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Paper51405

Association of amyloidosis with erythema nodosum leprosum reactions and recurrent neutrophil leucocytosis in leprosy.

McAdam KP and 4 others. Lancet, 1975. Cited by 73 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Association of amyloidosis with erythema nodosum leprosum reactions and recurrent neutrophil leucocytosis in leprosy. Papers, record 51405. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/51405

Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Association of amyloidosis with erythema nodosum leprosum reactions and recurrent neutrophil leucocytosis in leprosy.
PubMed
Journal
Lancet
PubMed
Year
1975
PubMed
Publication types
Journal Article
PubMed
Citations, by iCite
73
iCite
Relative citation ratio, by iCite
2.79
iCite
Revised in PubMed
2019-06-10
PubMed

Authors5

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1McAdam KPK P
2Anders RFR F
3Smith SRS R
4Russell DAD A
5Price MAM A

Source PubMed

Topics9

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
AmyloidosisMajor topic
Erythema NodosumMajor topic
LeprosyMajor topic
LeukocytosisMajor topic
NeutrophilsMajor topic
Blood Proteins
Humans
Leukocyte Count
Recurrence

Source PubMed

Cited by18

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 73 citations.

PMIDTitleJournalYearIn PapersSource
38,440,733Leprosy: treatment, prevention, immune response and gene function.Front Immunol2024https://conductscience.org/library/papers/38440733PubMed
25,651,321Leprosy nephropathy: a review of clinical and histopathological features.Rev Inst Med Trop Sao Paulo2015https://conductscience.org/library/papers/25651321PubMed
16,532,294Oculo-renal disorders in infectious diseases.Int Ophthalmol2004https://conductscience.org/library/papers/16532294PubMed
15,829,582Protein fibrils in nature can enhance amyloid protein A amyloidosis in mice: Cross-seeding as a disease mechanism.Proc Natl Acad Sci U S A2005https://conductscience.org/library/papers/15829582PubMed
7,276,825Studies of endotoxin-induced decrease in lipoprotein lipase activity.J Exp Med1981https://conductscience.org/library/papers/7276825PubMed
6,774,048Heterogeneity of human serum amyloid A proteins.J Exp Med1980https://conductscience.org/library/papers/6774048PubMed
6,647,188Renal lesions in leprosy amongst north Indian patients.Postgrad Med J1983https://conductscience.org/library/papers/6647188PubMed
3,140,793Effects of dimethyl sulphoxide on the synthesis of plasma proteins in the human hepatoma HepG2. Induction of an acute-phase-like reaction.Biochem J1988https://conductscience.org/library/papers/3140793PubMed
1,715,262Current concepts in the pathogenesis of leprosy. Clinical, pathological, immunological and chemotherapeutic aspects.Drugs1991https://conductscience.org/library/papers/1715262PubMed
978,136Murine model for human secondary amyloidosis: genetic variability of the acute-phase serum protein SAA response to endotoxins and casein.J Exp Med1976https://conductscience.org/library/papers/978136PubMed
849,644Cell-mediated immunity in amyloidosis secondary to lepromatous leprosy.Clin Exp Immunol1977https://conductscience.org/library/papers/849644PubMed
765,426Amyloid-related serum protein SAA in endotoxin-induced amyloidosis of the mink.J Exp Med1976https://conductscience.org/library/papers/765426PubMed
678,446Induction of the acute-phase serum protein SAA requires both RNA and protein synthesis.Br J Exp Pathol1978https://conductscience.org/library/papers/678446PubMed
621,279Changes in human serum amyloid A and C-reactive protein after etiocholanolone-induced inflammation.J Clin Invest1978https://conductscience.org/library/papers/621279PubMed
428,149Levels of the serum amyloid A protein (SAA) in normal persons of different age groups.Clin Exp Immunol1979https://conductscience.org/library/papers/428149PubMed
365,885Genetic disease and amyloid.J Clin Pathol Suppl (R Coll Pathol)1978https://conductscience.org/library/papers/365885PubMed
342,667Genetic control of endotoxic responses in mice.J Exp Med1978https://conductscience.org/library/papers/342667PubMed
311,782Serum amyloid A: evidence for its origin in polymorphonuclear leukocytes.J Clin Invest1978https://conductscience.org/library/papers/311782PubMed

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

None in this release

  • Grants
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  • Comments and corrections
  • References
  • Genes
  • Drugs
  • Data

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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