Paper · 42872022
Divergent Hypoxia-Inducible Factor 2‑Alpha Signaling in Alcoholic Liver Disease and Metabolic Dysfunction-Associated Steatotic Liver Disease.
Santra A and 6 others. ACS Pharmacol Transl Sci, 2026.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. Divergent Hypoxia-Inducible Factor 2‑Alpha Signaling in Alcoholic Liver Disease and Metabolic Dysfunction-Associated Steatotic Liver Disease. Papers, record 42872022. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/42872022
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- Divergent Hypoxia-Inducible Factor 2‑Alpha Signaling in Alcoholic Liver Disease and Metabolic Dysfunction-Associated Steatotic Liver Disease.
- Journal
- ACS Pharmacol Transl Sci
- Year
- 2026
- Publication types
- Journal Article, Review
- Revised in PubMed
- 2026-10-15
Source PubMed
Topics0
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Trials0
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Comments and corrections0
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References75
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 38,548,246 | SIRT3 and SIRT4 double-genes remodeled the mitochondrial network to induce hepatocellular carcinoma cell line differentiation and suppress malignant phenotypes. | Biochem Pharmacol | 2024 | https://conductscience.org/library/papers/38548246 | PubMed |
| 2 | 28,891,317 | The Role of Sirtuins in Antioxidant and Redox Signaling. | Antioxid Redox Signal | 2018 | https://conductscience.org/library/papers/28891317 | PubMed |
| 3 | 37,398,564 | The Wnt/β-catenin signaling pathway plays a role in drug-induced liver injury by regulating cytochrome P450 2E1 expression. | Toxicol Res | 2023 | https://conductscience.org/library/papers/37398564 | PubMed |
| 4 | 34,692,739 | Hypoxia-Inducible Factors as Key Players in the Pathogenesis of Non-alcoholic Fatty Liver Disease and Non-alcoholic Steatohepatitis. | Front Med (Lausanne) | 2021 | https://conductscience.org/library/papers/34692739 | PubMed |
| 5 | 39,462,179 | Metabolic Reprogramming in Cancer: Implications for Immunosuppressive Microenvironment. | Immunology | 2025 | https://conductscience.org/library/papers/39462179 | PubMed |
| 6 | 24,895,555 | The hypoxia-inducible factor pathway, prolyl hydroxylase domain protein inhibitors, and their roles in bone repair and regeneration. | Biomed Res Int | 2014 | https://conductscience.org/library/papers/24895555 | PubMed |
| 7 | 18,259,202 | Role and regulation of prolyl hydroxylase domain proteins. | Cell Death Differ | 2008 | https://conductscience.org/library/papers/18259202 | PubMed |
| 8 | 30,761,299 | Hypoxia-Modified Cancer Cell Metabolism. | Front Cell Dev Biol | 2019 | https://conductscience.org/library/papers/30761299 | PubMed |
| 9 | 40,130,250 | Molecular regulators of alcoholic liver disease: a comprehensive analysis of microRNAs and long non-coding RNAs. | Front Med (Lausanne) | 2025 | https://conductscience.org/library/papers/40130250 | PubMed |
| 10 | 34,956,204 | The Gut-Liver Axis in Health and Disease: The Role of Gut Microbiota-Derived Signals in Liver Injury and Regeneration. | Front Immunol | 2021 | https://conductscience.org/library/papers/34956204 | PubMed |
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Drugs0
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Data0
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Data dates
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- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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