Paper · 39031286
The roles of FGFR3 and c-MYC in urothelial bladder cancer.
Bogale DE. Discov Oncol, 2024. Cited by 20 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. The roles of FGFR3 and c-MYC in urothelial bladder cancer. Papers, record 39031286. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/39031286
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Trials0
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References175
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 24,360,661 | FGFR3 overexpression is prognostic of adverse outcome for muscle-invasive bladder carcinoma treated with adjuvant chemotherapy. | Urol Oncol | 2014 | https://conductscience.org/library/papers/24360661 | PubMed |
| 2 | 10,096,250 | Over-expression and amplification of the c-myc gene in human urothelial carcinoma. | Int J Cancer | 1999 | https://conductscience.org/library/papers/10096250 | PubMed |
| 3 | 16,597,617 | Receptor specificity of the fibroblast growth factor family. The complete mammalian FGF family. | J Biol Chem | 2006 | https://conductscience.org/library/papers/16597617 | PubMed |
| 4 | 27,381,494 | A place for precision medicine in bladder cancer: targeting the FGFRs. | Future Oncol | 2016 | https://conductscience.org/library/papers/27381494 | PubMed |
| 5 | 24,937,673 | Expression of hypoxia-inducible factor-1α predicts benefit from hypoxia modification in invasive bladder cancer. | Br J Cancer | 2014 | https://conductscience.org/library/papers/24937673 | PubMed |
| 6 | 33,317,057 | Inhibition of the FGF/FGFR System Induces Apoptosis in Lung Cancer Cells via c-Myc Downregulation and Oxidative Stress. | Int J Mol Sci | 2020 | https://conductscience.org/library/papers/33317057 | PubMed |
| 7 | 24,302,556 | Cell-autonomous and non-cell-autonomous mechanisms of transformation by amplified FGFR1 in lung cancer. | Cancer Discov | 2014 | https://conductscience.org/library/papers/24302556 | PubMed |
| 8 | 34,508,258 | The MYC oncogene - the grand orchestrator of cancer growth and immune evasion. | Nat Rev Clin Oncol | 2022 | https://conductscience.org/library/papers/34508258 | PubMed |
| 9 | 26,869,289 | Oncogenic Gene Fusion FGFR3-TACC3 Is Regulated by Tyrosine Phosphorylation. | Mol Cancer Res | 2016 | https://conductscience.org/library/papers/26869289 | PubMed |
| 10 | 32,260,326 | Blocking Myc to Treat Cancer: Reflecting on Two Decades of Omomyc. | Cells | 2020 | https://conductscience.org/library/papers/32260326 | PubMed |
1 to 10 of 175
See all 175 referencesCited by17
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 20 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 42,966,755 | Conformational heterogeneity: a fundamental mechanism of adaptive phenotypic plasticity and therapeutic resistance. | Br J Cancer | 2026 | https://conductscience.org/library/papers/42966755 | PubMed |
| 42,941,789 | pH-Responsive Erdafitinib Liposomes with FGFR3 Targeting for Intravesical Bladder Cancer Therapy. | Int J Mol Sci | 2026 | https://conductscience.org/library/papers/42941789 | PubMed |
| 42,633,326 | Advances in FGF/FGFR Signaling: Implications for Disease and Therapy. | MedComm (2020) | 2026 | https://conductscience.org/library/papers/42633326 | PubMed |
| 42,470,241 | Shared Genetics of Kidney Function Traits and Bladder Cancer: A Genome-Wide Cross-Trait Analysis. | Cancer Med | 2026 | https://conductscience.org/library/papers/42470241 | PubMed |
| 42,429,045 | FGFR1 signaling in rheumatoid arthritis: Mechanisms of bone destruction and therapeutic targeting (Review). | Int J Mol Med | 2026 | https://conductscience.org/library/papers/42429045 | PubMed |
| 42,366,674 | Biomarker testing patterns among patients newly diagnosed with metastatic non-small cell lung cancer, prostate cancer, and bladder cancer. | Oncologist | 2026 | https://conductscience.org/library/papers/42366674 | PubMed |
| 42,301,608 | Filamin C activates MAPK/ERK signaling to facilitate epithelial-mesenchymal transition in bladder cancer. | Clin Transl Oncol | 2026 | https://conductscience.org/library/papers/42301608 | PubMed |
| 42,283,962 | CHD6 promotes triple-negative breast cancer progression and is associated with PI3K/AKT and MAPK pathways. | Int J Clin Oncol | 2026 | https://conductscience.org/library/papers/42283962 | PubMed |
| 42,177,363 | FGFR3 oncogenic activation drives oxidative metabolic reprogramming in bladder cancer: a systems metabolomics approach. | Commun Biol | 2026 | https://conductscience.org/library/papers/42177363 | PubMed |
| 41,681,916 | FGFR Testing in Metastatic Urothelial Carcinoma-Who, When, and How to Test. | Cancers (Basel) | 2026 | https://conductscience.org/library/papers/41681916 | PubMed |
1 to 10 of 17
See all 17 cited byGenes2
The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.
| Gene | NCBI Gene ID | Linked by | In Genes | Source |
|---|---|---|---|---|
| FGFR3 | 2,261 | PubTator's text mining | https://conductscience.org/library/genes/2261 | NCBI Gene |
| MYC | 4,609 | PubTator's text mining | https://conductscience.org/library/genes/4609 | NCBI Gene |
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Data dates
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- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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