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Paper · 30833752

References60

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
124,115,766Exploiting the bad eating habits of Ras-driven cancers.Genes Dev2013https://conductscience.org/library/papers/24115766PubMed
221,127,245Autophagy and metabolism.Science2010https://conductscience.org/library/papers/21127245PubMed
322,081,344Antibody validation by Western blotting.Methods Mol Biol2012https://conductscience.org/library/papers/22081344PubMed
426,725,216Long-Term ERK Inhibition in KRAS-Mutant Pancreatic Cancer Is Associated with MYC Degradation and Senescence-like Growth Suppression.Cancer Cell2016https://conductscience.org/library/papers/26725216PubMed
528,446,508Enrichment of PI3K-AKT-mTOR Pathway Activation in Hepatic Metastases from Breast Cancer.Clin Cancer Res2017https://conductscience.org/library/papers/28446508PubMed
629,467,321Development of MK-8353, an orally administered ERK1/2 inhibitor, in patients with advanced solid tumors.JCI Insight2018https://conductscience.org/library/papers/29467321PubMed
730,833,748Protective autophagy elicited by RAF→MEK→ERK inhibition suggests a treatment strategy for RAS-driven cancers.Nat Med2019https://conductscience.org/library/papers/30833748PubMed
825,833,948Pharmacological inhibition of ULK1 kinase blocks mammalian target of rapamycin (mTOR)-dependent autophagy.J Biol Chem2015https://conductscience.org/library/papers/25833948PubMed
926,732,095Oncogenic BRAF Deletions That Function as Homodimers and Are Sensitive to Inhibition by RAF Dimer Inhibitor LY3009120.Cancer Discov2016https://conductscience.org/library/papers/26732095PubMed
1021,406,549Pancreatic cancers require autophagy for tumor growth.Genes Dev2011https://conductscience.org/library/papers/21406549PubMed
1129,229,669KRAS: The Critical Driver and Therapeutic Target for Pancreatic Cancer.Cold Spring Harb Perspect Med2018https://conductscience.org/library/papers/29229669PubMed
1223,614,898Discovery of a novel ERK inhibitor with activity in models of acquired resistance to BRAF and MEK inhibitors.Cancer Discov2013https://conductscience.org/library/papers/23614898PubMed
1322,419,781IsoCor: correcting MS data in isotope labeling experiments.Bioinformatics2012https://conductscience.org/library/papers/22419781PubMed
1425,119,024Oncogene ablation-resistant pancreatic cancer cells depend on mitochondrial function.Nature2014https://conductscience.org/library/papers/25119024PubMed
1515,169,837LC3, GABARAP and GATE16 localize to autophagosomal membrane depending on form-II formation.J Cell Sci2004https://conductscience.org/library/papers/15169837PubMed
168,799,277Correction of 13C mass isotopomer distributions for natural stable isotope abundance.J Mass Spectrom1996https://conductscience.org/library/papers/8799277PubMed
1728,302,824Drugging RAS: Know the enemy.Science2017https://conductscience.org/library/papers/28302824PubMed
1826,468,985Functional signaling pathway analysis of lung adenocarcinomas identifies novel therapeutic targets for KRAS mutant tumors.Oncotarget2015https://conductscience.org/library/papers/26468985PubMed
1927,379,200(13)C MRS and LC-MS Flux Analysis of Tumor Intermediary Metabolism.Front Oncol2016https://conductscience.org/library/papers/27379200PubMed
2030,759,290Society of Surgical Oncology 72nd Annual Cancer Symposium.Ann Surg Oncol2019https://conductscience.org/library/papers/30759290PubMed

papers-2026-11-12-0.1.0

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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