Paper6229596
Allosuppressor and allohelper T cells in acute and chronic graft-vs-host disease. V. F1 mice with secondary chronic GVHD contain F1-reactive allohelper but no allosuppressor T cells.
Pals ST and 2 others. J Exp Med, 1984. Cited by 40 papers, by iCite.
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ConductScience Library. Allosuppressor and allohelper T cells in acute and chronic graft-vs-host disease. V. F1 mice with secondary chronic GVHD contain F1-reactive allohelper but no allosuppressor T cells. Papers, record 6229596. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/6229596
Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- Allosuppressor and allohelper T cells in acute and chronic graft-vs-host disease. V. F1 mice with secondary chronic GVHD contain F1-reactive allohelper but no allosuppressor T cells.
- PubMed
Topics18
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Graft vs Host Disease | Major topic |
| T-Lymphocytes, Helper-Inducer | Major topic |
| T-Lymphocytes, Regulatory | Major topic |
| Acute Disease | |
| Animals | |
| Chronic Disease | |
| Crosses, Genetic | |
| Female | |
| Hematopoiesis | |
| Hematopoietic Stem Cells | |
| Immune Tolerance | |
| Immunization, Passive | |
| Lymphoid Tissue | |
| Mice | |
| Mice, Inbred BALB C | |
| Mice, Inbred C57BL | |
| Radiation Chimera | |
| Spleen |
Source PubMed
References34
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 6,445,861 | Suppressive mechanisms involving sensitization and tolerance in contact allergy. | Immunol Rev | 1980 | https://conductscience.org/library/papers/6445861 | PubMed |
| 2 | 4,404,878 | Serologically defined and lymphocyte-defined components of the major histocompatibility complex in the mouse. | J Exp Med | 1972 | https://conductscience.org/library/papers/4404878 | PubMed |
| 3 | 7,410,851 | Models for the production of stable hematopoietic chimerism across major histocompatibility barriers in adults. | J Immunol | 1980 | https://conductscience.org/library/papers/7410851 | PubMed |
| 4 | 6,448,894 | Cell-mediated immunity to non-HLA antigens of the host by donor lymphocytes in patients with chronic graft-vs-host disease. | J Immunol | 1980 | https://conductscience.org/library/papers/6448894 | PubMed |
| 5 | 6,452,977 | Immune function in fully allogeneic mouse bone marrow chimeras. | Clin Immunol Immunopathol | 1981 | https://conductscience.org/library/papers/6452977 | PubMed |
| 6 | 6,166,716 | Self recognition in allogeneic radiation bone marrow chimeras. A radiation-resistant host element dictates the self specificity and immune response gene phenotype of T-helper cells. | J Exp Med | 1981 | https://conductscience.org/library/papers/6166716 | PubMed |
| 7 | 6,454,750 | Capacity of genetically different T lymphocytes to induce lethal graft-versus-host disease correlates with their capacity to generate suppression but not with their capacity to generate anti-F1 killer cells. A non-H-2 locus determines the inability to induce lethal graft-versus-host disease. | J Exp Med | 1981 | https://conductscience.org/library/papers/6454750 | PubMed |
| 8 | 7,021,693 | Improvement of the protein A plaque assay for immunoglobulin secreting cells by using immunoglobulin-depleted guinea pig serum as a source of complement. | J Immunol Methods | 1981 | https://conductscience.org/library/papers/7021693 | PubMed |
| 9 | 6,461,714 | Allosuppressor and allohelper T cells in acute and chronic graft-vs-host disease. I. Alloreactive suppressor cells rather than killer T cells appear to be the decisive effector cells in lethal graft-vs.-host disease. | J Exp Med | 1982 | https://conductscience.org/library/papers/6461714 | PubMed |
| 10 | 6,978,376 | Diseases caused by reactions of T lymphocytes towards incompatible structures of the major histocompatibility complex. VI. Autoantibodies characteristic of systemic lupus erythematosus induced by abnormal T-B cell cooperation across I-E. | J Exp Med | 1982 | https://conductscience.org/library/papers/6978376 | PubMed |
| 11 | 6,978,818 | A systemic lupus erythematosus (SLE)-like disease in mice induced by abnormal T-B cell cooperation. Preferential formation of autoantibodies characteristic of SLE. | Eur J Immunol | 1982 | https://conductscience.org/library/papers/6978818 | PubMed |
| 12 | 6,213,709 | Modulation of F1 cytotoxic potentials by GvHR: suppression of cytotoxic T cell responses of F1 mice correlates with F1 inability to resist the proliferation of GvHR-inducing parental T lymphocytes. | J Immunol | 1982 | https://conductscience.org/library/papers/6213709 | PubMed |
| 13 | 7,050,708 | Identification by HLA typing of intrauterine-derived maternal T cells in four patients with severe combined immunodeficiency. | N Engl J Med | 1982 | https://conductscience.org/library/papers/7050708 | PubMed |
| 14 | 4,266,664 | A major role for the Ir-1 region of the mouse H-2 complex in the mixed leukocyte reaction. | Transplant Proc | 1973 | https://conductscience.org/library/papers/4266664 | PubMed |
| 15 | 4,587,740 | A rapid method for the isolation of functional thymus-derived murine lymphocytes. | Eur J Immunol | 1973 | https://conductscience.org/library/papers/4587740 | PubMed |
| 16 | 4,151,282 | Cellular immunology and the pathogenesis of graft versus host reactions. | Prog Allergy | 1971 | https://conductscience.org/library/papers/4151282 | PubMed |
| 17 | 124,761 | Association of immunity and tolerance to host H-2 determinants in irradiated F1 hybrid mice reconstituted with bone marrow cells from one parental strain. | J Exp Med | 1975 | https://conductscience.org/library/papers/124761 | PubMed |
| 18 | 51,901 | Collaboration of histoincompatible T and B lymphocytes using cells from tetraparental bone marrow chimeras. | J Exp Med | 1975 | https://conductscience.org/library/papers/51901 | PubMed |
| 19 | 14,213 | Suppressor T cells arising in mice undergoing a graft-vs-host response. | J Immunol | 1977 | https://conductscience.org/library/papers/14213 | PubMed |
| 20 | 16,073 | The Ly phenotype of suppressor T cells arising in mice subjected to a graft-versus-host reaction. | J Exp Med | 1977 | https://conductscience.org/library/papers/16073 | PubMed |
Cited by7
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 40 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 40,433,377 | Identification and validation of tissue-based gene biomarkers for acute intestinal graft-versus-host disease(AIGVHD). | Front Immunol | 2025 | https://conductscience.org/library/papers/40433377 | PubMed |
| 16,791,603 | Nephrotic syndrome in a child after umbilical-cord-blood transplantation. | Pediatr Nephrol | 2006 | https://conductscience.org/library/papers/16791603 | PubMed |
| 8,958,055 | Completely allogeneic spleen cells induced cytolytic neonatal tolerance to alloantigens, but failed to establish allo-helper interactions with host T cells. | Immunology | 1996 | https://conductscience.org/library/papers/8958055 | PubMed |
| 2,957,456 | Regulation of autoimmunity and donor cell engraftment by recipient Lyt-2+ cells during the graft-versus-host reaction. | J Exp Med | 1987 | https://conductscience.org/library/papers/2957456 | PubMed |
| 2,873,640 | Bone marrow transplantation: the genetic and cellular basis of resistance to engraftment and acute graft-versus-host disease. | Surv Immunol Res | 1985 | https://conductscience.org/library/papers/2873640 | PubMed |
| 2,531,761 | Intestinal graft-versus-host disease is initiated by donor T cells distinct from classic cytotoxic T lymphocytes. | J Clin Invest | 1989 | https://conductscience.org/library/papers/2531761 | PubMed |
| 2,148,286 | Persistence of allospecific helper T cells is required for maintaining autoantibody formation in lupus-like graft-versus-host disease. | Clin Exp Immunol | 1990 | https://conductscience.org/library/papers/2148286 | PubMed |
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- 2026-11-11
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- 2026-09-18
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- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
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- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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