Paper42683000
Bile acid-axis dysregulation in MASLD/MASH progression: from metabolic mismatch to inflammatory-fibrotic remodeling.
Du Y and 7 others. Front Med (Lausanne), 2026. Cited by 0 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. Bile acid-axis dysregulation in MASLD/MASH progression: from metabolic mismatch to inflammatory-fibrotic remodeling. Papers, record 42683000. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/42683000
Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- Bile acid-axis dysregulation in MASLD/MASH progression: from metabolic mismatch to inflammatory-fibrotic remodeling.
- PubMed
Topics0
NLM's MeSH indexing of the paper, its major topics first.
None in this release.
References93
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 32,362,324 | Niche-Specific Reprogramming of Epigenetic Landscapes Drives Myeloid Cell Diversity in Nonalcoholic Steatohepatitis. | Immunity | 2020 | https://conductscience.org/library/papers/32362324 | PubMed |
| 2 | 39,062,555 | Bile Acids as Emerging Players at the Intersection of Steatotic Liver Disease and Cardiovascular Diseases. | Biomolecules | 2024 | https://conductscience.org/library/papers/39062555 | PubMed |
| 3 | 33,989,548 | Mechanisms and disease consequences of nonalcoholic fatty liver disease. | Cell | 2021 | https://conductscience.org/library/papers/33989548 | PubMed |
| 4 | 42,456,558 | Effects of multistrain probiotic supplementation on hepatic function and anthropometric parameters in patients with metabolic dysfunction-associated steatotic liver disease: a double-blind, randomized controlled trial. | Nutrition | 2026 | https://conductscience.org/library/papers/42456558 | PubMed |
| 5 | 38,512,763 | Breaking the barriers: the role of gut homeostasis in Metabolic-Associated Steatotic Liver Disease (MASLD). | Gut Microbes | 2024 | https://conductscience.org/library/papers/38512763 | PubMed |
| 6 | 33,128,017 | Molecular and cellular mechanisms of liver fibrosis and its regression. | Nat Rev Gastroenterol Hepatol | 2021 | https://conductscience.org/library/papers/33128017 | PubMed |
| 7 | 37,508,557 | Bile Acids, Intestinal Barrier Dysfunction, and Related Diseases. | Cells | 2023 | https://conductscience.org/library/papers/37508557 | PubMed |
| 8 | 20,093,562 | The hepatic response to FGF19 is impaired in patients with nonalcoholic fatty liver disease and insulin resistance. | Am J Physiol Gastrointest Liver Physiol | 2010 | https://conductscience.org/library/papers/20093562 | PubMed |
| 9 | 38,851,997 | EASL-EASD-EASO Clinical Practice Guidelines on the management of metabolic dysfunction-associated steatotic liver disease (MASLD). | J Hepatol | 2024 | https://conductscience.org/library/papers/38851997 | PubMed |
| 10 | 36,052,074 | Bile acids in immunity: Bidirectional mediators between the host and the microbiota. | Front Immunol | 2022 | https://conductscience.org/library/papers/36052074 | PubMed |
| 11 | 35,325,622 | Aldafermin in patients with non-alcoholic steatohepatitis (ALPINE 2/3): a randomised, double-blind, placebo-controlled, phase 2b trial. | Lancet Gastroenterol Hepatol | 2022 | https://conductscience.org/library/papers/35325622 | PubMed |
| 12 | 35,434,590 | Metabolic signatures across the full spectrum of non-alcoholic fatty liver disease. | JHEP Rep | 2022 | https://conductscience.org/library/papers/35434590 | PubMed |
| 13 | 35,798,021 | The prevalence and incidence of NAFLD worldwide: a systematic review and meta-analysis. | Lancet Gastroenterol Hepatol | 2022 | https://conductscience.org/library/papers/35798021 | PubMed |
| 14 | 33,440,159 | Dynamic Shifts in the Composition of Resident and Recruited Macrophages Influence Tissue Remodeling in NASH. | Cell Rep | 2021 | https://conductscience.org/library/papers/33440159 | PubMed |
| 15 | 38,653,239 | Gut symbionts alleviate MASH through a secondary bile acid biosynthetic pathway. | Cell | 2024 | https://conductscience.org/library/papers/38653239 | PubMed |
| 16 | 39,218,222 | Dysglycemia and liver lipid content determine the relationship of insulin resistance with hepatic OXPHOS capacity in obesity. | J Hepatol | 2025 | https://conductscience.org/library/papers/39218222 | PubMed |
| 17 | 40,814,400 | Microbiota-derived bile acid metabolic enzymes and their impacts on host health. | Cell Insight | 2025 | https://conductscience.org/library/papers/40814400 | PubMed |
| 18 | 36,107,630 | Haploinsufficiency of CYP8B1 associates with increased insulin sensitivity in humans. | J Clin Invest | 2022 | https://conductscience.org/library/papers/36107630 | PubMed |
| 19 | 40,344,326 | Conjugated Lithocholic Acid Activates Hepatic TGR5 to Promote Lipotoxicity and MASLD-MASH Transition by Disrupting Carnitine Biosynthesis. | Adv Sci (Weinh) | 2025 | https://conductscience.org/library/papers/40344326 | PubMed |
| 20 | 41,753,757 | The Gut-Liver Axis in MASLD: From Host-Microbiome Crosstalk to Precision Therapeutics. | Microorganisms | 2026 | https://conductscience.org/library/papers/41753757 | PubMed |
Genes3
The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.
| Gene | NCBI Gene ID | Linked by | In Genes | Source |
|---|---|---|---|---|
| FGF19 | 9,965 | PubTator's text mining | https://conductscience.org/library/genes/9965 | NCBI Gene |
| GPBAR1 | 151,306 | PubTator's text mining | https://conductscience.org/library/genes/151306 | NCBI Gene |
| NR1H4 | 9,971 | PubTator's text mining | https://conductscience.org/library/genes/9971 | NCBI Gene |
Data dates
The date of each source this release read.
- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
None in this release
- Grants
- Trials
- Comments and corrections
- Cited by
- Drugs
- Data
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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