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Paper · 42876144

A Hypertrophic Stimulus Drives Age-Dependent Transcript Isoform Switching With and Without Differential Gene Expression in Skeletal Muscle.

Koopmans PJ and 5 others. Aging Cell, 2026.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. A Hypertrophic Stimulus Drives Age-Dependent Transcript Isoform Switching With and Without Differential Gene Expression in Skeletal Muscle. Papers, record 42876144. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/42876144

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
A Hypertrophic Stimulus Drives Age-Dependent Transcript Isoform Switching With and Without Differential Gene Expression in Skeletal Muscle.
Journal
Aging Cell
Year
2026
Publication types
Journal Article
Revised in PubMed
2026-10-29

Source PubMed

Authors6

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Koopmans PJPieter Jhttps://orcid.org/0000-0002-4797-5265
2Stokes ALAbbey L
3Nelson CEChristopher Ehttps://orcid.org/0000-0002-8423-8854
4Wen YYuanhttps://orcid.org/0000-0002-3210-1629
5Ismaeel AAhmedhttps://orcid.org/0000-0003-4025-3945
6Murach KAKevin Ahttps://orcid.org/0000-0003-2783-7137

Source PubMed

Topics7

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Muscle, SkeletalMajor topic
AgingMajor topic
Gene Expression RegulationMajor topic
Animals
Mice
Protein Isoforms
Hypertrophy

Source PubMed

Grants8

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes itAgencyCountryNIH projectStated bySource
AR081367NIH HHSUnited StatesPubMedPubMed
AG088465NIH HHSUnited StatesPubMedPubMed
K22 AG093185NIA NIH HHSUnited StatesK22AG093185PubMedNIH RePORTER
American Federation for Aging ResearchPubMedPubMed
GM156679NIH HHSUnited StatesPubMedPubMed
AG093185NIH HHSUnited StatesPubMedPubMed
GM155433NIH HHSUnited StatesPubMedPubMed
AG063944NIH HHSUnited StatesPubMedPubMed

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References30

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
137,118,371The energy-splicing resilience axis hypothesis of aging.Nat Aging2022https://conductscience.org/library/papers/37118371PubMed
216,996,505Muscle atrophy in transgenic mice expressing a human TSC1 transgene.FEBS Lett2006https://conductscience.org/library/papers/16996505PubMed
340,595,512Aerobic and resistance exercise-regulated phosphoproteome and acetylproteome modifications in human skeletal muscle.Nat Commun2025https://conductscience.org/library/papers/40595512PubMed
430,115,379Runx1 promotes satellite cell proliferation during ischemia - Induced muscle regeneration.Biochem Biophys Res Commun2018https://conductscience.org/library/papers/30115379PubMed
530,989,184IsoformSwitchAnalyzeR: analysis of changes in genome-wide patterns of alternative splicing and its functional consequences.Bioinformatics2019https://conductscience.org/library/papers/30989184PubMed
628,703,423The emerging role of alternative splicing in senescence and aging.Aging Cell2017https://conductscience.org/library/papers/28703423PubMed
732,188,845Full-length transcript characterization of SF3B1 mutation in chronic lymphocytic leukemia reveals downregulation of retained introns.Nat Commun2020https://conductscience.org/library/papers/32188845PubMed
842,324,502Transcriptomic responses to endurance exercise training in rats.BMC Genom Data2026https://conductscience.org/library/papers/42324502PubMed
941,772,759Loss of Splicing Homeostasis as a Hallmark of Aging.Mol Cell Biol2026https://conductscience.org/library/papers/41772759PubMed
1031,642,809Discovery proteomics in aging human skeletal muscle finds change in spliceosome, immunity, proteostasis and mitochondria.Elife2019https://conductscience.org/library/papers/31642809PubMed

1 to 10 of 30

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Cited by0

The papers in our copy of PubMed whose reference list names this one, newest first.

None in this release.

Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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