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Paper · 41206016

Glucagon, the Alpha Cell, and Potential Targets for Diabetes Treatment.

Dhanvantari S and 1 other. Endocrinology, 2025. Cited by 2 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Glucagon, the Alpha Cell, and Potential Targets for Diabetes Treatment. Papers, record 41206016. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/41206016

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Glucagon, the Alpha Cell, and Potential Targets for Diabetes Treatment.
PubMed
Journal
Endocrinology
PubMed
Year
2025
PubMed
Publication types
Journal Article, Review
PubMed
Citations, by iCite
2
iCite
Revised in PubMed
2026-11-06
PubMed

Authors2

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Dhanvantari SSavitahttps://orcid.org/0000-0001-6453-1386
2Dean EDE Daniellehttps://orcid.org/0000-0002-7157-5683

Source PubMed

Topics6

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
GlucagonMajor topic
Glucagon-Secreting CellsMajor topic
Diabetes MellitusMajor topic
Humans
Animals
Signal Transduction

Source PubMed

Grants4

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes itAgencyCountryNIH projectStated bySource
P30 DK020593NIDDK NIH HHSUnited StatesP30DK020593PubMedNIH RePORTER
R01 DK132669NIDDK NIH HHSUnited StatesR01DK132669PubMedNIH RePORTER
Natural Sciences and Engineering Research Council of CanadaPubMedPubMed
R01 DK132669aNIDDK NIH HHSUnited StatesR01DK132669PubMedNIH RePORTER

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References118

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
112,388,744Identification of a chromogranin A domain that mediates binding to secretogranin III and targeting to secretory granules in pituitary cells and pancreatic beta-cells.Mol Biol Cell2002https://conductscience.org/library/papers/12388744PubMed
230,713,523Plasticity in the Glucagon Interactome Reveals Novel Proteins That Regulate Glucagon Secretion in α-TC1-6 Cells.Front Endocrinol (Lausanne)2018https://conductscience.org/library/papers/30713523PubMed
338,124,716Single-molecule analysis of intracellular insulin granule behavior and its application to analyzing cytoskeletal dependence and pathophysiological implications.Front Physiol2023https://conductscience.org/library/papers/38124716PubMed
415,994,347Glucose dependence of the regulated secretory pathway in alphaTC1-6 cells.Endocrinology2005https://conductscience.org/library/papers/15994347PubMed
518,549,247The trans-Golgi proteins SCLIP and SCG10 interact with chromogranin A to regulate neuroendocrine secretion.Biochemistry2008https://conductscience.org/library/papers/18549247PubMed
630,187,652GLP-1 suppresses glucagon secretion in human pancreatic alpha-cells by inhibition of P/Q-type Ca2+ channels.Physiol Rep2018https://conductscience.org/library/papers/30187652PubMed
714,630,721Impaired intestinal proglucagon processing in mice lacking prohormone convertase 1.Endocrinology2004https://conductscience.org/library/papers/14630721PubMed
817,718,510Biogenesis of the secretory granule: chromogranin A coiled-coil structure results in unusual physical properties and suggests a mechanism for granule core condensation.Biochemistry2007https://conductscience.org/library/papers/17718510PubMed
933,239,450Glucagon Resistance and Decreased Susceptibility to Diabetes in a Model of Chronic Hyperglucagonemia.Diabetes2021https://conductscience.org/library/papers/33239450PubMed
108,721,980Role of prohormone convertases in the tissue-specific processing of proglucagon.Mol Endocrinol1996https://conductscience.org/library/papers/8721980PubMed

1 to 10 of 118

See all 118 references

Cited by1

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 2 citations.

PMIDTitleJournalYearIn Papers
42,625,306Stathmin-2 mediates paracrine hormone regulation of glucagon through lysosomal trafficking in αTC1-6 cells.J Physiol2026https://conductscience.org/library/papers/42625306

Source PubMed

Genes1

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

GeneNCBI Gene IDLinked byIn Genes
GCG2,641PubTator's text mininghttps://conductscience.org/library/genes/2641

Source NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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The record and each of its lists are JSON, twenty rows a page. No key.

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