Paper · 42429045
FGFR1 signaling in rheumatoid arthritis: Mechanisms of bone destruction and therapeutic targeting (Review).
Qian C and 7 others. Int J Mol Med, 2026. Cited by 0 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. FGFR1 signaling in rheumatoid arthritis: Mechanisms of bone destruction and therapeutic targeting (Review). Papers, record 42429045. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/42429045
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- FGFR1 signaling in rheumatoid arthritis: Mechanisms of bone destruction and therapeutic targeting (Review).
- PubMed
Topics8
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Arthritis, Rheumatoid | Major topic |
| Signal Transduction | Major topic |
| Receptor, Fibroblast Growth Factor, Type 1 | Major topic |
| Bone and Bones | Major topic |
| Humans | |
| Animals | |
| Osteoclasts | |
| Molecular Targeted Therapy |
Source PubMed
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Trials0
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None in this release.
Comments and corrections0
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None in this release.
References246
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 38,842,387 | Macrophage-derived FGFR1 drives atherosclerosis through PLCγ-mediated activation of NF-κB inflammatory signalling pathway. | Cardiovasc Res | 2024 | https://conductscience.org/library/papers/38842387 | PubMed |
| 2 | 28,923,346 | Role of fibroblast growth factor receptors (FGFR) and FGFR like-1 (FGFRL1) in mesenchymal stromal cell differentiation to osteoblasts and adipocytes. | Mol Cell Endocrinol | 2018 | https://conductscience.org/library/papers/28923346 | PubMed |
| 3 | 11,031,111 | Characterization of a novel protein (FGFRL1) from human cartilage related to FGF receptors. | Genomics | 2000 | https://conductscience.org/library/papers/11031111 | PubMed |
| 4 | 38,395,926 | Receptor-based pharmacophore modeling, molecular docking, synthesis and biological evaluation of novel VEGFR-2, FGFR-1, and BRAF multi-kinase inhibitors. | BMC Chem | 2024 | https://conductscience.org/library/papers/38395926 | PubMed |
| 5 | 36,002,765 | TNFAIP3 mediates FGFR1 activation-induced breast cancer angiogenesis by promoting VEGFA expression and secretion. | Clin Transl Oncol | 2022 | https://conductscience.org/library/papers/36002765 | PubMed |
| 6 | 20,193,003 | Fibroblast-like synoviocytes: key effector cells in rheumatoid arthritis. | Immunol Rev | 2010 | https://conductscience.org/library/papers/20193003 | PubMed |
| 7 | 40,822,517 | Narirutin mitigates inflammatory arthritis and osteoporosis through modulating macrophage phenotype and osteoclastogenesis. | J Orthop Translat | 2025 | https://conductscience.org/library/papers/40822517 | PubMed |
| 8 | 40,846,102 | Knockdown of MiD49 and MiD51 alleviates collagen-induced arthritis and suppresses mitophagy and fatty acid oxidation (FAO) in rheumatoid arthritis fibroblast-like synoviocytes. | Free Radic Biol Med | 2025 | https://conductscience.org/library/papers/40846102 | PubMed |
| 9 | 9,435,295 | Fibroblast growth factor receptor 2 (FGFR2)-mediated reciprocal regulation loop between FGF8 and FGF10 is essential for limb induction. | Development | 1998 | https://conductscience.org/library/papers/9435295 | PubMed |
| 10 | 10,490,103 | Structural basis for FGF receptor dimerization and activation. | Cell | 1999 | https://conductscience.org/library/papers/10490103 | PubMed |
1 to 10 of 246
See all 246 referencesCited by0
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Genes0
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None in this release.
Drugs0
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Data0
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Data dates
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- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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