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Paper · 32674458

Peroxisome Metabolism in Cancer.

Kim JA. Cells, 2020. Cited by 97 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Peroxisome Metabolism in Cancer. Papers, record 32674458. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/32674458

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Peroxisome Metabolism in Cancer.
PubMed
Journal
Cells
PubMed
Year
2020
PubMed
Publication types
Journal Article, Research Support, Non-U.S. Gov't, Review
PubMed
Citations, by iCite
97
iCite
Relative citation ratio, by iCite
4.47
iCite
Revised in PubMed
2021-03-04
PubMed

Authors1

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Kim JAJung-Ae

Source PubMed

Topics8

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
NeoplasmsMajor topic
PeroxisomesMajor topic
Animals
Biosynthetic Pathways
Homeostasis
Humans
Models, Biological
Reactive Oxygen Species

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References122

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
124,841,318Role and mechanism of the alkylglycerone phosphate synthase in suppressing the invasion potential of human glioma and hepatic carcinoma cells in vitro.Oncol Rep2014https://conductscience.org/library/papers/24841318PubMed
225,639,644Localization of MCT2 at peroxisomes is associated with malignant transformation in prostate cancer.J Cell Mol Med2015https://conductscience.org/library/papers/25639644PubMed
324,712,326Trifluoroibuprofen inhibits α-methylacyl coenzyme A racemase (AMACR/P504S), reduces cancer cell proliferation and inhibits in vivo tumor growth in aggressive prostate cancer models.Anticancer Agents Med Chem2014https://conductscience.org/library/papers/24712326PubMed
417,008,281Abundant expression of AMACR in many distinct tumour types.Pathology2006https://conductscience.org/library/papers/17008281PubMed
528,527,945Targeting lipid metabolism of cancer cells: A promising therapeutic strategy for cancer.Cancer Lett2017https://conductscience.org/library/papers/28527945PubMed
623,980,144Ether lipid generating enzyme AGPS alters the balance of structural and signaling lipids to fuel cancer pathogenicity.Proc Natl Acad Sci U S A2013https://conductscience.org/library/papers/23980144PubMed
718,279,392Alpha-methylacyl-CoA racemase--an 'obscure' metabolic enzyme takes centre stage.FEBS J2008https://conductscience.org/library/papers/18279392PubMed
812,705,896Peroxisomal membrane monocarboxylate transporters: evidence for a redox shuttle system?Biochem Biophys Res Commun2003https://conductscience.org/library/papers/12705896PubMed
926,344,566ATM functions at the peroxisome to induce pexophagy in response to ROS.Nat Cell Biol2015https://conductscience.org/library/papers/26344566PubMed
1010,725,470Hydrogen peroxide generation in peroxisome proliferator-induced oncogenesis.Mutat Res2000https://conductscience.org/library/papers/10725470PubMed

Cited by64

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 97 citations.

PMIDTitleJournalYearIn PapersSource
42,964,345Cinobufagin induces peroxisome dysfunction through suppression of the peroxisomal biogenesis factor proteins and interferes with lipid metabolism to affect the growth of liver cancer cells.Front Pharmacol2026https://conductscience.org/library/papers/42964345PubMed
42,792,605Exploratory Transcriptomic, Genomic, Pharmacogenomic, and Cellular Evaluation of FKBP4 in Lung Adenocarcinoma.Biology (Basel)2026https://conductscience.org/library/papers/42792605PubMed
42,769,853FSCN1 Modulates Fatty Acid Metabolism and the Coordinated Activation of AKT/mTOR and p38 MAPK Pathways in Colorectal Cancer Cells.Oncol Res2026https://conductscience.org/library/papers/42769853PubMed
42,372,295Continuous multi-omics pathway enrichment analysis resolves hidden functional heterogeneity.Brief Bioinform2026https://conductscience.org/library/papers/42372295PubMed
42,352,361Peroxisomes in Liver Diseases: From Metabolite Quality Control to Inter-Organelle and Inter-Organ Signaling.Biomolecules2026https://conductscience.org/library/papers/42352361PubMed
42,298,691Analysis and validation of abnormal signaling pathways and immune cell infiltration characteristics in digestive system cancers based on peroxisome-related genes.Biol Direct2026https://conductscience.org/library/papers/42298691PubMed
42,290,140Peroxisomal Lipid Metabolism as a Therapeutic Target in Leukemia.Mol Nutr Food Res2026https://conductscience.org/library/papers/42290140PubMed
41,977,190Oxidative Stress in Multiple Myeloma: Pathogenic Mechanisms, Biomarkers, and Redox-Targeted Therapeutic Strategies.Int J Mol Sci2026https://conductscience.org/library/papers/41977190PubMed
41,902,194Remodeling of Metabolic and Secretory Organelles During Oncogenic and Oncomodulatory Viral Infections.Viruses2026https://conductscience.org/library/papers/41902194PubMed
41,858,347Peroxisomal activity drives aggressive bladder cancer phenotypes and reveals erythorbic acid as a potential therapeutic modulator.Front Oncol2026https://conductscience.org/library/papers/41858347PubMed

1 to 10 of 64

See all 64 cited by

Genes46

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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