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Paper · 216768

Primary anti-viral cytotoxic T-cell responses in semiallogeneic chimeras are not absolutely restricted to host H-2 type.

Blanden RV and 1 other. J Exp Med, 1979. Cited by 40 papers, by iCite.

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ConductScience Library. Primary anti-viral cytotoxic T-cell responses in semiallogeneic chimeras are not absolutely restricted to host H-2 type. Papers, record 216768. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/216768

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Primary anti-viral cytotoxic T-cell responses in semiallogeneic chimeras are not absolutely restricted to host H-2 type.
PubMed
Journal
J Exp Med
PubMed
Year
1979
PubMed
Publication types
Journal Article
PubMed
Citations, by iCite
40
iCite
Relative citation ratio, by iCite
0.94
iCite
Revised in PubMed
2019-05-08
PubMed

Authors2

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Blanden RVR V
2Andrew MEM E

Source PubMed

Topics10

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Cytotoxicity, ImmunologicMajor topic
Ectromelia, InfectiousMajor topic
H-2 AntigensMajor topic
Poxviridae InfectionsMajor topic
T-LymphocytesMajor topic
Animals
Ectromelia virus
Immunologic Memory
Mice
Radiation Chimera

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References9

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
177,550A dual recognition model for cytotoxic T cells based on thymic selection of precursors with low affinity for Self H-2 antigens.Scand J Immunol1978https://conductscience.org/library/papers/77550PubMed
263,988Two different VH gene products make up the T-cell receptors.Scand J Immunol1976https://conductscience.org/library/papers/63988PubMed
3193,995In vitro primary induction of cytotoxic T cells against virus-infected syngeneic cells.J Immunol Methods1977https://conductscience.org/library/papers/193995PubMed
4305,459On the thymus in the differentiation of "H-2 self-recognition" by T cells: evidence for dual recognition?J Exp Med1978https://conductscience.org/library/papers/305459PubMed
5307,765Major histocompatibility complex-linked immune-responsiveness is acquired by lymphocytes of low-responder mice differentiating in thymus of high-responder mice.Proc Natl Acad Sci U S A1978https://conductscience.org/library/papers/307765PubMed
6302,918In a radiation chimaera, host H-2 antigens determine immune responsiveness of donor cytotoxic cells.Nature1977https://conductscience.org/library/papers/302918PubMed
71,086,219Cell-medicated cytotoxicity against ectromelia virus-infected target cells. III. Role of the H-2 gene complex.Eur J Immunol1975https://conductscience.org/library/papers/1086219PubMed
8179,718The cell-mediated immune response to ectromelia virus infection. II. Secondary response in vitro and kinetics of memory T cell production in vivo.Cell Immunol1976https://conductscience.org/library/papers/179718PubMed
94,864,541Current concepts of the immunological function of the thymus.Physiol Rev1967https://conductscience.org/library/papers/4864541PubMed

Cited by16

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 40 citations.

PMIDTitleJournalYearIn PapersSource
6,976,410Antigen recognition by cloned cytotoxic T lymphocytes follows rules predicted by the altered-self hypothesis.J Exp Med1982https://conductscience.org/library/papers/6976410PubMed
6,975,939Influence of H-2 antigen expression on killer T cell specificity, differentiation, and induction.Proc Natl Acad Sci U S A1981https://conductscience.org/library/papers/6975939PubMed
6,973,002H-2 restriction as a consequence of intentional priming. Frequency analysis of alloantigen-restricted, trinitrophenyl-specific cytotoxic T lymphocyte precursors within thymocytes of normal mice.J Exp Med1981https://conductscience.org/library/papers/6973002PubMed
6,972,043T cells respond preferentially to antigens that are similar to self H-2.Proc Natl Acad Sci U S A1981https://conductscience.org/library/papers/6972043PubMed
6,969,780Cytotoxic T lymphocyte responses by chimeric thymocytes. Self-recognition is determined early in T cell development.J Exp Med1981https://conductscience.org/library/papers/6969780PubMed
6,967,190MHC restriction, self-tolerance and the thymus.Nature1980https://conductscience.org/library/papers/6967190PubMed
6,966,321Self H-2 antigens influence the specificity of alloreactive cells.J Exp Med1980https://conductscience.org/library/papers/6966321PubMed
6,311,733H-2-linked control of resistance to ectromelia virus infection in B10 congenic mice.Immunogenetics1983https://conductscience.org/library/papers/6311733PubMed
6,261,255
H-2 restriction as a consequence of intentional priming: T cells of fully allogeneic chimeric mice as well as of normal mice respond to foreign antigens in the context of H-2 determinants not encountered on thymic epithelial cells.
Proc Natl Acad Sci U S A1980https://conductscience.org/library/papers/6261255PubMed
6,256,466Sendai virus-specific, H-2-restricted cytotoxic T lymphocyte responses of nude mice grafted with allogeneic or semi-allogeneic thymus glands.J Exp Med1980https://conductscience.org/library/papers/6256466PubMed

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Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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