Paper · 12784342

Nuclear accumulation of basic fibroblast growth factor in human astrocytic tumors.

Fukui S and 11 others. Cancer, 2003. Cited by 36 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Nuclear accumulation of basic fibroblast growth factor in human astrocytic tumors. Papers, record 12784342. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/12784342

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: Nuclear accumulation of basic fibroblast growth factor in human astrocytic tumors.; PubMed
Journal: Cancer; PubMed
Year: 2003; PubMed
Publication types: Journal Article; PubMed
PubMed: https://pubmed.ncbi.nlm.nih.gov/12784342/; PubMed
DOI: https://doi.org/10.1002/cncr.11450; PubMed
The journal: https://conductscience.org/library/journal-index/0374236; PubMed
Citations, by iCite: 36; iCite
Relative citation ratio, by iCite: 0.69; iCite
Revised in PubMed: 2004-11-17; PubMed

Authors12

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename
1	Fukui S	Shinji
2	Nawashiro H	Hiroshi
3	Otani N	Naoki
4	Ooigawa H	Hidetoshi
5	Nomura N	Namiko
6	Yano A	Akiko
7	Miyazawa T	Takahito
8	Ohnuki A	Akira
9	Tsuzuki N	Nobusuke
10	Katoh H	Hiroshi

1 to 10 of 12Source PubMed

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Topics12

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
Astrocytoma	Major topic
Brain Neoplasms	Major topic
Fibroblast Growth Factor 2	Major topic
Adult
Cell Nucleus
Female
Glioblastoma
Humans
Male
Middle Aged
Multivariate Analysis
Prognosis

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References0

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

None in this release.

Cited by17

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 36 citations.

PMID	Title	Journal	Year	In Papers	Source
42,939,589	The Impact of FGF Expression in Breast Cancer.	Curr Issues Mol Biol	2026	https://conductscience.org/library/papers/42939589	PubMed
32,383,752	Regulation of mRNA export through API5 and nuclear FGF2 interaction.	Nucleic Acids Res	2020	https://conductscience.org/library/papers/32383752	PubMed
27,189,511	Silence of bFGF enhances chemosensitivity of glioma cells to temozolomide through the MAPK signal pathway.	Acta Biochim Biophys Sin (Shanghai)	2016	https://conductscience.org/library/papers/27189511	PubMed
27,154,171	Expression of fibroblast growth factor receptor family members is associated with prognosis in early stage cervical cancer patients.	J Transl Med	2016	https://conductscience.org/library/papers/27154171	PubMed
26,813,160	Neuron-derived FGF10 ameliorates cerebral ischemia injury via inhibiting NF-κB-dependent neuroinflammation and activating PI3K/Akt survival signaling pathway in mice.	Sci Rep	2016	https://conductscience.org/library/papers/26813160	PubMed
26,465,941	Fibroblast Growth Factor (FGF-2) and Its Receptors FGFR-2 and FGFR-3 May Be Putative Biomarkers of Malignant Transformation of Potentially Malignant Oral Lesions into Oral Squamous Cell Carcinoma.	PLoS One	2015	https://conductscience.org/library/papers/26465941	PubMed
26,141,457	Nuclear basic fibroblast growth factor regulates triple-negative breast cancer chemo-resistance.	Breast Cancer Res	2015	https://conductscience.org/library/papers/26141457	PubMed
25,048,528	Blocking the bFGF/STAT3 interaction through specific signaling pathways induces apoptosis in glioblastoma cells.	J Neurooncol	2014	https://conductscience.org/library/papers/25048528	PubMed
24,552,587	Chromatin-remodeling factors mediate the balance of sense-antisense transcription at the FGF2 locus.	Mol Endocrinol	2014	https://conductscience.org/library/papers/24552587	PubMed
23,630,597	Better prognosis of patients with glioma expressing FGF2-dependent PDGFRA irrespective of morphological diagnosis.	PLoS One	2013	https://conductscience.org/library/papers/23630597	PubMed

1 to 10 of 17

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Genes3

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

Gene	NCBI Gene ID	Linked by	In Genes	Source
FGF2	2,247	PubTator's text mining	https://conductscience.org/library/genes/2247	NCBI Gene
FGFR1	2,260	PubTator's text mining	https://conductscience.org/library/genes/2260	NCBI Gene
PCNA	5,111	PubTator's text mining	https://conductscience.org/library/genes/5111	NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

Sources 11

ChEMBL, through Open TargetsCC BY-SA 3.0
ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

Each source's terms

ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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