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Paper · 35856444

References43

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
131,489,377Combinatorial morphogenetic and mechanical cues to mimic bone development for defect repair.Sci Adv2019https://conductscience.org/library/papers/31489377PubMed
216,314,833Frequent epigenetic silencing of the bone morphogenetic protein 2 gene through methylation in gastric carcinomas.Oncogene2006https://conductscience.org/library/papers/16314833PubMed
319,688,119The Bone Morphogenesis Protein-2 (BMP-2) is associated with progression to metastatic disease in gastric cancer.Cancer Res Treat2008https://conductscience.org/library/papers/19688119PubMed
4221,908Solubilized and insolubilized bone morphogenetic protein.Proc Natl Acad Sci U S A1979https://conductscience.org/library/papers/221908PubMed
533,275,394Is Use of BMP-2 Associated with Tumor Growth and Osteoblastic Differentiation in Murine Models of Osteosarcoma?Clin Orthop Relat Res2020https://conductscience.org/library/papers/33275394PubMed
610,910,126Application of efficient and specific gene transfer systems and organ culture techniques for the elucidation of mechanisms of epithelial-mesenchymal interaction in the developing gut.Dev Growth Differ2000https://conductscience.org/library/papers/10910126PubMed
712,860,278Aberrant signaling of TGF-beta1 by the mutant Smad4 in gastric cancer cells.Cancer Lett2003https://conductscience.org/library/papers/12860278PubMed
830,944,675Epidemiology of gastric cancer: global trends, risk factors and prevention.Prz Gastroenterol2019https://conductscience.org/library/papers/30944675PubMed
919,835,871BMP2 accelerates the motility and invasiveness of gastric cancer cells via activation of the phosphatidylinositol 3-kinase (PI3K)/Akt pathway.Exp Cell Res2010https://conductscience.org/library/papers/19835871PubMed
1026,256,958Evolution of Gastric Cancer Treatment: From the Golden Age of Surgery to an Era of Precision Medicine.Yonsei Med J2015https://conductscience.org/library/papers/26256958PubMed
1125,698,539C23 protein meditates bone morphogenetic protein-2-mediated EMT via up-regulation of Erk1/2 and Akt in gastric cancer.Med Oncol2015https://conductscience.org/library/papers/25698539PubMed
123,201,241Novel regulators of bone formation: molecular clones and activities.Science1988https://conductscience.org/library/papers/3201241PubMed
1330,747,230Bile acids promote gastric intestinal metaplasia by upregulating CDX2 and MUC2 expression via the FXR/NF-κB signalling pathway.Int J Oncol2019https://conductscience.org/library/papers/30747230PubMed
1433,234,056Combination of BMP2 and EZH2 Inhibition to Stimulate Osteogenesis in a 3D Bone Reconstruction Model.Tissue Eng Part A2021https://conductscience.org/library/papers/33234056PubMed
1515,003,517BMP-2 modulates the proliferation and differentiation of normal and cancerous gastric cells.Biochem Biophys Res Commun2004https://conductscience.org/library/papers/15003517PubMed
1629,180,570Perturbation of canonical and non-canonical BMP signaling affects migration, polarity and dendritogenesis of mouse cortical neurons.Development2018https://conductscience.org/library/papers/29180570PubMed
1731,298,369Effects of microRNA-378 on epithelial-mesenchymal transition, migration, invasion and prognosis in gastric carcinoma by targeting BMP2.Eur Rev Med Pharmacol Sci2019https://conductscience.org/library/papers/31298369PubMed
1821,570,392Metastatic function of BMP-2 in gastric cancer cells: the role of PI3K/AKT, MAPK, the NF-κB pathway, and MMP-9 expression.Exp Cell Res2011https://conductscience.org/library/papers/21570392PubMed
1920,346,678Polycomb group protein-mediated repression of transcription.Trends Biochem Sci2010https://conductscience.org/library/papers/20346678PubMed
2020,804,967Polycomb group proteins: multi-faceted regulators of somatic stem cells and cancer.Cell Stem Cell2010https://conductscience.org/library/papers/20804967PubMed

papers-2026-11-12-0.1.0

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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