Paper410029
Potentiation of a primary in vivo antibody response by alloantisera against gene products of the I region of the H-2 complex.
Pierres M and 3 others. Proc Natl Acad Sci U S A, 1977. Cited by 39 papers, by iCite.
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ConductScience Library. Potentiation of a primary in vivo antibody response by alloantisera against gene products of the I region of the H-2 complex. Papers, record 410029. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/410029
Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- Potentiation of a primary in vivo antibody response by alloantisera against gene products of the I region of the H-2 complex.
- PubMed
Topics10
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Genes, MHC Class II | Major topic |
| Isoantibodies | Major topic |
| Animals | |
| Erythrocytes | |
| Immunoglobulin G | |
| Immunoglobulin M | |
| Mice | |
| Mice, Inbred Strains | |
| Sheep | |
| Species Specificity |
Source PubMed
References21
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 68,991 | Immunosuppressive factor(s) specific for L-glutamic acid50-L-tyrosine50 (GT) II. Presence of I-J determinants on the GT-suppressive factor. | J Exp Med | 1977 | https://conductscience.org/library/papers/68991 | PubMed |
| 2 | 62,670 | Expression of new idiotypes following neonatal idiotypic suppression of a dominant clone. | Eur J Immunol | 1976 | https://conductscience.org/library/papers/62670 | PubMed |
| 3 | 67,169 | T-T interactions in the induction of suppressor and helper T cells: analysis of membrane phenotype of precursor and amplifier cells. | J Exp Med | 1977 | https://conductscience.org/library/papers/67169 | PubMed |
| 4 | 299,766 | Identification of a B-cell surface structure involved in antigen-dependent triggering: absence of this structure on B cells from CBA/N mutant mice. | J Exp Med | 1977 | https://conductscience.org/library/papers/299766 | PubMed |
| 5 | 4,546,826 | Idiotype suppression. I. Influence of the dose and of the effector functions of anti-idiotypic antibody on the production of an idiotype. | Eur J Immunol | 1974 | https://conductscience.org/library/papers/4546826 | PubMed |
| 6 | 47,219 | The H-2 major histocompatibility complex and the I immune response region: genetic variation, function, and organization. | Adv Immunol | 1975 | https://conductscience.org/library/papers/47219 | PubMed |
| 7 | 4,610,076 | Immune responses in vitro. XI. Suppression of primary IgM and IgG plaque-forming cell responses in vitro by alloantisera against leukocyte alloantigens. | J Exp Med | 1974 | https://conductscience.org/library/papers/4610076 | PubMed |
| 8 | 55,062 | The histocompatibility-linked immune response genes. | Adv Cancer Res | 1975 | https://conductscience.org/library/papers/55062 | PubMed |
| 9 | 52,924 | Immunological suppression of idiotypic specificities. | Transplant Rev | 1975 | https://conductscience.org/library/papers/52924 | PubMed |
| 10 | 170,342 | Anomalous reactions of mouse alloantisera with cultured tumor cells. II. Cytotoxicity is caused by antibodies to leukemia viruses. | J Immunol | 1975 | https://conductscience.org/library/papers/170342 | PubMed |
| 11 | 4,934,502 | Immune responses in vitro. 3. Development of primary gamma-M, gamma-G, and gamma-A plaque-forming cell responses in mouse spleen cell cultures stimulated with heterologous erythrocytes. | J Exp Med | 1971 | https://conductscience.org/library/papers/4934502 | PubMed |
| 12 | 1,085,339 | Properties of the antigen-specific suppressive T-cell factor in the regulation of antibody response of the mouse. IV. Special subregion assignment of the gene(s) that codes for the suppressive T-cell factor in the H-2 histocompatibility complex. | J Exp Med | 1976 | https://conductscience.org/library/papers/1085339 | PubMed |
| 13 | 1,085,338 | A new I subregion (I-J) marked by a locus (Ia-4) controlling surface determinants on suppressor T lymphocytes. | J Exp Med | 1976 | https://conductscience.org/library/papers/1085338 | PubMed |
| 14 | 1,085,337 | Selective expression of H-2 (i-region) loci controlling determinants on helper and suppressor T lymphocytes. | J Exp Med | 1976 | https://conductscience.org/library/papers/1085337 | PubMed |
| 15 | 1,101,471 | Low zone tolerance and suppressor T cells. | Transplant Rev | 1975 | https://conductscience.org/library/papers/1101471 | PubMed |
| 16 | 1,100,764 | Antigen-specific T-cell factor in cell cooperation. Mapping within the I region of the H-2 complex and ability to cooperate across allogeneic barriers. | J Exp Med | 1975 | https://conductscience.org/library/papers/1100764 | PubMed |
| 17 | 1,079,849 | The role of macrophages in the generation of T-helper cells. II. The genetic control of the macrophage-T-cell interaction for helper cell induction with soluble antigens. | J Exp Med | 1975 | https://conductscience.org/library/papers/1079849 | PubMed |
| 18 | 1,078,737 | The function and interrelationships of T-cell receptors, Ir genes and other histocompatibility gene products. | Transplant Rev | 1975 | https://conductscience.org/library/papers/1078737 | PubMed |
| 19 | 932,641 | Genetic control of specific immune suppression. III. Mapping of H-2 complex complementing genes controlling immune suppression by the random copolymer L-glutamic acid50-L-tyrosine50 (GT). | J Exp Med | 1976 | https://conductscience.org/library/papers/932641 | PubMed |
| 20 | 1,118,728 | Inhibition of immune responses in vitro by specific antiserums to Ia antigens. | Science | 1975 | https://conductscience.org/library/papers/1118728 | PubMed |
Cited by13
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 39 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 6,972,040 | Modulation of Schistosoma mansoni egg-induced granuloma formation: I-J restriction of T cell-mediated suppression in a chronic parasitic infection. | Proc Natl Acad Sci U S A | 1981 | https://conductscience.org/library/papers/6972040 | PubMed |
| 6,947,275 | In vivo effects of antibodies to immune response gene products: prevention of experimental allergic encephalitis. | Proc Natl Acad Sci U S A | 1981 | https://conductscience.org/library/papers/6947275 | PubMed |
| 6,810,361 | Demonstration of active tolerance in maintenance of established islet of Langerhans allografts. | Proc Natl Acad Sci U S A | 1982 | https://conductscience.org/library/papers/6810361 | PubMed |
| 6,795,305 | In vivo effects of antibodies to immune response gene products. I. Haplotype-specific suppression of humoral immune responses with a monoclonal anti-I-A. | J Exp Med | 1981 | https://conductscience.org/library/papers/6795305 | PubMed |
| 6,795,302 | Regulation of immune responses by I-J gene products. I. Production and characterization of anti-I-J monoclonal antibodies. | J Exp Med | 1981 | https://conductscience.org/library/papers/6795302 | PubMed |
| 6,459,991 | Genetic control of allogeneic interactions in the guinea-pig. IV. The specific elimination of MLC suppressor cells by the in vivo administration of antisera against a GPLA-B region-controlled determinant. | Immunology | 1982 | https://conductscience.org/library/papers/6459991 | PubMed |
| 6,222,135 | Early development of the T cell repertoire. In vivo treatment of neonatal mice with anti-Ia antibodies interferes with differentiation of I-restricted T cells but not K/D-restricted T cells. | J Exp Med | 1983 | https://conductscience.org/library/papers/6222135 | PubMed |
| 311,007 | Direct evidence for loss of human suppressor cells during active autoimmune disease. | Proc Natl Acad Sci U S A | 1978 | https://conductscience.org/library/papers/311007 | PubMed |
| 307,250 | Regulation of antibody heterogeneity by suppressor T cells: diminishing suppressor T cell activity increases the number of dinitrophenyl clones in mice immunized with dinitrophenyl-poly(Glu,Lys,Phe) or dinitrophenyl-poly(Glu,Lys,Ala). | Proc Natl Acad Sci U S A | 1978 | https://conductscience.org/library/papers/307250 | PubMed |
| 270,747 | Reduction of syngeneic tumor growth by an anti-I-J-alloantiserum. | Proc Natl Acad Sci U S A | 1977 | https://conductscience.org/library/papers/270747 | PubMed |
| 102,725 | The involvement of suppressor T cells in Ir gene regulation of secondary antibody responses of primed (responder X nonresponder)F1 mice to macrophage-bound L-glutamic acid60-L-alanine30-L-tyrosine. | J Exp Med | 1978 | https://conductscience.org/library/papers/102725 | PubMed |
| 84,040 | Regulatory mechanisms in cell-mediated immune responses. VII. Presence of I-C subregion determinants on mixed leukocyte reaction suppressor factor. | J Exp Med | 1979 | https://conductscience.org/library/papers/84040 | PubMed |
| 75,939 | In vivo effects of anti-Ia alloantisera. I. Elimination of specific suppression by in vivo administration of antisera specific for I-J controlled determinants. | J Exp Med | 1978 | https://conductscience.org/library/papers/75939 | PubMed |
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- iCiteCC BY 4.0
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- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
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- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
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