Paper · 42112339

WSTF-associated regulation of GLYCTK and metabolic adaptation in colorectal cancer.

Zhou L and 8 others. Front Immunol, 2026. Cited by 0 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. WSTF-associated regulation of GLYCTK and metabolic adaptation in colorectal cancer. Papers, record 42112339. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/42112339

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: WSTF-associated regulation of GLYCTK and metabolic adaptation in colorectal cancer.; PubMed
Journal: Front Immunol; PubMed
Year: 2026; PubMed
Publication types: Journal Article; PubMed
PubMed: https://pubmed.ncbi.nlm.nih.gov/42112339/; PubMed
DOI: https://doi.org/10.3389/fimmu.2026.1819148; PubMed
PMC: https://pmc.ncbi.nlm.nih.gov/articles/PMC13150450/; PubMed
The journal: https://conductscience.org/library/journal-index/101560960; PubMed
Citations, by iCite: 0; iCite
Revised in PubMed: 2026-08-13; PubMed

Authors9

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename
1	Zhou L	Liming
2	Liu Y	Yan
3	Li Y	Yufeng
4	Zheng S	Shasha
5	Guo J	Jimin
6	Chen S	Shuai
7	Yu Y	Yuan
8	Fang W	Wei
9	Li J	Jingwu

Source PubMed

Topics7

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
Colorectal Neoplasms	Major topic
Gene Expression Regulation, Neoplastic	Major topic
Transcription Factors	Major topic
Humans
Metabolic Reprogramming
Cell Line, Tumor
Tumor Microenvironment

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References37

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

Order	PMID	Title	Journal	Year	In Papers	Source
1	9,828,126	A novel human gene, WSTF, is deleted in Williams syndrome.	Genomics	1998	https://conductscience.org/library/papers/9828126	PubMed
2	32,518,374	WSTF acetylation by MOF promotes WSTF activities and oncogenic functions.	Oncogene	2020	https://conductscience.org/library/papers/32518374	PubMed
3	28,610,873	Williams syndrome transcription factor (WSTF) acts as an activator of estrogen receptor signaling in breast cancer cells and the effect can be abrogated by 1α,25-dihydroxyvitamin D3.	J Steroid Biochem Mol Biol	2018	https://conductscience.org/library/papers/28610873	PubMed
4	16,603,771	The WSTF-SNF2h chromatin remodeling complex interacts with several nuclear proteins in transcription.	J Biol Chem	2006	https://conductscience.org/library/papers/16603771	PubMed
5	36,653,562	FinnGen provides genetic insights from a well-phenotyped isolated population.	Nature	2023	https://conductscience.org/library/papers/36653562	PubMed
6	34,140,529	Williams syndrome.	Nat Rev Dis Primers	2021	https://conductscience.org/library/papers/34140529	PubMed
7	21,559,432	The chromatin remodelling complex B-WICH changes the chromatin structure and recruits histone acetyl-transferases to active rRNA genes.	PLoS One	2011	https://conductscience.org/library/papers/21559432	PubMed
8	19,092,802	WSTF regulates the H2A.X DNA damage response via a novel tyrosine kinase activity.	Nature	2009	https://conductscience.org/library/papers/19092802	PubMed
9	34,475,573	Large-scale cis- and trans-eQTL analyses identify thousands of genetic loci and polygenic scores that regulate blood gene expression.	Nat Genet	2021	https://conductscience.org/library/papers/34475573	PubMed
10	19,234,442	Tyrosine dephosphorylation of H2AX modulates apoptosis and survival decisions.	Nature	2009	https://conductscience.org/library/papers/19234442	PubMed

1 to 10 of 37

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Cited by0

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 0 citations.

None in this release.

Genes3

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

Gene	NCBI Gene ID	Linked by	In Genes	Source
BAZ1B	9,031	PubTator's text mining	https://conductscience.org/library/genes/9031	NCBI Gene
GLYCTK	132,158	PubTator's text mining	https://conductscience.org/library/genes/132158	NCBI Gene
MYC	4,609	PubTator's text mining	https://conductscience.org/library/genes/4609	NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

Sources 11

ChEMBL, through Open TargetsCC BY-SA 3.0
ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

Each source's terms

ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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