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Paper · 4130108

Mechanisms of passive sensitization. IV. Dissociation of IgE molecules from basophil receptors at acid pH.

Ishizaka T and 1 other. J Immunol, 1974. Cited by 125 papers, by iCite.

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ConductScience Library. Mechanisms of passive sensitization. IV. Dissociation of IgE molecules from basophil receptors at acid pH. Papers, record 4130108. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/4130108

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Mechanisms of passive sensitization. IV. Dissociation of IgE molecules from basophil receptors at acid pH.
PubMed
Journal
J Immunol
PubMed
Year
1974
PubMed
Publication types
Journal Article
PubMed
Citations, by iCite
125
iCite
Relative citation ratio, by iCite
3.47
iCite
Revised in PubMed
2011-11-17
PubMed

Authors2

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Ishizaka TT
2Ishizaka KK

Source PubMed

Topics20

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
BasophilsMajor topic
Binding Sites, AntibodyMajor topic
Immunity, Maternally-AcquiredMajor topic
Immunoglobulin EMajor topic
Animals
Autoradiography
Buffers
Chromatography, DEAE-Cellulose
Histamine Release
Humans
Hydrogen-Ion Concentration
Immune Sera
Immunoglobulin Fab Fragments
Immunoglobulin Fc Fragments
Immunologic Techniques
Iodine Radioisotopes
Myeloma Proteins
Papain
Rabbits
Staining and Labeling

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References0

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

None in this release.

Cited by29

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 125 citations.

PMIDTitleJournalYearIn PapersSource
42,587,790Frequency of Local Allergic Asthma in Bronchial Asthma Patients: Role of Conjunctival Challenge and Neutrophil Histamine Release.Cells2026https://conductscience.org/library/papers/42587790PubMed
36,385,515Progenitor cell-derived basophils: A novel barcoded passive degranulation assay in allergic diseases.Clin Exp Allergy2023https://conductscience.org/library/papers/36385515PubMed
32,695,309AllergoOncology: ultra-low IgE, a potential novel biomarker in cancer-a Position Paper of the European Academy of Allergy and Clinical Immunology (EAACI).Clin Transl Allergy2020https://conductscience.org/library/papers/32695309PubMed
31,941,161Acid Stripping of Surface IgE Antibodies Bound to FcεRI is Unsuitable for the Functional Assays that Require Long-Term Culture of Basophils and Entire Removal of Surface IgE.Int J Mol Sci2020https://conductscience.org/library/papers/31941161PubMed
29,375,554Expression of Membrane-Bound CC Chemokine Ligand 20 on Follicular T Helper Cells in T-B-Cell Conjugates.Front Immunol2017https://conductscience.org/library/papers/29375554PubMed
26,945,493The discovery of IgE 50 years later.Ann Allergy Asthma Immunol2016https://conductscience.org/library/papers/26945493PubMed
25,490,087Detection of true IgE-expressing mouse B lineage cells.J Vis Exp2014https://conductscience.org/library/papers/25490087PubMed
24,632,082Regulatory constraints in the generation and differentiation of IgE-expressing B cells.Curr Opin Immunol2014https://conductscience.org/library/papers/24632082PubMed
24,194,423Granulocytes (the red, white, and blue) in hypersensitivity reactions: A review.Inflammation1976https://conductscience.org/library/papers/24194423PubMed
11,876,750IgE is expressed on, but not produced by, fetal cells in the human placenta irrespective of maternal atopy.Clin Exp Immunol2002https://conductscience.org/library/papers/11876750PubMed

1 to 10 of 29

See all 29 cited by

Genes1

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

GeneNCBI Gene IDLinked byIn Genes
IGHE3,497PubTator's text mininghttps://conductscience.org/library/genes/3497

Source NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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