Paper · 75237
H-2-restricted cytotoxic effectors generated in vitro by the addition of trinitrophenyl-conjugated soluble proteins.
Schmitt-Verhulst AM and 4 others. J Exp Med, 1978. Cited by 115 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
Cite this record
ConductScience Library. H-2-restricted cytotoxic effectors generated in vitro by the addition of trinitrophenyl-conjugated soluble proteins. Papers, record 75237. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/75237
On this page
Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- H-2-restricted cytotoxic effectors generated in vitro by the addition of trinitrophenyl-conjugated soluble proteins.
- PubMed
Topics14
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Cytotoxicity, Immunologic | Major topic |
| H-2 Antigens | Major topic |
| Nitrobenzenes | Major topic |
| T-Lymphocytes | Major topic |
| Trinitrobenzenes | Major topic |
| Animals | |
| Carrier Proteins | |
| Cells, Cultured | |
| Dose-Response Relationship, Immunologic | |
| Mice | |
| Serum Albumin, Bovine | |
| Solubility | |
| Spleen | |
| gamma-Globulins |
Source PubMed
Grants0
Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.
None in this release.
Trials0
The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.
None in this release.
Comments and corrections0
PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.
None in this release.
References28
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 1,079,848 | On the role of the H-2 histocompatibility complex in determining the specificity of cytotoxic effector cells sensitized against syngeneic trinitrophenyl-modified targets. | J Exp Med | 1975 | https://conductscience.org/library/papers/1079848 | PubMed |
| 2 | 1,079,924 | Interaction antigens detected by cytotoxic T cells with the major histocompatibility complex as modifier. | Nature | 1975 | https://conductscience.org/library/papers/1079924 | PubMed |
| 3 | 1,081,575 | In vitro cell-mediated immune responses to the male specific(H-Y) antigen in mice. | J Exp Med | 1975 | https://conductscience.org/library/papers/1081575 | PubMed |
| 4 | 1,086,219 | Cell-medicated cytotoxicity against ectromelia virus-infected target cells. III. Role of the H-2 gene complex. | Eur J Immunol | 1975 | https://conductscience.org/library/papers/1086219 | PubMed |
| 5 | 1,086,229 | Target cell-dependent T cell-mediated lysis of vaccinia virus-infected cells. | Eur J Immunol | 1975 | https://conductscience.org/library/papers/1086229 | PubMed |
| 6 | 1,087,329 | Multiple H-2 linked immune response gene control of H-2 D-associated T-cell-mediated lympholysis to trinitrophenyl-modified autologous cells: Ir-like genes mapping to the left of I-A and within the I region. | J Exp Med | 1976 | https://conductscience.org/library/papers/1087329 | PubMed |
| 7 | 1,244,419 | Cell-mediated lympholysis of trinitrophenyl-modified autologous lymphocytes. Confirmation of genetic control of response to trinitrophenyl-modified H-2 antigens by the use of anti-H-2 and anti-Ia antibodies. | J Exp Med | 1976 | https://conductscience.org/library/papers/1244419 | PubMed |
| 8 | 177,884 | H-2 region product as determinant in immune cytolysis of syngeneic tumour cells by anti-MSV T lymphocytes. | Nature | 1976 | https://conductscience.org/library/papers/177884 | PubMed |
| 9 | 233,918 | Joint recognition by cytotoxic T cells of inactivated Sendai virus and products of the major histocompatibility complex. | J Exp Med | 1977 | https://conductscience.org/library/papers/233918 | PubMed |
| 10 | 325,135 | Antigen handling by guinea pig macrophages: further evidence for the sequestration of antigen relevant for activation of primed T lymphocytes. | J Immunol | 1977 | https://conductscience.org/library/papers/325135 | PubMed |
1 to 10 of 28
See all 28 referencesCited by24
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 115 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 37,626,879 | Enhanced Membrane Fluidization and Cholesterol Displacement by 1-Heptanol Inhibit Mast Cell Effector Functions. | Cells | 2023 | https://conductscience.org/library/papers/37626879 | PubMed |
| 31,993,060 | Cytoskeletal Protein 4.1R Is a Positive Regulator of the FcεRI Signaling and Chemotaxis in Mast Cells. | Front Immunol | 2019 | https://conductscience.org/library/papers/31993060 | PubMed |
| 30,116,247 | Positive and Negative Regulatory Roles of C-Terminal Src Kinase (CSK) in FcεRI-Mediated Mast Cell Activation, Independent of the Transmembrane Adaptor PAG/CSK-Binding Protein. | Front Immunol | 2018 | https://conductscience.org/library/papers/30116247 | PubMed |
| 26,929,198 | New Regulatory Roles of Galectin-3 in High-Affinity IgE Receptor Signaling. | Mol Cell Biol | 2016 | https://conductscience.org/library/papers/26929198 | PubMed |
| 21,302,107 | Murine cytotoxic T-cell response to alphavirus is associated mainly withH- 2D ( k ). | Immunogenetics | 1978 | https://conductscience.org/library/papers/21302107 | PubMed |
| 17,587,407 | Rab27b regulates mast cell granule dynamics and secretion. | Traffic | 2007 | https://conductscience.org/library/papers/17587407 | PubMed |
| 10,049,941 | Cytotoxicity is mandatory for CD8(+) T cell-mediated contact hypersensitivity. | J Exp Med | 1999 | https://conductscience.org/library/papers/10049941 | PubMed |
| 8,406,579 | Anti-leucocyte function-associated antigen-1 antibodies inhibit T-cell activation following low-avidity and adhesion-independent interactions. | Immunology | 1993 | https://conductscience.org/library/papers/8406579 | PubMed |
| 7,492,304 | Biochemical and pharmacological studies with KT7692 and LY294002 on the role of phosphatidylinositol 3-kinase in Fc epsilon RI-mediated signal transduction. | Biochem J | 1995 | https://conductscience.org/library/papers/7492304 | PubMed |
| 7,320,686 | Protection against graft vs. host-associated immunosuppression in F1 mice. I. Activation of F1 regulatory cells by host-specific anti-major histocompatibility complex antibodies. | J Exp Med | 1981 | https://conductscience.org/library/papers/7320686 | PubMed |
1 to 10 of 24
See all 24 cited byGenes0
The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.
None in this release.
Drugs0
The drugs whose trials, in the Pipelines release, list the paper.
None in this release.
Data0
The data bank entries PubMed's record names. Trial numbers are under Trials.
None in this release.
Data dates
The date of each source this release read.
- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
For software
Read this paper from software.
The record and each of its lists are JSON, twenty rows a page. No key.
GET https://conductscience.org/api/papers/v1/75237
GET https://conductscience.org/api/papers/v1/75237/references?page=2