Paper · 42957749
Transcription factor EB as a novel target for dimethyl fumarate to restore endothelial function in diabetes.
He L and 9 others. Acta Pharmacol Sin, 2026.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. Transcription factor EB as a novel target for dimethyl fumarate to restore endothelial function in diabetes. Papers, record 42957749. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/42957749
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Paper
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- Title
- Transcription factor EB as a novel target for dimethyl fumarate to restore endothelial function in diabetes.
- Journal
- Acta Pharmacol Sin
- Year
- 2026
- Publication types
- Journal Article
- Revised in PubMed
- 2026-10-30
Source PubMed
Topics0
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Trials0
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Comments and corrections0
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References20
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 30,170,601 | Endothelial dysfunction and platelet hyperactivity in type 2 diabetes mellitus: molecular insights and therapeutic strategies. | Cardiovasc Diabetol | 2018 | https://conductscience.org/library/papers/30170601 | PubMed |
| 2 | 40,111,409 | Erythrocyte-derived extracellular vesicles induce endothelial dysfunction through arginase-1 and oxidative stress in type 2 diabetes. | J Clin Invest | 2025 | https://conductscience.org/library/papers/40111409 | PubMed |
| 3 | 39,241,746 | A systems view of the vascular endothelium in health and disease. | Cell | 2024 | https://conductscience.org/library/papers/39241746 | PubMed |
| 4 | 30,099,904 | [Diabetes mellitus: endothelial dysfunction and changes in hemostasis]. | Orv Hetil | 2018 | https://conductscience.org/library/papers/30099904 | PubMed |
| 5 | 40,588,351 | Vascular dysfunction in diabetes and pharmacotherapeutic opportunities: a focus on endothelial cell health. | Am J Physiol Heart Circ Physiol | 2025 | https://conductscience.org/library/papers/40588351 | PubMed |
| 6 | 31,300,673 | Therapeutic efficacy of dimethyl fumarate in relapsing-remitting multiple sclerosis associates with ROS pathway in monocytes. | Nat Commun | 2019 | https://conductscience.org/library/papers/31300673 | PubMed |
| 7 | 25,009,787 | Dimethylfumarate attenuates restenosis after acute vascular injury by cell-specific and Nrf2-dependent mechanisms. | Redox Biol | 2014 | https://conductscience.org/library/papers/25009787 | PubMed |
| 8 | 33,876,201 | Dimethyl fumarate induces ferroptosis and impairs NF-κB/STAT3 signaling in DLBCL. | Blood | 2021 | https://conductscience.org/library/papers/33876201 | PubMed |
| 9 | 31,645,538 | The Effects of Dimethyl Fumarate on Atherosclerosis in the Apolipoprotein E-Deficient Mouse Model with Streptozotocin-Induced Hyperglycemia Mediated By the Nuclear Factor Erythroid 2-Related Factor 2/Antioxidant Response Element (Nrf2/ARE) Signaling Pathway. | Med Sci Monit | 2019 | https://conductscience.org/library/papers/31645538 | PubMed |
| 10 | 33,921,821 | Noise-Induced Vascular Dysfunction, Oxidative Stress, and Inflammation Are Improved by Pharmacological Modulation of the NRF2/HO-1 Axis. | Antioxidants (Basel) | 2021 | https://conductscience.org/library/papers/33921821 | PubMed |
1 to 10 of 20
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Data0
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Data dates
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- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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