/fission in the regulation of skeletal muscle metabolism.Metabolism2023https://conductscience.org/library/papers/37164310PubMed
235,712,028The Effect of Aerobic Exercise on the Oxidative Capacity of Skeletal Muscle Mitochondria in Mice with Impaired Glucose Tolerance.J Diabetes Res2022https://conductscience.org/library/papers/35712028PubMed
333,826,918The many actions of insulin in skeletal muscle, the paramount tissue determining glycemia.Cell Metab2021https://conductscience.org/library/papers/33826918PubMed
438,540,669HIIT Promotes M2 Macrophage Polarization and Sympathetic Nerve Density to Induce Adipose Tissue Browning in T2DM Mice.Biomolecules2024https://conductscience.org/library/papers/38540669PubMed
532,922,365High-Intensity Interval Training Restores Glycolipid Metabolism and Mitochondrial Function in Skeletal Muscle of Mice With Type 2 Diabetes.Front Endocrinol (Lausanne)2020https://conductscience.org/library/papers/32922365PubMed
640,070,461Aerobic exercise improves inflammation and insulin resistance in skeletal muscle by regulating miR-221-3p via JAK/STAT signaling pathway.Front Physiol2025https://conductscience.org/library/papers/40070461PubMed
739,715,985
Aerobic exercise timing affects mitochondrial dynamics and insulin resistance by regulating the circadian clock protein expression and NAD+-SIRT1-PPARα-MFN2 pathway in the skeletal muscle of high-fat-diet-induced diabetes mice.
J Physiol Biochem2025https://conductscience.org/library/papers/39715985PubMed
835,851,220The Role of Mesenchymal Stem Cells in Regulating PDGF and VEGF during Pancreatic Islet Cells Regeneration in Diabetic Animal Model.Folia Med (Plovdiv)2021https://conductscience.org/library/papers/35851220PubMed
936,545,931Structural functionality of skeletal muscle mitochondria and its correlation with metabolic diseases.Clin Sci (Lond)2022https://conductscience.org/library/papers/36545931PubMed
1041,008,530Beneficial Effects of Different Types of Exercise on Diabetic Cardiomyopathy.Biomolecules2025https://conductscience.org/library/papers/41008530PubMed

Cited by1

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 1 citation.

PMIDTitleJournalYearIn Papers
42,927,614BDH1 Attenuates Macrophage Foam Cell Formation and Inflammation in Atherosclerosis Via Modulating Histone H3K9 Acylation Balance and Activating PPAR Signaling.J Biochem Mol Toxicol2026https://conductscience.org/library/papers/42927614

Source PubMed

Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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