Paper · 42962029

Photosynthetic chimeric stem cells function as mitochondrial batteries.

Chen Z and 11 others. Sci Adv, 2026.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Photosynthetic chimeric stem cells function as mitochondrial batteries. Papers, record 42962029. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/42962029

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: Photosynthetic chimeric stem cells function as mitochondrial batteries.
Journal: Sci Adv
Year: 2026
Publication types: Journal Article
PubMed: https://pubmed.ncbi.nlm.nih.gov/42962029/
DOI: https://doi.org/10.1126/sciadv.aef8069
PMC: https://pmc.ncbi.nlm.nih.gov/articles/PMC13721958/
The journal: https://conductscience.org/library/journal-index/101653440
Revised in PubMed: 2026-11-01

Source PubMed

Authors12

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename	ORCID iD
1	Chen Z	Zhengtai	https://orcid.org/0009-0009-3357-1524
2	Sun H	Hanxiao	https://orcid.org/0009-0001-9355-0380
3	Zheng Y	Yuanyang	https://orcid.org/0009-0003-6916-5444
4	Jiang X	Xuan	https://orcid.org/0009-0007-4817-4682
5	He Y	Yan
6	Ye K	Kai	https://orcid.org/0009-0004-1500-0000
7	Zhang Y	Yufan	https://orcid.org/0000-0002-1014-5073
8	Mao Z	Zhengwei	https://orcid.org/0000-0001-7990-2856
9	Zhou Z	Ziyi	https://orcid.org/0000-0002-8267-8178
10	Lin X	Xiaoxi	https://orcid.org/0000-0002-7325-1367

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Topics15

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
Mitochondria	Major topic
Photosynthesis	Major topic
Mesenchymal Stem Cells	Major topic
Chimera	Major topic
Stem Cells	Major topic
Animals
Mice
Fibroblast Growth Factors
Cell Differentiation
Wound Healing
RNA, Messenger
Swine
Thylakoids
Humans
Energy Metabolism

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References38

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

Order	PMID	Title	Journal	Year	In Papers	Source
1	31,269,442	Nitric Oxide Modulates Metabolic Remodeling in Inflammatory Macrophages through TCA Cycle Regulation and Itaconate Accumulation.	Cell Rep	2019	https://conductscience.org/library/papers/31269442	PubMed
2	32,879,300	FGF/FGFR signaling in health and disease.	Signal Transduct Target Ther	2020	https://conductscience.org/library/papers/32879300	PubMed
3	20,616,029	Fibroblast growth factor 21 regulates energy metabolism by activating the AMPK-SIRT1-PGC-1alpha pathway.	Proc Natl Acad Sci U S A	2010	https://conductscience.org/library/papers/20616029	PubMed
4	41,134,634	Nanomaterial-induced mitochondrial biogenesis enhances intercellular mitochondrial transfer efficiency.	Proc Natl Acad Sci U S A	2025	https://conductscience.org/library/papers/41134634	PubMed
5	41,232,872	Polysaccharide hydrogel loaded with dual-drug liposomes for accelerating diabetic wound healing through mitochondrial protection and anti-inflammatory activity.	Int J Biol Macromol	2025	https://conductscience.org/library/papers/41232872	PubMed
6	25,999,420	Regulated necrotic cell death: the passive aggressive side of Bax and Bak.	Circ Res	2015	https://conductscience.org/library/papers/25999420	PubMed
7	40,383,752	Baicalein based nano-delivery system restores mitochondrial homeostasis through PPAR signaling pathway to promote wound healing in diabetes.	J Nanobiotechnology	2025	https://conductscience.org/library/papers/40383752	PubMed
8	39,546,755	Mitophagy-Enhanced Nanoparticle-Engineered Mitochondria Restore Homeostasis of Mitochondrial Pool for Alleviating Pulmonary Fibrosis.	ACS Nano	2024	https://conductscience.org/library/papers/39546755	PubMed
9	41,344,083	Mitochondria in pyroptosis: Mechanisms and implications.	Mol Immunol	2026	https://conductscience.org/library/papers/41344083	PubMed
10	41,346,305	NADH-Reductive Stress Induced by Dihydrolipoamide Dehydrogenase Activation Contributes to Cuproptosis.	Adv Sci (Weinh)	2026	https://conductscience.org/library/papers/41346305	PubMed

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Cited by0

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None in this release.

Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

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None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

Sources 11

ChEMBL, through Open TargetsCC BY-SA 3.0
ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

Each source's terms

ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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