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Paper · 32043969

Etv transcription factors functionally diverge from their upstream FGF signaling in lens development.

Garg A and 8 others. Elife, 2020. Cited by 24 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Etv transcription factors functionally diverge from their upstream FGF signaling in lens development. Papers, record 32043969. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/32043969

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Etv transcription factors functionally diverge from their upstream FGF signaling in lens development.
PubMed
Journal
Elife
PubMed
Year
2020
PubMed
Publication types
Journal Article, Research Support, N.I.H., Extramural, Research Support, Non-U.S. Gov't
PubMed
Citations, by iCite
24
iCite
Relative citation ratio, by iCite
1.12
iCite
Revised in PubMed
2024-04-23
PubMed

Authors9

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Garg AAnkur
2Hannan AAbdul
3Wang QQian
4Makrides NNeoklis
5Zhong JJian
6Li HHongge
7Yoon SSungtae
8Mao YYingyu
9Zhang XXinhttps://orcid.org/0000-0001-5555-0825

Source PubMed

Topics14

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Cell DifferentiationMajor topic
DNA-Binding ProteinsMajor topic
Lens, CrystallineMajor topic
Receptors, Fibroblast Growth FactorMajor topic
Receptors, NotchMajor topic
Transcription FactorsMajor topic
Animals
Crystallins
Epithelial Cells
Jagged-1 Protein
MAP Kinase Signaling System
Mice
Proto-Oncogene Proteins c-maf
TOR Serine-Threonine Kinases

Source PubMed

Grants5

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes itAgencyCountryNIH projectStated bySource
EY017061NEI NIH HHSUnited StatesR01EY017061PubMed and NIHNIH RePORTER
R01 EY017061NEI NIH HHSUnited StatesR01EY017061PubMed and NIHNIH RePORTER
EY025933NEI NIH HHSUnited StatesR01EY025933PubMed and NIHNIH RePORTER
P30 EY019007NEI NIH HHSUnited StatesP30EY019007PubMedNIH RePORTER
R01 EY025933NEI NIH HHSUnited StatesR01EY025933PubMed and NIHNIH RePORTER

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References51

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
111,714,669Fgf receptor signaling plays a role in lens induction.Development2001https://conductscience.org/library/papers/11714669PubMed
217,709,399The Notch signaling pathway controls the size of the ocular lens by directly suppressing p57Kip2 expression.Mol Cell Biol2007https://conductscience.org/library/papers/17709399PubMed
321,402,584Biological glass: structural determinants of eye lens transparency.Philos Trans R Soc Lond B Biol Sci2011https://conductscience.org/library/papers/21402584PubMed
426,302,409Autophagy in the lens.Exp Eye Res2016https://conductscience.org/library/papers/26302409PubMed
511,748,143FGF-induced lens cell proliferation and differentiation is dependent on MAPK (ERK1/2) signalling.Development2001https://conductscience.org/library/papers/11748143PubMed
611,413,000Zebrafish pea3 and erm are general targets of FGF8 signaling.Curr Biol2001https://conductscience.org/library/papers/11413000PubMed
712,402,043Sprouty1 and Sprouty2 provide a control mechanism for the Ras/MAPK signalling pathway.Nat Cell Biol2002https://conductscience.org/library/papers/12402043PubMed
89,640,329Disregulation of ocular morphogenesis by lens-specific expression of FGF-3/int-2 in transgenic mice.Dev Biol1998https://conductscience.org/library/papers/9640329PubMed
917,116,415An essential role for FGF receptor signaling in lens development.Semin Cell Dev Biol2006https://conductscience.org/library/papers/17116415PubMed
102,632,221Fibroblast growth factor (FGF) induces different responses in lens epithelial cells depending on its concentration.Development1989https://conductscience.org/library/papers/2632221PubMed

1 to 10 of 51

See all 51 references

Cited by17

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 24 citations.

PMIDTitleJournalYearIn PapersSource
42,762,961Receptor tyrosine kinase signaling in mammalian eye development.Dev Biol2026https://conductscience.org/library/papers/42762961PubMed
42,620,374ETV1 Couples Reciprocal FGF23 and PTH Signaling to Renal CYP24A1 Regulation.Res Sq2026https://conductscience.org/library/papers/42620374PubMed
41,684,939Single-Cell Profiling of the Developing Organ of Corti Identifies Etv4/5/1 as Key Regulators of Pillar Cell Identity.bioRxiv2026https://conductscience.org/library/papers/41684939PubMed
40,866,999Sex-biased transcriptome in in vitro produced bovine early embryos.Cell Biosci2025https://conductscience.org/library/papers/40866999PubMed
40,327,534Shc1 cooperates with Frs2 and Shp2 to recruit Grb2 in FGF-induced lens development.Elife2025https://conductscience.org/library/papers/40327534PubMed
40,060,475mTOR regulates Wnt signaling to promote tension-mediated lens vesicle closure.bioRxiv2025https://conductscience.org/library/papers/40060475PubMed
40,011,454ETVs dictate hPSC differentiation by tuning biophysical properties.Nat Commun2025https://conductscience.org/library/papers/40011454PubMed
39,269,548The Role of Pea3 Transcription Factor Subfamily in the Nervous System.Mol Neurobiol2025https://conductscience.org/library/papers/39269548PubMed
37,887,322Eye Lens Organoids Made Simple: Characterization of a New Three-Dimensional Organoid Model for Lens Development and Pathology.Cells2023https://conductscience.org/library/papers/37887322PubMed
37,701,781Preferential FGF18/FGFR activity in pseudoglandular versus canalicular stage human lung fibroblasts.Front Cell Dev Biol2023https://conductscience.org/library/papers/37701781PubMed

1 to 10 of 17

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Genes4

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

GeneNCBI Gene IDLinked byIn GenesSource
ETV12,115PubTator's text mininghttps://conductscience.org/library/genes/2115NCBI Gene
ETV42,118PubTator's text mininghttps://conductscience.org/library/genes/2118NCBI Gene
ETV52,119PubTator's text mininghttps://conductscience.org/library/genes/2119NCBI Gene
MAPK15,594PubTator's text mininghttps://conductscience.org/library/genes/5594NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data2

The data bank entries PubMed's record names. Trial numbers are under Trials.

Data bankAccessionSource
GEOGSE137215GEO
GEOGSE124497GEO

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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