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Paper · 319458

A mutant of Escherichia coli showing constitutive expression of the lysogenic induction and error-prone DNA repair pathways.

Mount DW. Proc Natl Acad Sci U S A, 1977. Cited by 238 papers, by iCite.

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ConductScience Library. A mutant of Escherichia coli showing constitutive expression of the lysogenic induction and error-prone DNA repair pathways. Papers, record 319458. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/319458

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
A mutant of Escherichia coli showing constitutive expression of the lysogenic induction and error-prone DNA repair pathways.
PubMed
Journal
Proc Natl Acad Sci U S A
PubMed
Year
1977
PubMed
Publication types
Journal Article, Research Support, U.S. Gov't, Non-P.H.S.
PubMed
Citations, by iCite
238
iCite
Relative citation ratio, by iCite
5.71
iCite
Revised in PubMed
2022-02-24
PubMed

Authors1

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Mount DWD W

Source PubMed

Topics11

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
ColiphagesMajor topic
DNA RepairMajor topic
Escherichia coliMajor topic
GenesMajor topic
LysogenyMajor topic
MutationMajor topic
Virus ReplicationMajor topic
Enzyme Repression
Genes, Regulator
Mitomycins
Ultraviolet Rays

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References25

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
1765,722recA+-dependent inactivation of the lambda repressor in Escherichia coli lysogens by gamma-radiation and by tif expression.Mol Gen Genet1975https://conductscience.org/library/papers/765722PubMed
216,591,470ISOLATION OF THE lambda PHAGE REPRESSOR.Proc Natl Acad Sci U S A1967https://conductscience.org/library/papers/16591470PubMed
3934,047A method for the isolation of phage mutants altered in their response ot lysogenic induction.Mol Gen Genet1976https://conductscience.org/library/papers/934047PubMed
41,090,931Proteolytic cleavage of bacteriophage lambda repressor in induction.Proc Natl Acad Sci U S A1975https://conductscience.org/library/papers/1090931PubMed
51,094,463Model for regulation of Escherichia coli DNA repair functions.Proc Natl Acad Sci U S A1975https://conductscience.org/library/papers/1094463PubMed
61,101,265Inducible error-prone repair in Escherichia coli.Proc Natl Acad Sci U S A1975https://conductscience.org/library/papers/1101265PubMed
71,107,802Prophage induction and cell division in E. coli. III. Mutations sfiA and sfiB restore division in tif and lon strains and permit the expression of mutator properties of tif.Mol Gen Genet1975https://conductscience.org/library/papers/1107802PubMed
84,205,905Recombination deficient mutants of E. coli and other bacteria.Annu Rev Genet1973https://conductscience.org/library/papers/4205905PubMed
94,267,058Lysogenic induction.Prog Nucleic Acid Res Mol Biol1973https://conductscience.org/library/papers/4267058PubMed
104,343,824Dominant mutations (lex) in Escherichia coli K-12 which affect radiation sensitivity and frequency of ultraviolet lght-induced mutations.J Bacteriol1972https://conductscience.org/library/papers/4343824PubMed

1 to 10 of 25

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Cited by141

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 238 citations.

PMIDTitleJournalYearIn PapersSource
40,892,829Conservation and divergence of ciprofloxacin persister survival mechanisms between Pseudomonas aeruginosa and Escherichia coli.PLoS Genet2025https://conductscience.org/library/papers/40892829PubMed
38,564,687Differential impacts of DNA repair machinery on fluoroquinolone persisters with different chromosome abundances.mBio2024https://conductscience.org/library/papers/38564687PubMed
35,208,744Fluoroquinolone Persistence in Escherichia coli Requires DNA Repair despite Differing between Starving Populations.Microorganisms2022https://conductscience.org/library/papers/35208744PubMed
27,536,734Systematically Altering Bacterial SOS Activity under Stress Reveals Therapeutic Strategies for Potentiating Antibiotics.mSphere2016https://conductscience.org/library/papers/27536734PubMed
26,330,511Stationary-Phase Persisters to Ofloxacin Sustain DNA Damage and Require Repair Systems Only during Recovery.mBio2015https://conductscience.org/library/papers/26330511PubMed
25,824,947Suppression of the E. coli SOS response by dNTP pool changes.Nucleic Acids Res2015https://conductscience.org/library/papers/25824947PubMed
23,276,924A proposal: Source of single strand DNA that elicits the SOS response.Front Biosci (Landmark Ed)2013https://conductscience.org/library/papers/23276924PubMed
23,144,626Mechanisms employed by Escherichia coli to prevent ribonucleotide incorporation into genomic DNA by Pol V.PLoS Genet2012https://conductscience.org/library/papers/23144626PubMed
22,843,848Factors limiting SOS expression in log-phase cells of Escherichia coli.J Bacteriol2012https://conductscience.org/library/papers/22843848PubMed
22,439,817Identification of genes expressed in cultures of E. coli lysogens carrying the Shiga toxin-encoding prophage Φ24B.BMC Microbiol2012https://conductscience.org/library/papers/22439817PubMed

1 to 10 of 141

See all 141 cited by

Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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