Paper · 9828126

A novel human gene, WSTF, is deleted in Williams syndrome.

Lu X and 3 others. Genomics, 1998. Cited by 94 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. A novel human gene, WSTF, is deleted in Williams syndrome. Papers, record 9828126. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/9828126

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: A novel human gene, WSTF, is deleted in Williams syndrome.; PubMed
Journal: Genomics; PubMed
Year: 1998; PubMed
Publication types: Journal Article, Research Support, Non-U.S. Gov't, Research Support, U.S. Gov't, P.H.S.; PubMed
PubMed: https://pubmed.ncbi.nlm.nih.gov/9828126/; PubMed
DOI: https://doi.org/10.1006/geno.1998.5578; PubMed
The journal: https://conductscience.org/library/journal-index/8800135; PubMed
Citations, by iCite: 94; iCite
Relative citation ratio, by iCite: 1.48; iCite
Revised in PubMed: 2022-03-09; PubMed

Authors4

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename
1	Lu X	X
2	Meng X	X
3	Morris CA	C A
4	Keating MT	M T

Source PubMed

Topics15

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
Gene Deletion	Major topic
Williams Syndrome	Major topic
Amino Acid Sequence
Base Sequence
Chromosomes, Human, Pair 7
Cloning, Molecular
DNA Primers
Exons
Humans
In Situ Hybridization, Fluorescence
Introns
Molecular Sequence Data
RNA, Messenger
Sequence Alignment
Sequence Analysis, DNA

Source PubMed

Grants4

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes it	Agency	Country	NIH project	Stated by	Source
MO1-RR0064	NCRR NIH HHS	United States	M01RR000064	PubMed and NIH	NIH RePORTER
R01HD29957	NICHD NIH HHS	United States	R01HD029957	PubMed and NIH	NIH RePORTER
R01NS35102-01	NINDS NIH HHS	United States	R01NS351020	PubMed	NIH RePORTER
			R01NS035102	NIH	NIH RePORTER

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References0

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

None in this release.

Cited by43

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 94 citations.

PMID	Title	Journal	Year	In Papers	Source
42,258,541	WSTF deficiency reprograms regulatory networks by linking locus-specific chromatin remodeling to altered isoform expression and misdirected signaling.	Nucleic Acids Res	2026	https://conductscience.org/library/papers/42258541	PubMed
42,112,339	WSTF-associated regulation of GLYCTK and metabolic adaptation in colorectal cancer.	Front Immunol	2026	https://conductscience.org/library/papers/42112339	PubMed
39,212,120	Two paralogous PHD finger proteins participate in natural genome editing in Paramecium tetraurelia.	J Cell Sci	2024	https://conductscience.org/library/papers/39212120	PubMed
38,884,720	Epigenetics.	Adv Exp Med Biol	2024	https://conductscience.org/library/papers/38884720	PubMed
37,964,651	Epigenetic regulation of craniofacial development and disease.	Birth Defects Res	2024	https://conductscience.org/library/papers/37964651	PubMed
37,588,924	Evolution and dysfunction of human cognitive and social traits: A transcriptional regulation perspective.	Evol Hum Sci	2022	https://conductscience.org/library/papers/37588924	PubMed
36,582,821	baz1b loss-of-function in zebrafish produces phenotypic alterations consistent with the domestication syndrome.	iScience	2023	https://conductscience.org/library/papers/36582821	PubMed
34,736,517	The emerging role of ISWI chromatin remodeling complexes in cancer.	J Exp Clin Cancer Res	2021	https://conductscience.org/library/papers/34736517	PubMed
34,680,936	BAZ1B the Protean Protein.	Genes (Basel)	2021	https://conductscience.org/library/papers/34680936	PubMed
32,849,845	Neurodevelopmental Disorders Caused by Defective Chromatin Remodeling: Phenotypic Complexity Is Highlighted by a Review of ATRX Function.	Front Genet	2020	https://conductscience.org/library/papers/32849845	PubMed

1 to 10 of 43

See all 43 cited by

Genes1

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

Gene	NCBI Gene ID	Linked by	In Genes
BAZ1B	9,031	Swiss-Prot's references	https://conductscience.org/library/genes/9031

Source NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data1

The data bank entries PubMed's record names. Trial numbers are under Trials.

Data bank	Accession
GENBANK	AF072810

Source GENBANK

Data dates

The date of each source this release read.

PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

Sources 11

ChEMBL, through Open TargetsCC BY-SA 3.0
ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

Each source's terms

ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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