ConductScience.org, home

Paper · 16267828

Phosphodiesterase-Ialpha/autotaxin (PD-Ialpha/ATX): a multifunctional protein involved in central nervous system development and disease.

Dennis J and 2 others. J Neurosci Res, 2005. Cited by 42 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

Download JSON
Cite this record

ConductScience Library. Phosphodiesterase-Ialpha/autotaxin (PD-Ialpha/ATX): a multifunctional protein involved in central nervous system development and disease. Papers, record 16267828. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/16267828

On this page

Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Phosphodiesterase-Ialpha/autotaxin (PD-Ialpha/ATX): a multifunctional protein involved in central nervous system development and disease.
PubMed
Journal
J Neurosci Res
PubMed
Year
2005
PubMed
Publication types
Journal Article, Research Support, N.I.H., Extramural, Research Support, Non-U.S. Gov't, Review
PubMed
Citations, by iCite
42
iCite
Relative citation ratio, by iCite
0.93
iCite
Revised in PubMed
2022-03-09
PubMed

Authors3

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Dennis JJameel
2Nogaroli LLuciana
3Fuss BBabette

Source PubMed

Topics6

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Central Nervous SystemMajor topic
Central Nervous System DiseasesMajor topic
Multienzyme ComplexesMajor topic
Phosphodiesterase IMajor topic
Animals
Humans

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References0

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

None in this release.

Cited by28

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 42 citations.

PMIDTitleJournalYearIn PapersSource
34,620,987The enpp4 ectonucleotidase regulates kidney patterning signalling networks in Xenopus embryos.Commun Biol2021https://conductscience.org/library/papers/34620987PubMed
33,151,452Lysophosphatidic Acid Signalling in Nervous System Development and Function.Neuromolecular Med2021https://conductscience.org/library/papers/33151452PubMed
32,240,164Genetic deletion of Autotaxin from CD11b+ cells decreases the severity of experimental autoimmune encephalomyelitis.PLoS One2020https://conductscience.org/library/papers/32240164PubMed
31,652,837Deregulated Lysophosphatidic Acid Metabolism and Signaling in Liver Cancer.Cancers (Basel)2019https://conductscience.org/library/papers/31652837PubMed
29,377,124Purinergic signaling in oligodendrocyte development and function.J Neurochem2018https://conductscience.org/library/papers/29377124PubMed
28,521,702Role of Ectonucleotidases in Synapse Formation During Brain Development: Physiological and Pathological Implications.Curr Neuropharmacol2019https://conductscience.org/library/papers/28521702PubMed
27,731,925G-protein-coupled receptor signaling and neural tube closure defects.Birth Defects Res2017https://conductscience.org/library/papers/27731925PubMed
27,659,560HIV-1 Tat Inhibits Autotaxin Lysophospholipase D Activity and Modulates Oligodendrocyte Differentiation.ASN Neuro2016https://conductscience.org/library/papers/27659560PubMed
26,966,392Lysophospholipid receptors LPA1-3 are not required for the inhibitory effects of LPA on mouse retinal growth cones.Eye Brain2010https://conductscience.org/library/papers/26966392PubMed
26,269,646The Autotaxin-Lysophosphatidic Acid Axis Modulates Histone Acetylation and Gene Expression during Oligodendrocyte Differentiation.J Neurosci2015https://conductscience.org/library/papers/26269646PubMed

1 to 10 of 28

See all 28 cited by

Genes1

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

GeneNCBI Gene IDLinked byIn Genes
ENPP25,168PubTator's text mininghttps://conductscience.org/library/genes/5168

Source NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

For software

Read this paper from software.

The record and each of its lists are JSON, twenty rows a page. No key.

GET https://conductscience.org/api/papers/v1/16267828

GET https://conductscience.org/api/papers/v1/16267828/cited_by?page=2