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Paper · 40581663

References51

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
118,227,510The T790M mutation in EGFR kinase causes drug resistance by increasing the affinity for ATP.Proc Natl Acad Sci U S A2008https://conductscience.org/library/papers/18227510PubMed
233,062,157Development of a Potent Brain-Penetrant EGFR Tyrosine Kinase Inhibitor against Malignant Brain Tumors.ACS Med Chem Lett2020https://conductscience.org/library/papers/33062157PubMed
336,370,105The STRING database in 2023: protein-protein association networks and functional enrichment analyses for any sequenced genome of interest.Nucleic Acids Res2023https://conductscience.org/library/papers/36370105PubMed
424,120,142The somatic genomic landscape of glioblastoma.Cell2013https://conductscience.org/library/papers/24120142PubMed
529,446,774Proteome-wide identification of ubiquitin interactions using UbIA-MS.Nat Protoc2018https://conductscience.org/library/papers/29446774PubMed
628,379,424Combination therapy with potent PI3K and MAPK inhibitors overcomes adaptive kinome resistance to single agents in preclinical models of glioblastoma.Neuro Oncol2017https://conductscience.org/library/papers/28379424PubMed
723,104,886STAR: ultrafast universal RNA-seq aligner.Bioinformatics2013https://conductscience.org/library/papers/23104886PubMed
831,601,994The great escape: tumour cell plasticity in resistance to targeted therapy.Nat Rev Drug Discov2020https://conductscience.org/library/papers/31601994PubMed
98,845,302Epidermal growth factor ligand-independent, unregulated, cell-transforming potential of a naturally occurring human mutant EGFRvIII gene.Cell Growth Differ1995https://conductscience.org/library/papers/8845302PubMed
1028,791,656EGFR as a Target for Glioblastoma Treatment: An Unfulfilled Promise.CNS Drugs2017https://conductscience.org/library/papers/28791656PubMed
1112,471,243The protein kinase complement of the human genome.Science2002https://conductscience.org/library/papers/12471243PubMed
1222,137,795Mosaic amplification of multiple receptor tyrosine kinase genes in glioblastoma.Cancer Cell2011https://conductscience.org/library/papers/22137795PubMed
1329,975,751PIK3CA missense mutations promote glioblastoma pathogenesis, but do not enhance targeted PI3K inhibition.PLoS One2018https://conductscience.org/library/papers/29975751PubMed
1410,982,861STRING: a web-server to retrieve and display the repeatedly occurring neighbourhood of a gene.Nucleic Acids Res2000https://conductscience.org/library/papers/10982861PubMed
1515,831,234Patient tumor EGFR and PDGFRA gene amplifications retained in an invasive intracranial xenograft model of glioblastoma multiforme.Neuro Oncol2005https://conductscience.org/library/papers/15831234PubMed
1620,040,089A reproducible brain tumour model established from human glioblastoma biopsies.BMC Cancer2009https://conductscience.org/library/papers/20040089PubMed
1720,129,251Integrated genomic analysis identifies clinically relevant subtypes of glioblastoma characterized by abnormalities in PDGFRA, IDH1, EGFR, and NF1.Cancer Cell2010https://conductscience.org/library/papers/20129251PubMed
1825,516,281Moderated estimation of fold change and dispersion for RNA-seq data with DESeq2.Genome Biol2014https://conductscience.org/library/papers/25516281PubMed
1922,020,333Acquired MET expression confers resistance to EGFR inhibition in a mouse model of glioblastoma multiforme.Oncogene2012https://conductscience.org/library/papers/22020333PubMed
2015,221,011The tumor-specific de2-7 epidermal growth factor receptor (EGFR) promotes cells survival and heterodimerizes with the wild-type EGFR.Oncogene2004https://conductscience.org/library/papers/15221011PubMed

papers-2026-11-12-0.1.0

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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