Paper68079

Immunosuppressive factor(s) specific for L-glutamic acid50-L-tyrosine50 (GT). I. Production, characterization, and lack of H-2 restriction for activity in recipient strain.

Waltenbaugh C and 3 others. J Immunol, 1977. Cited by 53 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

Cite this record

ConductScience Library. Immunosuppressive factor(s) specific for L-glutamic acid50-L-tyrosine50 (GT). I. Production, characterization, and lack of H-2 restriction for activity in recipient strain. Papers, record 68079. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/68079

Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: Immunosuppressive factor(s) specific for L-glutamic acid50-L-tyrosine50 (GT). I. Production, characterization, and lack of H-2 restriction for activity in recipient strain.; PubMed
Journal: J Immunol; PubMed
Year: 1977; PubMed
Publication types: Journal Article, Research Support, U.S. Gov't, P.H.S.; PubMed
PubMed: https://pubmed.ncbi.nlm.nih.gov/68079/; PubMed
The journal: https://conductscience.org/library/journal-index/2985117R; PubMed
Citations, by iCite: 53; iCite
Relative citation ratio, by iCite: 1.19; iCite
Revised in PubMed: 2017-11-16; PubMed

Authors4

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename
1	Waltenbaugh C	C
2	Debré P	P
3	Thèze J	J
4	Benacerraf B	B

Source PubMed

Topics15

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
Antibody Formation	Major topic
Epitopes	Major topic
Glutamates	Major topic
Histocompatibility	Major topic
Immunosuppressive Agents	Major topic
Tyrosine	Major topic
Animals
Cell Extracts
Hemolytic Plaque Technique
Lymphocytes
Mice
Mice, Inbred A
Mice, Inbred BALB C
Molecular Weight
Serum Albumin, Bovine

Source PubMed

Show 9 more topicsShow fewer topics

Cited by11

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 53 citations.

PMID	Title	Journal	Year	In Papers	Source
6,801,188	Regulation of immune responses by I-J gene products. II. Presence of Both I-Jb and I-Jk suppressor factors in (nonsuppressor x nonsuppressor) F1 mice.	J Exp Med	1982	https://conductscience.org/library/papers/6801188	PubMed
6,795,302	Regulation of immune responses by I-J gene products. I. Production and characterization of anti-I-J monoclonal antibodies.	J Exp Med	1981	https://conductscience.org/library/papers/6795302	PubMed
6,792,727	Helper and suppressor T cell factors.	Springer Semin Immunopathol	1980	https://conductscience.org/library/papers/6792727	PubMed
6,606,014	Antigen-specific suppressor T cell interactions. II. Characterization of two different types of suppressor T cell factors specific for L-glutamic acid50-L-tyrosine50 (GT) and L-glutamic acid60-L-alanine30-L-tyrosine10 (GAT).	J Exp Med	1983	https://conductscience.org/library/papers/6606014	PubMed
6,445,400	Antigen-specific T cell-mediated suppression. V. H-2-linked genetic control of distinct antigen-specific defects in the production and activity of L-glutamic acid50-L-tyrosine50 suppressor factor.	J Exp Med	1980	https://conductscience.org/library/papers/6445400	PubMed
6,176,537	Monoclonal antibodies as a tool for phylogenetic studies of major histocompatibility antigens and beta 2-microglobulin.	Immunogenetics	1982	https://conductscience.org/library/papers/6176537	PubMed
6,168,725	H-2-restricted helper factor secreted by clone hybridoma cells.	J Exp Med	1981	https://conductscience.org/library/papers/6168725	PubMed
6,165,799	Antigen- and receptor-driven regulatory mechanisms. VII. H-2-restricted anti-idiotypic suppressor factor from efferent suppressor T cells.	J Exp Med	1981	https://conductscience.org/library/papers/6165799	PubMed
3,081,680	Regulation of immune responses by I-J gene products. VI. Recognition of I-E molecules by I-J-bearing suppressor factors.	J Exp Med	1986	https://conductscience.org/library/papers/3081680	PubMed
148,486	Suppression of the mixed lymphocyte reaction in man by a soluble T-cell factor. Specificity of the factor for both responder and stimulator.	J Exp Med	1978	https://conductscience.org/library/papers/148486	PubMed
75,231	Antigen-specific T-cell-mediated suppression. I. Induction of L-glutamic acid60-L-alanine30-L-tyrosine10 specific suppressor T cells in vitro requires both antigen-specific T-cell-suppressor factor and antigen.	J Exp Med	1978	https://conductscience.org/library/papers/75231	PubMed

Show 5 more cited byShow fewer cited by

Data dates

The date of each source this release read.

PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

None in this release

Grants
Trials
Comments and corrections
References
Genes
Drugs
Data

Sources 11

ChEMBL, through Open TargetsCC BY-SA 3.0
ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

Each source's terms

ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

For software

Read this paper from software.

The record and each of its lists are JSON, twenty rows a page. No key.

GET https://conductscience.org/api/papers/v1/68079

GET https://conductscience.org/api/papers/v1/68079/authors

API reference For AI assistants