Gene · 51441
Papers3,768
Newest first. Swiss-Prot's references are curated; PubTator's text mining finds papers that name the gene, not always ones about it.
41 to 60 of 3,768
Download this page as JSON| Title | Journal | Year | PMID | Found by | Source |
|---|---|---|---|---|---|
| METTL3/FTO drives ferroptosis in colorectal cancer by targeting m6A modification of MUC1 mRNA and YTHDF2-dependent SLC7A11/GSH pathway. | J Gastroenterol | 2026 | 42384087 | PubTator, text mining | PubMed |
| m6A-FORM: An m6A-focused Foundation Model for Decoding m6A Regulatory Function. | ArXiv | 2026 | 42328286 | PubTator, text mining | PubMed |
| Neuronal YTHDF2 suppresses innate immune activation in Aβ pathology by promoting m6A-dependent decay of cytosolic mitochondrial mRNAs. | Sci Adv | 2026 | 42319929 | PubTator, text mining | PubMed |
| Expression of YTHDF1 and YTHDF2 in lung squamous cell carcinoma and their correlation with PD-L1. | J Thorac Dis | 2026 | 42306733 | PubTator, text mining | PubMed |
| Hypoxia-induced m6A demethylation promotes hepatocellular carcinoma progression by preventing the degradation of Gal-1 mRNA. | Genes Dis | 2026 | 42290655 | PubTator, text mining | PubMed |
| Enhancing targeted strategies for cancer immunotherapy by elucidating mRNA processing mechanisms. | Front Immunol | 2026 | 42266677 | PubTator, text mining | PubMed |
| FTO-mediated m6A demethylation of BCL6 promotes gastric cancer progression by suppressing ferroptosis. | RNA Biol | 2026 | 42262009 | PubTator, text mining | PubMed |
| ALKBH5 promotes the progression of cisplatin-resistant oral squamous cell carcinoma by regulating FOXA1 expression via m6A RNA methylation : Funding. | Daru | 2026 | 42251238 | PubTator, text mining | PubMed |
| Mechanism of METTL3 promoting rituximab resistance via β-catenin/MYC axis in double-expressor diffuse large B cell lymphoma. | Pathol Res Pract | 2026 | 42242087 | PubTator, text mining | PubMed |
| Landscape of N6-methyladenosine (m6A) methylation in porcine cells identifies candidate genes associated with porcine epidemic diarrhea virus infection. | Front Microbiol | 2026 | 42232905 | PubTator, text mining | PubMed |
| ALKBH5 aggravates scarring after glaucoma surgery via m6A-YTHDF2-mediated NLRP3 mRNA stabilization. | Cell Death Differ | 2026 | 42218253 | PubTator, text mining | PubMed |
| Myeloid-specific Ythdf2 deletion alleviates acute lung injury by attenuating macrophage inflammation. | Mol Immunol | 2026 | 42217284 | PubTator, text mining | PubMed |
| Correction: ELF1-mediated transactivation of METTL3/YTHDF2 promotes nucleus pulposus cell senescence via m6A-dependent destabilization of E2F3 mRNA in intervertebral disc degeneration. | Cell Death Discov | 2026 | 42215439 | PubTator, text mining | PubMed |
| METTL3-catalyzed m6A modification of NSUN4 mediates m5C modification of SLC2A3 mRNA to regulate PDGF-BB-induced proliferation, hyaluronan production and glycolysis in human orbital fibroblasts. | J Bioenerg Biomembr | 2026 | 42209904 | PubTator, text mining | PubMed |
| SARS-CoV-2 N Protein Hijacks the m6A Reader YTHDF2 to Suppress Antiviral Gene Expression. | Viruses | 2026 | 42198699 | PubTator, text mining | PubMed |
METTL3 depletion mitigates hypoxic pulmonary arterial smooth muscle cell over-proliferation via YTHDF2-dependent regulation of FOXO1 mRNA N6-methyladenosine modification: a potential mechanism of hypoxic pulmonary hypertension. | Life Sci | 2026 | 42162687 | PubTator, text mining | PubMed |
| Hypoxia-mediated epigenetic silencing of YTHDF2 promotes endometriosis progression via the NFE2L1-NF-κB axis. | Int J Biol Macromol | 2026 | 42162597 | PubTator, text mining | PubMed |
| YTHDF2-driven degradation of BARX2 mRNA accelerates osteoarthritis progression by enhancing the TOP2A/WNT/β-Catenin axis. | Commun Biol | 2026 | 42162214 | PubTator, text mining | PubMed |
| The m6A reader protein YTHDF2 facilitates HTLV-1 infectious and mitotic propagation by stabilizing Tax RNA. | J Virol | 2026 | 42138387 | PubTator, text mining | PubMed |
| Anzi Tiaochong Tang inhibits trophoblast ferroptosis via the METTL14/m6A/SLC39A14 axis in recurrent spontaneous abortion. | Front Pharmacol | 2026 | 42137328 | PubTator, text mining | PubMed |
gene-search-2026-11-11-0.2.0
Sources 12
- ClinicalTrials.gov's posted results, through CTTI's AACTFreely available. Acknowledgement requested
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- EMBL-EBI's patent proteinsNo added restrictions. Attribution expected
- GenBank's patent filesNo restrictions from NCBI. Submitters may hold rights
- HGNCCC0
- NCBI GenePublic domain (US government)
- NIH's project files and grant-paper linksPublic domain
- Open TargetsCC0 1.0
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ClinicalTrials.gov's posted results, through CTTI's AACTClinicalTrials.gov data via Aggregate Analysis of ClinicalTrials.gov (AACT), CTTI. CTTI makes its materials freely available and requests acknowledgement. https://aact.ctti-clinicaltrials.org/documentation/299
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- EMBL-EBI's patent proteinsEMBL-EBI's patent protein files from the EPO, JPO, KIPO and USPTO. EMBL-EBI: "EMBL-EBI itself places no additional restrictions on the use or redistribution of the data available via its Data Resources and Tools other than those provided by the original data owners" and "EMBL-EBI expects attribution (e.g. in publications, services or products) for the use of any of its Data Resources and Tools." Read on 11 Nov 2026 at https://www.ebi.ac.uk/about/terms-of-use
- GenBank's patent filesGenBank's patent files. NCBI: "NCBI places no restrictions on the use or distribution of the GenBank data. However, some submitters may claim patent, copyright, or other intellectual property rights in all or a portion of the data they have submitted." Read on 11 Nov 2026 at https://www.ncbi.nlm.nih.gov/genbank/about/
- HGNCCC0: https://www.genenames.org/about/license/
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH's project files and grant-paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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