Paper264694
A soluble ATP-dependent proteolytic system responsible for the degradation of abnormal proteins in reticulocytes.
Etlinger JD and 1 other. Proc Natl Acad Sci U S A, 1977. Cited by 515 papers, by iCite.
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ConductScience Library. A soluble ATP-dependent proteolytic system responsible for the degradation of abnormal proteins in reticulocytes. Papers, record 264694. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/264694
Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- A soluble ATP-dependent proteolytic system responsible for the degradation of abnormal proteins in reticulocytes.
- PubMed
Topics16
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Adenosine Triphosphate | Major topic |
| Blood Proteins | Major topic |
| Peptide Hydrolases | Major topic |
| Peptides | Major topic |
| Reticulocytes | Major topic |
| Animals | |
| Cell-Free System | |
| Globins | |
| Phenanthrolines | |
| Protease Inhibitors | |
| Puromycin | |
| Rabbits | |
| Solubility | |
| Sulfhydryl Reagents | |
| Tosylphenylalanyl Chloromethyl Ketone | |
| Valine |
Source PubMed
References18
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 14,240,649 | CHARACTERISTICS OF THE INHIBITION OF HEMOGLOBIN SYNTHESIS IN RABBIT RETICULOCYTES BY THREO-ALPHA-AMINO-BETA-CHLOROBUTYRIC ACID. | Biochim Biophys Acta | 1964 | https://conductscience.org/library/papers/14240649 | PubMed |
| 2 | 1,112,824 | Effect of insulin on protein turnover in heart muscle. | J Biol Chem | 1975 | https://conductscience.org/library/papers/1112824 | PubMed |
| 3 | 4,554,917 | Effects of protease inhibitors on protein breakdown in Escherichia coli. | J Biol Chem | 1972 | https://conductscience.org/library/papers/4554917 | PubMed |
| 4 | 4,560,693 | Correlation between rates of degradation of bacterial proteins in vivo and their sensitivity to proteases. | Proc Natl Acad Sci U S A | 1972 | https://conductscience.org/library/papers/4560693 | PubMed |
| 5 | 4,144,403 | On the enzymology of amino acid transport. | Science | 1973 | https://conductscience.org/library/papers/4144403 | PubMed |
| 6 | 4,369,108 | Rapid degradation of puromycyl peptides in hepatoma cells and reticulocytes. | FEBS Lett | 1974 | https://conductscience.org/library/papers/4369108 | PubMed |
| 7 | 4,310,604 | Control of gluconeogenesis from amino acids in the perfused rat liver. | J Biol Chem | 1969 | https://conductscience.org/library/papers/4310604 | PubMed |
| 8 | 182,157 | Selective control of the degradation of normal and aberrant proteins in Reuber H35 hepatoma cells. | Biochem J | 1976 | https://conductscience.org/library/papers/182157 | PubMed |
| 9 | 4,604,628 | Intracellular protein degradation in mammalian and bacterial cells. | Annu Rev Biochem | 1974 | https://conductscience.org/library/papers/4604628 | PubMed |
| 10 | 4,629,422 | An adenosine triphosphate-dependent stabilization of proteolytic activity in heterolysosomes. Evidence for a proton pump. | Biochem J | 1972 | https://conductscience.org/library/papers/4629422 | PubMed |
| 11 | 890 | ATP analogs. | Adv Enzymol Relat Areas Mol Biol | 1975 | https://conductscience.org/library/papers/890 | PubMed |
| 12 | 4,556,303 | Urea amidolyase. I. Properties of the enzyme from Candida utilis. | J Biol Chem | 1972 | https://conductscience.org/library/papers/4556303 | PubMed |
| 13 | 4,562,805 | Turnover of intracellular proteins. | Annu Rev Microbiol | 1972 | https://conductscience.org/library/papers/4562805 | PubMed |
| 14 | 786,161 | Intracellular protein degradation in mammalian and bacterial cells: Part 2. | Annu Rev Biochem | 1976 | https://conductscience.org/library/papers/786161 | PubMed |
| 15 | 805,138 | Removal of Z-lines and alpha-actinin from isolated myofibrils by a calcium-activated neutral protease. | J Biol Chem | 1975 | https://conductscience.org/library/papers/805138 | PubMed |
| 16 | 1,089,651 | Degradation of abnormal proteins in Escherichia coli. Formation of protein inclusions in cells exposed to amino acid analogs. | J Biol Chem | 1975 | https://conductscience.org/library/papers/1089651 | PubMed |
| 17 | 931,997 | Protein degradation in human fibroblasts (WI-38). Effects of aging, viral transformation, and amino acid analogs. | J Biol Chem | 1976 | https://conductscience.org/library/papers/931997 | PubMed |
| 18 | 5,260,144 | Chain termination: a test for a possible explanation of thalassemia. | Ann N Y Acad Sci | 1969 | https://conductscience.org/library/papers/5260144 | PubMed |
Cited by157
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 515 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 42,132,104 | A special issue of Essays in Biochemistry on proteasome and protein degradation. | Essays Biochem | 2026 | https://conductscience.org/library/papers/42132104 | PubMed |
| 41,780,752 | Plasticity in the structure and assembly of proteasomes. | J Biol Chem | 2026 | https://conductscience.org/library/papers/41780752 | PubMed |
| 41,655,698 | Molecular mechanisms of mitochondrial AAA+ proteases. | J Biol Chem | 2026 | https://conductscience.org/library/papers/41655698 | PubMed |
| 41,436,460 | Targeting ubiquitination in disease and therapy. | Signal Transduct Target Ther | 2025 | https://conductscience.org/library/papers/41436460 | PubMed |
| 40,867,639 | Extracellular Vesicles Derived from Breast Cancer Cells: Emerging Biomarkers of Tumor Progression and Metastasis. | Biomolecules | 2025 | https://conductscience.org/library/papers/40867639 | PubMed |
| 40,122,174 | Mapping the structural heterogeneity of Pup ligase PafA using H/D exchange mass spectrometry. | J Biol Chem | 2025 | https://conductscience.org/library/papers/40122174 | PubMed |
| 39,799,471 | Comparative Analysis of Acquired Resistance to Bortezomib in Prostate Cancer Cells Using Proteomic and Bioinformatic Tools. | J Cell Mol Med | 2025 | https://conductscience.org/library/papers/39799471 | PubMed |
| 39,513,615 | Selective protein degradation through chaperone‑mediated autophagy: Implications for cellular homeostasis and disease (Review). | Mol Med Rep | 2025 | https://conductscience.org/library/papers/39513615 | PubMed |
| 39,201,634 | Different Strategies to Overcome Resistance to Proteasome Inhibitors-A Summary 20 Years after Their Introduction. | Int J Mol Sci | 2024 | https://conductscience.org/library/papers/39201634 | PubMed |
| 37,759,726 | Structure, Function, and Allosteric Regulation of the 20S Proteasome by the 11S/PA28 Family of Proteasome Activators. | Biomolecules | 2023 | https://conductscience.org/library/papers/37759726 | PubMed |
| 37,508,510 | Targeted Protein Degradation: Principles and Applications of the Proteasome. | Cells | 2023 | https://conductscience.org/library/papers/37508510 | PubMed |
| 36,531,183 | Red blood cell proteomics reveal remnant protein biosynthesis and folding pathways in PIEZO1-related hereditary xerocytosis. | Front Physiol | 2022 | https://conductscience.org/library/papers/36531183 | PubMed |
| 36,237,424 | The emerging role of Deubiquitinases (DUBs) in parasites: A foresight review. | Front Cell Infect Microbiol | 2022 | https://conductscience.org/library/papers/36237424 | PubMed |
| 36,177,203 | Cystargolide-based amide and ester Pz analogues as proteasome inhibitors and anti-cancer agents. | R Soc Open Sci | 2022 | https://conductscience.org/library/papers/36177203 | PubMed |
| 35,631,358 | Covalent Inhibition of the Human 20S Proteasome with Homobelactosin C Inquired by QM/MM Studies. | Pharmaceuticals (Basel) | 2022 | https://conductscience.org/library/papers/35631358 | PubMed |
| 35,501,388 | POH1/Rpn11/PSMD14: a journey from basic research in fission yeast to a prognostic marker and a druggable target in cancer cells. | Br J Cancer | 2022 | https://conductscience.org/library/papers/35501388 | PubMed |
| 35,330,789 | Expanding the MuRF1 Universe with Quantitative Ubiquitylomics. | Function (Oxf) | 2021 | https://conductscience.org/library/papers/35330789 | PubMed |
| 34,792,731 | Carrying Excess Baggage Can Slowdown Life: Protein Clearance Machineries That Go Awry During Aging and the Relevance of Maintaining Them. | Mol Neurobiol | 2022 | https://conductscience.org/library/papers/34792731 | PubMed |
| 34,638,816 | Using the Zebrafish as a Genetic Model to Study Erythropoiesis. | Int J Mol Sci | 2021 | https://conductscience.org/library/papers/34638816 | PubMed |
| 33,905,676 | The proteasome and its role in the nervous system. | Cell Chem Biol | 2021 | https://conductscience.org/library/papers/33905676 | PubMed |
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- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
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- Comments and corrections
- Genes
- Drugs
- Data
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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