Paper · 39852234

High FGF18 expression levels predict poor prognosis in endometrial carcinoma patients and promote tumor growth and metastasis.

Lei ZY and 6 others. J Int Med Res, 2025. Cited by 0 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. High FGF18 expression levels predict poor prognosis in endometrial carcinoma patients and promote tumor growth and metastasis. Papers, record 39852234. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/39852234

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: High FGF18 expression levels predict poor prognosis in endometrial carcinoma patients and promote tumor growth and metastasis.; PubMed
Journal: J Int Med Res; PubMed
Year: 2025; PubMed
Publication types: Journal Article; PubMed
PubMed: https://pubmed.ncbi.nlm.nih.gov/39852234/; PubMed
DOI: https://doi.org/10.1177/03000605241311402; PubMed
PMC: https://pmc.ncbi.nlm.nih.gov/articles/PMC11760137/; PubMed
The journal: https://conductscience.org/library/journal-index/0346411; PubMed
Citations, by iCite: 0; iCite
Revised in PubMed: 2025-05-23; PubMed

Authors7

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename	ORCID iD
1	Lei ZY	Zheng-Yao
2	Wu J	Jian	https://orcid.org/0000-0003-2016-4275
3	Zhang BH	Bao-Hua
4	Xiang W	Wei
5	Wang M	Min
6	Li BB	Bin-Bin
7	Dai MC	Ming-Cheng	https://orcid.org/0000-0002-9987-3244

Source PubMed

Topics17

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
Endometrial Neoplasms	Major topic
Fibroblast Growth Factors	Major topic
Biomarkers, Tumor	Major topic
Humans
Female
Prognosis
Animals
Cell Proliferation
Cell Movement
Gene Expression Regulation, Neoplastic
Mice
Middle Aged
Cell Line, Tumor
Retrospective Studies
Neoplasm Invasiveness
Aged
Neoplasm Metastasis

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References34

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

Order	PMID	Title	Journal	Year	In Papers	Source
1	30,082,912	FGF18, a prominent player in FGF signaling, promotes gastric tumorigenesis through autocrine manner and is negatively regulated by miR-590-5p.	Oncogene	2019	https://conductscience.org/library/papers/30082912	PubMed
2	12,966,355	Simple, inexpensive method for automating tissue microarray production provides enhanced microarray reproducibility.	Appl Immunohistochem Mol Morphol	2003	https://conductscience.org/library/papers/12966355	PubMed
3	17,890,768	FGF18 in colorectal tumour cells: autocrine and paracrine effects.	Carcinogenesis	2008	https://conductscience.org/library/papers/17890768	PubMed
4	35,636,624	Deubiquitinase USP8 increases ID1 stability and promotes esophageal squamous cell carcinoma tumorigenesis.	Cancer Lett	2022	https://conductscience.org/library/papers/35636624	PubMed
5	30,332,563	Molecular Genetics of Endometrial Carcinoma.	Annu Rev Pathol	2019	https://conductscience.org/library/papers/30332563	PubMed
6	28,469,794	Silencing of CXCR4 and CXCR7 expression by RNA interference suppresses human endometrial carcinoma growth in vivo.	Am J Transl Res	2017	https://conductscience.org/library/papers/28469794	PubMed
7	9,742,123	FGF-18, a novel member of the fibroblast growth factor family, stimulates hepatic and intestinal proliferation.	Mol Cell Biol	1998	https://conductscience.org/library/papers/9742123	PubMed
8	26,427,667	FGF18 as a potential biomarker in serous and mucinous ovarian tumors.	Tumour Biol	2016	https://conductscience.org/library/papers/26427667	PubMed
9	37,436,087	Ferroptosis, Necroptosis, and Pyroptosis in Gastrointestinal Cancers: The Chief Culprits of Tumor Progression and Drug Resistance.	Adv Sci (Weinh)	2023	https://conductscience.org/library/papers/37436087	PubMed
10	26,872,609	The prognostic value of stromal and epithelial periostin expression in human breast cancer: correlation with clinical pathological features and mortality outcome.	BMC Cancer	2016	https://conductscience.org/library/papers/26872609	PubMed

1 to 10 of 34

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Cited by0

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 0 citations.

None in this release.

Genes1

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

Gene	NCBI Gene ID	Linked by	In Genes
FGF18	8,817	PubTator's text mining	https://conductscience.org/library/genes/8817

Source NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

Sources 11

ChEMBL, through Open TargetsCC BY-SA 3.0
ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

Each source's terms

ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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