Paper · 7907648

Functional role of the NH2-terminal cytoplasmic domain of a mammalian A-type K channel.

Tseng-Crank J and 3 others. J Gen Physiol, 1993. Cited by 58 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Functional role of the NH2-terminal cytoplasmic domain of a mammalian A-type K channel. Papers, record 7907648. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/7907648

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: Functional role of the NH2-terminal cytoplasmic domain of a mammalian A-type K channel.; PubMed
Journal: J Gen Physiol; PubMed
Year: 1993; PubMed
Publication types: Journal Article, Research Support, Non-U.S. Gov't, Research Support, U.S. Gov't, P.H.S.; PubMed
PubMed: https://pubmed.ncbi.nlm.nih.gov/7907648/; PubMed
DOI: https://doi.org/10.1085/jgp.102.6.1057; PubMed
PMC: https://pmc.ncbi.nlm.nih.gov/articles/PMC2229192/; PubMed
The journal: https://conductscience.org/library/journal-index/2985110R; PubMed
Citations, by iCite: 58; iCite
Relative citation ratio, by iCite: 1.29; iCite
Revised in PubMed: 2019-05-08; PubMed

Authors4

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename
1	Tseng-Crank J	J
2	Yao JA	J A
3	Berman MF	M F
4	Tseng GN	G N

Source PubMed

Topics13

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
Cytoplasm	Major topic
Potassium Channels	Major topic
Amino Acid Sequence
Animals
Base Sequence
DNA, Complementary
Electrophysiology
Ion Channel Gating
Molecular Sequence Data
Mutagenesis, Site-Directed
Oligonucleotides
Polymorphism, Restriction Fragment Length
Xenopus

Source PubMed

Grants2

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes it	Agency	Country	NIH project	Stated by	Source
HL-08508	NHLBI NIH HHS	United States	R01HL008508	PubMed and NIH	NIH RePORTER
			R37HL008508	NIH	NIH RePORTER

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References0

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

None in this release.

Cited by24

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 58 citations.

PMID	Title	Journal	Year	In Papers	Source
41,914,769	Differential negative dominance by KCNA2 variants associated with global developmental delay suggests KCNA2 haploinsufficiency in humans.	J Physiol	2026	https://conductscience.org/library/papers/41914769	PubMed
35,439,054	An epilepsy-associated KV1.2 charge-transfer-center mutation impairs KV1.2 and KV1.4 trafficking.	Proc Natl Acad Sci U S A	2022	https://conductscience.org/library/papers/35439054	PubMed
30,127,716	A-Type KV Channels in Dorsal Root Ganglion Neurons: Diversity, Function, and Dysfunction.	Front Mol Neurosci	2018	https://conductscience.org/library/papers/30127716	PubMed
23,325,261	Regulation of Kv2.1 K(+) conductance by cell surface channel density.	J Neurosci	2013	https://conductscience.org/library/papers/23325261	PubMed
23,251,506	Expanded functional diversity of shaker K(+) channels in cnidarians is driven by gene expansion.	PLoS One	2012	https://conductscience.org/library/papers/23251506	PubMed
22,522,931	Stereospecific binding of a disordered peptide segment mediates BK channel inactivation.	Nature	2012	https://conductscience.org/library/papers/22522931	PubMed
21,943,416	KCNE1 and KCNE2 inhibit forward trafficking of homomeric N-type voltage-gated potassium channels.	Biophys J	2011	https://conductscience.org/library/papers/21943416	PubMed
19,804,713	Differential regulation of action potentials by inactivating and noninactivating BK channels in rat adrenal chromaffin cells.	Biophys J	2009	https://conductscience.org/library/papers/19804713	PubMed
18,313,857	Kvbeta1.1 associates with Kvalpha1.4 in Chinese hamster ovary cells and pigeon type II vestibular hair cells and enhances the amplitude, inactivation and negatively shifts the steady-state inactivation range.	Neuroscience	2008	https://conductscience.org/library/papers/18313857	PubMed
16,735,752	Electrogenic Na/HCO3 cotransporter (NBCe1) variants expressed in Xenopus oocytes: functional comparison and roles of the amino and carboxy termini.	J Gen Physiol	2006	https://conductscience.org/library/papers/16735752	PubMed

1 to 10 of 24

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Genes1

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

Gene	NCBI Gene ID	Linked by	In Genes
KCNA4	3,739	PubTator's text mining	https://conductscience.org/library/genes/3739

Source NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

Sources 11

ChEMBL, through Open TargetsCC BY-SA 3.0
ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

Each source's terms

ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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