Paper62670
Expression of new idiotypes following neonatal idiotypic suppression of a dominant clone.
Augustin A and 1 other. Eur J Immunol, 1976. Cited by 96 papers, by iCite.
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ConductScience Library. Expression of new idiotypes following neonatal idiotypic suppression of a dominant clone. Papers, record 62670. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/62670
Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- Expression of new idiotypes following neonatal idiotypic suppression of a dominant clone.
- PubMed
Topics13
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Animals, Newborn | Major topic |
| Choline | Major topic |
| Immunoglobulin Allotypes | Major topic |
| Immunosuppression Therapy | Major topic |
| Phosphorylcholine | Major topic |
| Age Factors | |
| Animals | |
| Antibodies, Anti-Idiotypic | |
| Antigen-Antibody Reactions | |
| Epitopes | |
| Mice | |
| Mice, Inbred BALB C | |
| Myeloma Proteins |
Source PubMed
Cited by17
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 96 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 32,477,339 | Relevance of the Materno-Fetal Interface for the Induction of Antigen-Specific Immune Tolerance. | Front Immunol | 2020 | https://conductscience.org/library/papers/32477339 | PubMed |
| 9,989,978 | Neonatal idiotypic exposure alters subsequent cytokine, pathology, and survival patterns in experimental Schistosoma mansoni infections. | J Exp Med | 1999 | https://conductscience.org/library/papers/9989978 | PubMed |
| 9,393,802 | Oligoclonality of serum immunoglobulin G antibody responses to Streptococcus pneumoniae capsular polysaccharide serotypes 6B, 14, and 23F. | Infect Immun | 1997 | https://conductscience.org/library/papers/9393802 | PubMed |
| 8,943,710 | Autologous anti-idiotypic antibody response is regulated by the level of circulating complementary idiotype. | Immunology | 1996 | https://conductscience.org/library/papers/8943710 | PubMed |
| 7,252,411 | Antiphosphocholine antibodies found in normal mouse serum are protective against intravenous infection with type 3 streptococcus pneumoniae. | J Exp Med | 1981 | https://conductscience.org/library/papers/7252411 | PubMed |
| 6,972,994 | Generation of idiotype-specific T cell help through network perturbation. | J Exp Med | 1981 | https://conductscience.org/library/papers/6972994 | PubMed |
| 6,377,539 | Suppression of autoimmune diseases with anti-idiotypic antibodies: murine lupus nephritis as a model. | Springer Semin Immunopathol | 1984 | https://conductscience.org/library/papers/6377539 | PubMed |
| 6,222,133 | Induction of idiotype-specific suppressor T cells with antigen/antibody complexes. | J Exp Med | 1983 | https://conductscience.org/library/papers/6222133 | PubMed |
| 6,213,731 | Immunoglobulin idiotopes expressed by T cells. I. Expression of distinct idiotopes detected by monoclonal antibodies on antigen-specific suppressor T cells. | J Exp Med | 1982 | https://conductscience.org/library/papers/6213731 | PubMed |
| 6,163,071 | Autoregulation of immune responses via idiotype network interactions. | Microbiol Rev | 1980 | https://conductscience.org/library/papers/6163071 | PubMed |
| 3,081,440 | Isoelectric focusing spectra of anti-bacterial alpha-amylase antibody unique for antigen-induced suppression. | Immunology | 1986 | https://conductscience.org/library/papers/3081440 | PubMed |
| 2,580,040 | Cellular basis for neonatally induced T-suppressor activity. Primary B cell maturation is blocked by suppressor-helper interactions restricted by loci on chromosome 12. | J Exp Med | 1985 | https://conductscience.org/library/papers/2580040 | PubMed |
| 702,066 | Alteration of clonal profile. I. Effect of sublethal irradiation on the responses to phosphorylcholine in BALB/c mice. | J Exp Med | 1978 | https://conductscience.org/library/papers/702066 | PubMed |
| 410,029 | Potentiation of a primary in vivo antibody response by alloantisera against gene products of the I region of the H-2 complex. | Proc Natl Acad Sci U S A | 1977 | https://conductscience.org/library/papers/410029 | PubMed |
| 309,912 | Anti-idiotype induced regulation of helper cell function for the response to phosphorylcholine in adult BALB/c mice. | J Exp Med | 1978 | https://conductscience.org/library/papers/309912 | PubMed |
| 306,109 | Clonal dominance: loss and restoration in adoptive transfer. | Proc Natl Acad Sci U S A | 1978 | https://conductscience.org/library/papers/306109 | PubMed |
| 78,956 | Simultaneous complementary idiotypic responses: absence of reciprocal regulation. | J Exp Med | 1978 | https://conductscience.org/library/papers/78956 | PubMed |
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- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
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- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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