Paper · 47876
The mechanism of basophil histamine release induced by antigen and by the calcium ionophore A23187.
Lichtenstein LM. J Immunol, 1975. Cited by 189 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. The mechanism of basophil histamine release induced by antigen and by the calcium ionophore A23187. Papers, record 47876. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/47876
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- The mechanism of basophil histamine release induced by antigen and by the calcium ionophore A23187.
- PubMed
Topics13
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Anti-Bacterial Agents | Major topic |
| Antigens | Major topic |
| Basophils | Major topic |
| Calcimycin | Major topic |
| Histamine Release | Major topic |
| Calcium | |
| Chlorides | |
| Cyclic AMP | |
| Fluorometry | |
| Humans | |
| Lanthanum | |
| Pollen | |
| Prostaglandins |
Source PubMed
Grants0
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Trials0
The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.
None in this release.
Comments and corrections0
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References0
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Cited by34
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 189 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 41,306,684 | Label-free, machine-learning assisted impedance assessment of basophil activation. | Sens Actuators B Chem | 2026 | https://conductscience.org/library/papers/41306684 | PubMed |
| 40,683,570 | The Bruton tyrosine kinase inhibitor acalabrutinib aborts ongoing acute food-induced anaphylactic reactions in humanized mice. | J Allergy Clin Immunol | 2025 | https://conductscience.org/library/papers/40683570 | PubMed |
| 36,926,834 | Antibodies against Poly(ethylene glycol) Activate Innate Immune Cells and Induce Hypersensitivity Reactions to PEGylated Nanomedicines. | ACS Nano | 2023 | https://conductscience.org/library/papers/36926834 | PubMed |
| 24,371,118 | Histamine H₂ receptor signaling × environment interactions determine susceptibility to experimental allergic encephalomyelitis. | FASEB J | 2014 | https://conductscience.org/library/papers/24371118 | PubMed |
| 24,271,572 | Release and exchange of neurotransmitters in synaptosomes: Effects of the ionophore A23187 and of ouabain. | Neurochem Res | 1976 | https://conductscience.org/library/papers/24271572 | PubMed |
| 24,262,755 | IgE receptor-mediated mast-cell renin release. | Am J Pathol | 2014 | https://conductscience.org/library/papers/24262755 | PubMed |
| 18,664,579 | A pH-stabilizing role of voltage-gated proton channels in IgE-mediated activation of human basophils. | Proc Natl Acad Sci U S A | 2008 | https://conductscience.org/library/papers/18664579 | PubMed |
| 7,911,715 | Characterization of the histamine receptors in the guinea-pig lung: evidence for relaxant histamine H3 receptors in the trachea. | Br J Pharmacol | 1994 | https://conductscience.org/library/papers/7911715 | PubMed |
| 7,036,400 | A calcium antagonist, nifedipine, modifies exercise-induced asthma. | Thorax | 1981 | https://conductscience.org/library/papers/7036400 | PubMed |
| 6,776,037 | Platelet-activating factor in anaphylaxis and phagocytosis. I. Release from human peripheral polymorphonuclears and monocytes during the stimulation by ionophore A23187 and phagocytosis but not from degranulating basophils. | Immunology | 1980 | https://conductscience.org/library/papers/6776037 | PubMed |
1 to 10 of 34
See all 34 cited byGenes1
The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.
| Gene | NCBI Gene ID | Linked by | In Genes |
|---|---|---|---|
| IGHE | 3,497 | PubTator's text mining | https://conductscience.org/library/genes/3497 |
Source NCBI Gene
Drugs0
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Data dates
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- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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