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Paper · 78955

T-cell populations specifically depleted of alloreactive potential cannot be induced to lyse H-2-different virus-infected target cells.

Bennink JR and 1 other. J Exp Med, 1978. Cited by 39 papers, by iCite.

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ConductScience Library. T-cell populations specifically depleted of alloreactive potential cannot be induced to lyse H-2-different virus-infected target cells. Papers, record 78955. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/78955

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
T-cell populations specifically depleted of alloreactive potential cannot be induced to lyse H-2-different virus-infected target cells.
PubMed
Journal
J Exp Med
PubMed
Year
1978
PubMed
Publication types
Journal Article, Research Support, U.S. Gov't, P.H.S.
PubMed
Citations, by iCite
39
iCite
Relative citation ratio, by iCite
0.82
iCite
Revised in PubMed
2019-05-08
PubMed

Authors2

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Bennink JRJ R
2Doherty PCP C

Source PubMed

Topics14

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
EpitopesMajor topic
H-2 AntigensMajor topic
T-LymphocytesMajor topic
Animals
Antigens, Viral
Cross Reactions
Cytotoxicity, Immunologic
Fibroblasts
Immune Tolerance
Immunologic Memory
Influenza A virus
Mice
Radiation Chimera
Vaccinia virus

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References21

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
11,083,422Cross-priming for a secondary cytotoxic response to minor H antigens with H-2 congenic cells which do not cross-react in the cytotoxic assay.J Exp Med1976https://conductscience.org/library/papers/1083422PubMed
214,978,855The somatic generation of immune recognition.Eur J Immunol1971https://conductscience.org/library/papers/14978855PubMed
3199,844Non-infectious virus induces cytotoxic T lymphocytes and binds to target cells to permit their lysis.Nature1977https://conductscience.org/library/papers/199844PubMed
4233,901Generation of both cross-reactive and virus-specific T-cell populations after immunization with serologically distinct influenza A viruses.J Exp Med1977https://conductscience.org/library/papers/233901PubMed
5300,355Cytotoxic T cells specific for influenza virus-infected target cells.Immunology1977https://conductscience.org/library/papers/300355PubMed
6300,483Heterogeneity of the cytotoxic response of thymus-derived lymphocytes after immunization with influenza viruses.Proc Natl Acad Sci U S A1977https://conductscience.org/library/papers/300483PubMed
7301,550The generation of killer cells to trinitrophenyl-modified allogeneic targets by lymphocyte populations negatively selected to strong alloantigens.J Exp Med1977https://conductscience.org/library/papers/301550PubMed
8302,174The concept that surveillance of self is mediated via the same set of genes that determines recognition of allogenic cells.Cold Spring Harb Symp Quant Biol1977https://conductscience.org/library/papers/302174PubMed
9302,303Immunologic recognition of influenza virus-infected cells. II. Expression of influenza A matrix protein on the infected cell surface and its role in recognition by cross-reactive cytotoxic T cells.J Exp Med1977https://conductscience.org/library/papers/302303PubMed
10302,310Hemagglutinin-specific cytotoxic T-cell response during influenza infection.J Exp Med1977https://conductscience.org/library/papers/302310PubMed

1 to 10 of 21

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Cited by17

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 39 citations.

PMIDTitleJournalYearIn PapersSource
6,980,965Induction of neonatal tolerance to H-2k in B6 mice does not allow the emergence of T cells specific for H-2k plus vaccinia virus.J Exp Med1982https://conductscience.org/library/papers/6980965PubMed
6,967,190MHC restriction, self-tolerance and the thymus.Nature1980https://conductscience.org/library/papers/6967190PubMed
6,965,308Suppression of T cells specific for the nonthymic parental H-2 haplotype in thymus-grafted chimeras.J Exp Med1980https://conductscience.org/library/papers/6965308PubMed
6,965,305T cells that encounter virus in the complete absence of a particular H-2 antigen are nonresponsive when stimulated again in the context of that H-2 antigen.J Exp Med1980https://conductscience.org/library/papers/6965305PubMed
6,578,194Accessory cell and HLA compatibility requirements for the generation of specific in-vitro antibody responses to influenza virus by human blood lymphocytes.Immunology1983https://conductscience.org/library/papers/6578194PubMed
6,455,762T cell recognition of antigen in vivo: role of the H-2 complex.Springer Semin Immunopathol1980https://conductscience.org/library/papers/6455762PubMed
6,213,732Genetic control of the immune response to myoglobins. Both low and high responder T cells tolerant to the other major histocompatibility complex help high but not low responder B cells.J Exp Med1982https://conductscience.org/library/papers/6213732PubMed
6,166,716Self recognition in allogeneic radiation bone marrow chimeras. A radiation-resistant host element dictates the self specificity and immune response gene phenotype of T-helper cells.J Exp Med1981https://conductscience.org/library/papers/6166716PubMed
6,153,213Absence of H-2 restriction in primary and secondary mixed-lymphocyte reactions to strong M1s determinants.J Exp Med1980https://conductscience.org/library/papers/6153213PubMed
2,578,572Treatment of mice with polyinosinic-polycytidilic polyribonucleotide reduces T-cell involvement in a localized inflammatory response to vaccinia virus challenge.J Virol1985https://conductscience.org/library/papers/2578572PubMed

1 to 10 of 17

See all 17 cited by

Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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