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YTHDF2: YTH N6-methyladenosine RNA binding protein F2
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Gene · 51441

Papers3,768

Newest first. Swiss-Prot's references are curated; PubTator's text mining finds papers that name the gene, not always ones about it.

21 to 40 of 3,768

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TitleJournalYearPMIDFound bySource
FTO-mediated m6A demethylation of SNHG9 enhances PINK1-dependent mitophagy and reverses cisplatin resistance via nuclear EZH2 ubiquitination.Biochem Pharmacol202642600947PubTator, text miningPubMed
Ythdf family m6A readers promote retinal ganglion cell fate reprogramming and neurite development.Exp Eye Res202642595204PubTator, text miningPubMed
Polydatin in combination with hawthorn flavonoids alleviates atherosclerosis by inhibiting mitochondrial division through the YTHDF2-DRP1 axis.J Adv Res202642595105PubTator, text miningPubMed
USP33-mediated deubiquitination stabilizes YTHDF2 and promotes glioblastoma malignancy and temozolomide resistance.Biochem Biophys Res Commun202642594813PubTator, text miningPubMed
Expansion of functional human long-term HSCs through restraining excessive cell cycle activation.Blood202642561119PubTator, text miningPubMed
Vascular smooth muscle cell FTO deletion aggravates atherosclerotic plaque instability by promoting VSMC migration through the N6-methyladenosine modification of the COL3A1 mRNA.Sci China Life Sci202642557509PubTator, text miningPubMed
Reader-dependent functional duality of FTO: a context-switching node at the intersection of immune evasion and therapeutic resistance.Front Immunol202642548706PubTator, text miningPubMed
Combined anti-PD-1 with irreversible electroporation relieves immunosuppression and reduces hepatocellular carcinoma regrowth.JHEP Rep202642546925PubTator, text miningPubMed
From contested target to context-aware node: A three-layer regulatory code for interpreting FTO's oncogene-tumor suppressor paradox in cancer.Exp Cell Res202642546791PubTator, text miningPubMed
A multivalent RNA affinity tag enables selective purification of tagged RNAs and bound proteins.Nucleic Acids Res202642531073PubTator, text miningPubMed
N6-methyladenosine reader YTHDF2 facilitates malignant progression of gallbladder cancer by suppressing NME1 expression.Cancer Lett202642498073PubTator, text miningPubMed
Carboplatin Is Associated with Changes in Components of the m6A Machinery in Triple-Negative Breast Cancer Cells.ACS Omega202642495278PubTator, text miningPubMed
Retraction notice to "FTO-overexpressing extracellular vesicles from BM-MSCs reverse cellular senescence and aging to ameliorate osteoarthritis by modulating METTL3/ YTHDF2-mediated RNA m6A modifications" [International Journal of Biological Macromolecules 278 (2024) 134600].
Int J Biol Macromol202642481303PubTator, text miningPubMed
Dynamic m6A epitranscriptomic remodeling coordinates host transcriptional reprogramming during MPXV infection.Cell Commun Signal202642469869PubTator, text miningPubMed
ZDHHC18-YTHDF2 axis suppresses USP13 to drive cardiac hypertrophy.Pharmacol Res202642468582PubTator, text miningPubMed
YTHDF2-regulated hsa_circ_0005882 functions as a sponge for miR-654-3p to suppress nasopharyngeal carcinoma proliferation.Neoplasma202642429202PubTator, text miningPubMed
EIF4A3 facilitates METTL3-mediated m6A methylation of circPCMTD1 to drive bladder cancer progression via miR-3681-5p/HPSE axis.Cell Signal202642413642PubTator, text miningPubMed
Correction: YTHDF2 in peritumoral hepatocytes mediates chemotherapy-induced antitumor immune responses through CX3CL1-mediated CD8+ T cell recruitment.Mol Cancer202642400008PubTator, text miningPubMed
Gigantol induces ferroptosis in gastric cancer via methyltransferase-like 3/YTH domain-containing family protein 2-mediated β-catenin degradation.Phytomedicine202642391814PubTator, text miningPubMed
Interferon-elicited lipoprotein metabolism in lung fibroblasts facilitates premetastatic niche formation.Nat Commun202642386744PubTator, text miningPubMed

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  • ClinicalTrials.gov's posted results, through CTTI's AACTFreely available. Acknowledgement requested
  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • EMBL-EBI's patent proteinsNo added restrictions. Attribution expected
  • GenBank's patent filesNo restrictions from NCBI. Submitters may hold rights
  • HGNCCC0
  • NCBI GenePublic domain (US government)
  • NIH's project files and grant-paper linksPublic domain
  • Open TargetsCC0 1.0
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ClinicalTrials.gov's posted results, through CTTI's AACTClinicalTrials.gov data via Aggregate Analysis of ClinicalTrials.gov (AACT), CTTI. CTTI makes its materials freely available and requests acknowledgement. https://aact.ctti-clinicaltrials.org/documentation/299
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • EMBL-EBI's patent proteinsEMBL-EBI's patent protein files from the EPO, JPO, KIPO and USPTO. EMBL-EBI: "EMBL-EBI itself places no additional restrictions on the use or redistribution of the data available via its Data Resources and Tools other than those provided by the original data owners" and "EMBL-EBI expects attribution (e.g. in publications, services or products) for the use of any of its Data Resources and Tools." Read on 11 Nov 2026 at https://www.ebi.ac.uk/about/terms-of-use
  • GenBank's patent filesGenBank's patent files. NCBI: "NCBI places no restrictions on the use or distribution of the GenBank data. However, some submitters may claim patent, copyright, or other intellectual property rights in all or a portion of the data they have submitted." Read on 11 Nov 2026 at https://www.ncbi.nlm.nih.gov/genbank/about/
  • HGNCCC0: https://www.genenames.org/about/license/
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH's project files and grant-paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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