Paper46252
Suppression of reaginic antibody formation. I. Induction of hapten-specific tolerance.
Lee WY and 1 other. J Immunol, 1975. Cited by 81 papers, by iCite.
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ConductScience Library. Suppression of reaginic antibody formation. I. Induction of hapten-specific tolerance. Papers, record 46252. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/46252
Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
Topics16
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Antibodies | Major topic |
| Antibody Formation | Major topic |
| Haptens | Major topic |
| Immune Tolerance | Major topic |
| Reagins | Major topic |
| Animals | |
| Hemagglutinins | |
| Immunization | |
| Immunization, Secondary | |
| Immunosuppression Therapy | |
| Mice | |
| Nitrobenzenes | |
| Ovalbumin | |
| Passive Cutaneous Anaphylaxis | |
| Time Factors | |
| gamma-Globulins |
Source PubMed
Cited by10
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 81 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 7,275,187 | Adjuvant and suppressive effects of Grass Conjuvac and other alginate conjugates on IgG and IgE antibody responses in mice. | Immunology | 1981 | https://conductscience.org/library/papers/7275187 | PubMed |
| 7,084,424 | Specific immunosuppression of IgE response to hapten DNP by linked to monoclonal IgG1 in rats. | Experientia | 1982 | https://conductscience.org/library/papers/7084424 | PubMed |
| 6,706,377 | Suppression of the anti-trimellityl (TM) IgE response in mice by conjugates of TM with polyvinyl alcohol. | Immunology | 1984 | https://conductscience.org/library/papers/6706377 | PubMed |
| 6,436,172 | Opsonic, cytotoxic, precipitating, blocking of bacterial adherence, and other activities of monoclonal IgE antibody compared with IgA and IgM. | Immunology | 1984 | https://conductscience.org/library/papers/6436172 | PubMed |
| 6,206,698 | Desensitization of rat mast cells: studies of some effects of anti-allergic drugs. | Agents Actions | 1984 | https://conductscience.org/library/papers/6206698 | PubMed |
| 2,265,481 | Can persistent IgE responses be suppressed? | Clin Exp Immunol | 1990 | https://conductscience.org/library/papers/2265481 | PubMed |
| 955,678 | Inhibitory effect of carrier and hapten preimmunization on delayed hypersensitivity to the carrier. | Immunology | 1976 | https://conductscience.org/library/papers/955678 | PubMed |
| 562,920 | Regulation of reaginic antibody production in mice. I. Suppression by antigen of IgE antibody production in vitro. | J Exp Med | 1977 | https://conductscience.org/library/papers/562920 | PubMed |
| 86,508 | Hapten specific unresponsiveness in mice. I. Characteristics of unresponsiveness induced by 2,4,6-trinitrobenzene sulphonic acid. | Immunology | 1979 | https://conductscience.org/library/papers/86508 | PubMed |
| 77,751 | The use of hapten-polysaccharide conjugates for the induction of B-cell tolerance involving IgE responses. I. Preparation, characterization and specific tolerogenic activity of penicilloy l-substituted levans affecting IgE responses in normal and sensitized mice. | Clin Exp Immunol | 1978 | https://conductscience.org/library/papers/77751 | PubMed |
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- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
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Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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