Paper978136
Murine model for human secondary amyloidosis: genetic variability of the acute-phase serum protein SAA response to endotoxins and casein.
McAdam KP and 1 other. J Exp Med, 1976. Cited by 227 papers, by iCite.
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ConductScience Library. Murine model for human secondary amyloidosis: genetic variability of the acute-phase serum protein SAA response to endotoxins and casein. Papers, record 978136. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/978136
Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- Murine model for human secondary amyloidosis: genetic variability of the acute-phase serum protein SAA response to endotoxins and casein.
- PubMed
Topics13
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Amyloid | Major topic |
| Amyloidosis | Major topic |
| Blood Proteins | Major topic |
| Genetic Variation | Major topic |
| Acute Disease | |
| Animals | |
| Caseins | |
| Disease Models, Animal | |
| Mice | |
| Mice, Inbred Strains | |
| Polysaccharides, Bacterial | |
| Radioimmunoassay | |
| Species Specificity |
Source PubMed
References16
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 182,900 | Isolation of a lipid A bound polypeptide responsible for "LPS-initiated" mitogenesis of C3H/HeJ spleen cells. | J Exp Med | 1976 | https://conductscience.org/library/papers/182900 | PubMed |
| 2 | 5,545,341 | Murine amyloid fibril protein: isolation, purification and characterization. | J Histochem Cytochem | 1971 | https://conductscience.org/library/papers/5545341 | PubMed |
| 3 | 4,154,917 | Casein-induced experimental amyloidosis. III. Response to mitogens, allogeneic cells, and graft-versus-host reactions in the murine model. | Immunology | 1974 | https://conductscience.org/library/papers/4154917 | PubMed |
| 4 | 47,333 | Variation with age and disease of an amyloid A protein-related serum component. | J Clin Invest | 1975 | https://conductscience.org/library/papers/47333 | PubMed |
| 5 | 52,891 | Isolation and characterization of amyloid-related serum protein SAA as a low molecular weight protein. | Scand J Immunol | 1975 | https://conductscience.org/library/papers/52891 | PubMed |
| 6 | 50,401 | Suppression of in vitro antibody response by a serum factor (SAA) in experimentally induced amyloidosis. | J Exp Med | 1975 | https://conductscience.org/library/papers/50401 | PubMed |
| 7 | 51,405 | Association of amyloidosis with erythema nodosum leprosum reactions and recurrent neutrophil leucocytosis in leprosy. | Lancet | 1975 | https://conductscience.org/library/papers/51405 | PubMed |
| 8 | 4,427,088 | Blockage of amyloid induction by colchicine in an animal model. | J Exp Med | 1974 | https://conductscience.org/library/papers/4427088 | PubMed |
| 9 | 4,851,072 | Colchicine inhibition of casein-induced amyloidosis in mice. | Isr J Med Sci | 1974 | https://conductscience.org/library/papers/4851072 | PubMed |
| 10 | 815,910 | Mouse amyloid protein AA: Homology with nonimmunoglobulin protein of human and monkey amyloid substance. | Proc Natl Acad Sci U S A | 1976 | https://conductscience.org/library/papers/815910 | PubMed |
| 11 | 1,104,749 | Experimental amyloidosis: the inducer is a polyclonal B-cell activator to which susceptibility is under genetic control. | J Exp Med | 1975 | https://conductscience.org/library/papers/1104749 | PubMed |
| 12 | 1,104,747 | Immunologic properties of bacterial lipopolysaccharide (LPS). II. The unresponsiveness of C3H/HeJ Mouse spleen cells to LPS-induced mitogenesis is dependent on the method used to extract LPS. | J Exp Med | 1975 | https://conductscience.org/library/papers/1104747 | PubMed |
| 13 | 765,426 | Amyloid-related serum protein SAA in endotoxin-induced amyloidosis of the mink. | J Exp Med | 1976 | https://conductscience.org/library/papers/765426 | PubMed |
| 14 | 1,270,801 | Isolation and partial characterization of SAA-an amyloid-related protein from human serum. | J Immunol | 1976 | https://conductscience.org/library/papers/1270801 | PubMed |
| 15 | 1,254,964 | Amyloid fibril protein AA: purification and properties of the antigenically related serum component as determined by solid phase radioimmunoassay. | J Immunol | 1976 | https://conductscience.org/library/papers/1254964 | PubMed |
| 16 | 4,107,657 | Murine amyloidosis: immunologic characterization of amyloid fibril protein. | J Immunol | 1971 | https://conductscience.org/library/papers/4107657 | PubMed |
Cited by76
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 227 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 40,500,180 | Surveillance of amyloid A deposits in commercial beef liver and assessment of food hygiene risks. | J Vet Med Sci | 2025 | https://conductscience.org/library/papers/40500180 | PubMed |
| 39,839,724 | Identification and characterization of spontaneous AA amyloidosis in CD-1 mice used in toxicity studies: implications of SAA1 and SAA2 copy number variations. | J Toxicol Pathol | 2025 | https://conductscience.org/library/papers/39839724 | PubMed |
| 36,936,978 | C-reactive protein - My perspective on its first half century, 1930-1982. | Front Immunol | 2023 | https://conductscience.org/library/papers/36936978 | PubMed |
| 36,418,077 | Local administration of amyloid enhancing factor initiates in situ amyloid A deposition followed by systemic lesions in mice. | Exp Anim | 2023 | https://conductscience.org/library/papers/36418077 | PubMed |
| 36,059,501 | Integrative analyses of mRNA and microRNA expression profiles reveal the innate immune mechanism for the resistance to Vibrio parahaemolyticus infection in Epinephelus coioides. | Front Immunol | 2022 | https://conductscience.org/library/papers/36059501 | PubMed |
| 35,237,660 | Amyloid Formation by Globular Proteins: The Need to Narrow the Gap Between in Vitro and in Vivo Mechanisms. | Front Mol Biosci | 2022 | https://conductscience.org/library/papers/35237660 | PubMed |
| 34,401,710 | The potential role of glycosaminoglycans in serum amyloid A fibril formation by in silico approaches. | Matrix Biol Plus | 2021 | https://conductscience.org/library/papers/34401710 | PubMed |
| 33,907,055 | Species-barrier on the cross-species oral transmission of bovine AA amyloidosis in mice. | J Vet Med Sci | 2021 | https://conductscience.org/library/papers/33907055 | PubMed |
| 33,761,762 | Sexually Dimorphic Relationships Among Saa3 (Serum Amyloid A3), Inflammation, and Cholesterol Metabolism Modulate Atherosclerosis in Mice. | Arterioscler Thromb Vasc Biol | 2021 | https://conductscience.org/library/papers/33761762 | PubMed |
| 32,060,858 | Serum Amyloid A is Expressed in the Brain After Traumatic Brain Injury in a Sex-Dependent Manner. | Cell Mol Neurobiol | 2020 | https://conductscience.org/library/papers/32060858 | PubMed |
| 30,852,808 | Serum amyloid A levels are associated with polymorphic variants in the serum amyloid A 1 and 2 genes. | Ir J Med Sci | 2019 | https://conductscience.org/library/papers/30852808 | PubMed |
| 28,680,349 | Serum Level of HMGB1 Protein and Inflammatory Markers in Patients with Secondary Peritonitis: Time Course and the Association with Clinical Status. | J Med Biochem | 2017 | https://conductscience.org/library/papers/28680349 | PubMed |
| 27,898,742 | High-Density Lipoprotein (HDL) Counter-Regulates Serum Amyloid A (SAA)-Induced sPLA2-IIE and sPLA2-V Expression in Macrophages. | PLoS One | 2016 | https://conductscience.org/library/papers/27898742 | PubMed |
| 26,419,399 | Systemic Amyloid A Amyloidosis in Island Foxes (Urocyon littoralis): Severity and Risk Factors. | Vet Pathol | 2016 | https://conductscience.org/library/papers/26419399 | PubMed |
| 26,052,716 | Serum Amyloid A Stimulates PKR Expression and HMGB1 Release Possibly through TLR4/RAGE Receptors. | Mol Med | 2015 | https://conductscience.org/library/papers/26052716 | PubMed |
| 25,974,206 | Chronic inflammation aggravates metabolic disorders of hepatic fatty acids in high-fat diet-induced obese mice. | Sci Rep | 2015 | https://conductscience.org/library/papers/25974206 | PubMed |
| 25,429,466 | Proteomic analysis of highly prevalent amyloid A amyloidosis endemic to endangered island foxes. | PLoS One | 2014 | https://conductscience.org/library/papers/25429466 | PubMed |
| 25,251,243 | Deletion of serum amyloid A3 improves high fat high sucrose diet-induced adipose tissue inflammation and hyperlipidemia in female mice. | PLoS One | 2014 | https://conductscience.org/library/papers/25251243 | PubMed |
| 25,073,702 | Serum amyloid A is a retinol binding protein that transports retinol during bacterial infection. | Elife | 2014 | https://conductscience.org/library/papers/25073702 | PubMed |
| 24,878,279 | Divergent effect of glycosaminoglycans on the in vitro aggregation of serum amyloid A. | Biochimie | 2014 | https://conductscience.org/library/papers/24878279 | PubMed |
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- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
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- Data
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- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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