Paper976308
Quantitative contributions of IgG, IgM and C3 to erythrophagocytosis and rosette formation by peritoneal macrophages, and anti-opsonin activity of dextran sulfate 500.
Wellek B and 2 others. Eur J Immunol, 1976. Cited by 17 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
Cite this record
ConductScience Library. Quantitative contributions of IgG, IgM and C3 to erythrophagocytosis and rosette formation by peritoneal macrophages, and anti-opsonin activity of dextran sulfate 500. Papers, record 976308. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/976308
Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- Quantitative contributions of IgG, IgM and C3 to erythrophagocytosis and rosette formation by peritoneal macrophages, and anti-opsonin activity of dextran sulfate 500.
- PubMed
Topics15
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Dextrans | Major topic |
| Immunoglobulin G | Major topic |
| Immunoglobulin M | Major topic |
| Macrophages | Major topic |
| Opsonin Proteins | Major topic |
| Phagocytosis | Major topic |
| Animals | |
| Cell Adhesion | |
| Complement C3 | |
| Complement System Proteins | |
| Dose-Response Relationship, Immunologic | |
| Erythrocytes | |
| Guinea Pigs | |
| Hemolysis | |
| Immune Adherence Reaction |
Source PubMed
Cited by9
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 17 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 33,326,036 | The N2N3 domains of ClfA, FnbpA and FnbpB in Staphylococcus aureus bind to human complement factor H, and their antibodies enhance the bactericidal capability of human blood. | J Biochem | 2021 | https://conductscience.org/library/papers/33326036 | PubMed |
| 6,447,230 | Pathopysiological aspects of immune complex diseases. Part II. Phagocytosis, exocytosis, and pathogenic depositions. | Klin Wochenschr | 1980 | https://conductscience.org/library/papers/6447230 | PubMed |
| 6,396,178 | Fc receptor--more answers, more questions. | Folia Microbiol (Praha) | 1984 | https://conductscience.org/library/papers/6396178 | PubMed |
| 6,320,776 | Replication of HSV-1 in murine peritoneal macrophages: comparison of various virus strains with different properties. | Arch Virol | 1984 | https://conductscience.org/library/papers/6320776 | PubMed |
| 941,799 | Opsonizing activities of IgG, IgM antibodies and the C3b inactivator-cleaved third component of complement in macrophage phagocytosis. | Agents Actions | 1976 | https://conductscience.org/library/papers/941799 | PubMed |
| 833,545 | The role of membrane receptors for C3b and C3d in phagocytosis. | J Exp Med | 1977 | https://conductscience.org/library/papers/833545 | PubMed |
| 640,735 | Phagocytosis by sheep alveolar macrophages: relationship between opsonin concentration and light emission in the presence of luminol. | Infect Immun | 1978 | https://conductscience.org/library/papers/640735 | PubMed |
| 551,084 | [Humoral defence against infection (author's transl)]. | Infection | 1979 | https://conductscience.org/library/papers/551084 | PubMed |
| 363,971 | Nonspecific resistance to bacterial infections. Enhancement by ubiquinone-8. | J Exp Med | 1978 | https://conductscience.org/library/papers/363971 | PubMed |
Genes2
The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.
| Gene | NCBI Gene ID | Linked by | In Genes | Source |
|---|---|---|---|---|
| C3 | 718 | PubTator's text mining | https://conductscience.org/library/genes/718 | NCBI Gene |
| ERVK-3 | 100,862,689 | PubTator's text mining | https://conductscience.org/library/genes/100862689 | NCBI Gene |
Data dates
The date of each source this release read.
- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
None in this release
- Grants
- Trials
- Comments and corrections
- References
- Drugs
- Data
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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