ConductScience.org, home

Paper · 8698236

Differential splicing-in of a proline-rich exon converts alphaNAC into a muscle-specific transcription factor.

Yotov WV and 1 other. Genes Dev, 1996. Cited by 79 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

Download JSON
Cite this record

ConductScience Library. Differential splicing-in of a proline-rich exon converts alphaNAC into a muscle-specific transcription factor. Papers, record 8698236. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/8698236

On this page

Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Differential splicing-in of a proline-rich exon converts alphaNAC into a muscle-specific transcription factor.
PubMed
Journal
Genes Dev
PubMed
Year
1996
PubMed
Publication types
Journal Article, Research Support, Non-U.S. Gov't
PubMed
Citations, by iCite
79
iCite
Relative citation ratio, by iCite
1.3
iCite
Revised in PubMed
2019-05-16
PubMed

Authors2

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Yotov WVW V
2St-Arnaud RR

Source PubMed

Topics21

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
MusclesMajor topic
ProteinsMajor topic
Trans-ActivatorsMajor topic
Transcription FactorsMajor topic
Amino Acid Sequence
Animals
Base Sequence
Binding Sites
Cell Differentiation
Cell Line
Cloning, Molecular
DNA, Complementary
Exons
Mice
Molecular Chaperones
Molecular Sequence Data
Proline
RNA Splicing
RNA, Messenger
Transcriptional Activation
Transfection

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References0

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

None in this release.

Cited by35

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 79 citations.

PMIDTitleJournalYearIn PapersSource
42,876,144A Hypertrophic Stimulus Drives Age-Dependent Transcript Isoform Switching With and Without Differential Gene Expression in Skeletal Muscle.Aging Cell2026https://conductscience.org/library/papers/42876144PubMed
37,443,729Lysine Methyltransferase SMYD1 Regulates Myogenesis via skNAC Methylation.Cells2023https://conductscience.org/library/papers/37443729PubMed
34,589,886The SMYD1 and skNAC transcription factors contribute to neurodegenerative diseases.Brain Behav Immun Health2020https://conductscience.org/library/papers/34589886PubMed
34,490,239Lnc-GD2H Promotes Proliferation by Forming a Feedback Loop With c-Myc and Enhances Differentiation Through Interacting With NACA to Upregulate Myog in C2C12 Myoblasts.Front Cell Dev Biol2021https://conductscience.org/library/papers/34490239PubMed
33,875,709Ubiquitin specific peptidase Usp53 regulates osteoblast versus adipocyte lineage commitment.Sci Rep2021https://conductscience.org/library/papers/33875709PubMed
32,541,000Kir2.1 Interactome Mapping Uncovers PKP4 as a Modulator of the Kir2.1-Regulated Inward Rectifier Potassium Currents.Mol Cell Proteomics2020https://conductscience.org/library/papers/32541000PubMed
32,192,231The beta Subunit of Nascent Polypeptide Associated Complex Plays A Role in Flowers and Siliques Development of Arabidopsis thaliana.Int J Mol Sci2020https://conductscience.org/library/papers/32192231PubMed
30,948,508Dephosphorylation of the transcriptional cofactor NACA by the PP1A phosphatase enhances cJUN transcriptional activity and osteoblast differentiation.J Biol Chem2019https://conductscience.org/library/papers/30948508PubMed
29,329,560Overexpression of the double homeodomain protein DUX4c interferes with myofibrillogenesis and induces clustering of myonuclei.Skelet Muscle2018https://conductscience.org/library/papers/29329560PubMed
28,536,403Anti-Differentiation Effect of Oncogenic Met Receptor in Terminally-Differentiated Myotubes.Biomedicines2015https://conductscience.org/library/papers/28536403PubMed

1 to 10 of 35

See all 35 cited by

Genes1

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

GeneNCBI Gene IDLinked byIn Genes
NACA4,666PubTator's text mininghttps://conductscience.org/library/genes/4666

Source NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data2

The data bank entries PubMed's record names. Trial numbers are under Trials.

Data bankAccessionSource
GENBANKU48363GENBANK
GENBANKU48364GENBANK

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

For software

Read this paper from software.

The record and each of its lists are JSON, twenty rows a page. No key.

GET https://conductscience.org/api/papers/v1/8698236

GET https://conductscience.org/api/papers/v1/8698236/cited_by?page=2