Paper · 39043538

Quantitative and qualitative differences in the activation of a fibroblast growth factor receptor by different FGF ligands.

Krzyscik MA and 4 others. Cytokine Growth Factor Rev, 2024. Cited by 10 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Quantitative and qualitative differences in the activation of a fibroblast growth factor receptor by different FGF ligands. Papers, record 39043538. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/39043538

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title: Quantitative and qualitative differences in the activation of a fibroblast growth factor receptor by different FGF ligands.; PubMed
Journal: Cytokine Growth Factor Rev; PubMed
Year: 2024; PubMed
Publication types: Journal Article, Review, Research Support, N.I.H., Extramural; PubMed
PubMed: https://pubmed.ncbi.nlm.nih.gov/39043538/; PubMed
DOI: https://doi.org/10.1016/j.cytogfr.2024.07.002; PubMed
PMC: https://pmc.ncbi.nlm.nih.gov/articles/PMC11389727/; PubMed
The journal: https://conductscience.org/library/journal-index/9612306; PubMed
Citations, by iCite: 10; iCite
Relative citation ratio, by iCite: 1.36, provisional; iCite
Revised in PubMed: 2026-05-30; PubMed

Authors5

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

Order	Author	Forename
1	Krzyscik MA	Mateusz A
2	Karl K	Kelly
3	Dudeja P	Pooja
4	Krejci P	Pavel
5	Hristova K	Kalina

Source PubMed

Topics8

NLM's MeSH indexing of the paper, its major topics first.

MeSH term	Major
Fibroblast Growth Factors	Major topic
Signal Transduction	Major topic
Receptors, Fibroblast Growth Factor	Major topic
Receptor, Fibroblast Growth Factor, Type 1	Major topic
Animals
Humans
Ligands
Chondrocytes

Source PubMed

Grants1

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes it	Agency	Country	NIH project	Stated by
R01 GM068619	NIGMS NIH HHS	United States	R01GM068619	PubMed and NIH

Source NIH RePORTER

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References80

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

Order	PMID	Title	Journal	Year	In Papers	Source
1	26,731,578	Fibroblast growth factor 2 dimer with superagonist in vitro activity improves granulation tissue formation during wound healing.	Biomaterials	2016	https://conductscience.org/library/papers/26731578	PubMed
2	38,379,823	Computer-aided engineering of stabilized fibroblast growth factor 21.	Comput Struct Biotechnol J	2024	https://conductscience.org/library/papers/38379823	PubMed
3	6,141,562	Operational models of pharmacological agonism.	Proc R Soc Lond B Biol Sci	1983	https://conductscience.org/library/papers/6141562	PubMed
4	18,449,196	Genetic evidence that FGFs have an instructive role in limb proximal-distal patterning.	Nature	2008	https://conductscience.org/library/papers/18449196	PubMed
5	9,435,295	Fibroblast growth factor receptor 2 (FGFR2)-mediated reciprocal regulation loop between FGF8 and FGF10 is essential for limb induction.	Development	1998	https://conductscience.org/library/papers/9435295	PubMed
6	16,597,617	Receptor specificity of the fibroblast growth factor family. The complete mammalian FGF family.	J Biol Chem	2006	https://conductscience.org/library/papers/16597617	PubMed
7	7,516,330	Internalization of fibroblast growth factor receptor is inhibited by a point mutation at tyrosine 766.	J Biol Chem	1994	https://conductscience.org/library/papers/7516330	PubMed
8	20,940,169	Fibroblast growth factors: from molecular evolution to roles in development, metabolism and disease.	J Biochem	2011	https://conductscience.org/library/papers/20940169	PubMed
9	34,341,408	Characterization of the structural forces governing the reversibility of the thermal unfolding of the human acidic fibroblast growth factor.	Sci Rep	2021	https://conductscience.org/library/papers/34341408	PubMed
10	28,174,517	A Practical Guide to Approaching Biased Agonism at G Protein Coupled Receptors.	Front Neurosci	2017	https://conductscience.org/library/papers/28174517	PubMed

1 to 10 of 80

See all 80 references

Cited by9

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 10 citations.

PMID	Title	Journal	Year	In Papers	Source
42,974,000	Functionalization of polycaprolactone scaffolds for tissue engineering: molecular cues, bioactive cell-material interfaces, and translational perspectives.	Nanoscale Adv	2026	https://conductscience.org/library/papers/42974000	PubMed
42,961,165	Bazi Bushen Attenuates Skeletal Muscle Decline Associated With Oestrogen Deficiency and Modulates FGFR1/ERK Signalling.	J Cell Mol Med	2026	https://conductscience.org/library/papers/42961165	PubMed
42,939,685	Emerging Roles of FGFR3 and FGF23-Associated Signaling in Renal Fibrosis: Molecular Integration, Clinical Correlates, and Therapeutic Perspectives.	Biomolecules	2026	https://conductscience.org/library/papers/42939685	PubMed
42,542,977	Mechanisms and Manifestations of Ligand Bias in Signaling: Focus on Receptor Tyrosine Kinases.	Chem Rev	2026	https://conductscience.org/library/papers/42542977	PubMed
41,551,579	Mechanistic study of glutamine metabolic reprogramming driving non-small cell lung cancer progression via the FGF17-FGFR4 axis mediating epithelial-mesenchymal transition.	Front Mol Biosci	2025	https://conductscience.org/library/papers/41551579	PubMed
41,131,959	The role of fibroblast growth factors in cell and cancer metabolism.	FEBS Lett	2026	https://conductscience.org/library/papers/41131959	PubMed
40,518,115	FGFs/FGFRs and neuroinflammatory diseases: mechanism, drug therapies and delivery systems.	J Adv Res	2025	https://conductscience.org/library/papers/40518115	PubMed
39,840,811	Allosteric modulation of NF1 GAP: Differential distributions of catalytically competent populations in loss-of-function and gain-of-function mutants.	Protein Sci	2025	https://conductscience.org/library/papers/39840811	PubMed
39,467,624	Cordycepin Activates Autophagy to Suppress FGF9-induced TM3 Mouse Leydig Progenitor Cell Proliferation.	Cancer Genomics Proteomics	2024	https://conductscience.org/library/papers/39467624	PubMed

Genes0

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

None in this release.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed: 2026-11-06; PubMed
iCite: 2026-11-05; iCite
PMC's ID file: 2026-11-05; PMC's ID file
NIH RePORTER's grant and paper links: 2026-11-01; NIH RePORTER's grant and paper links
Trials: 2026-11-12
Gene search: 2026-11-11
Pipelines: 2026-09-18

Sources 11

ChEMBL, through Open TargetsCC BY-SA 3.0
ClinicalTrials.govNo license stated
HGNCCC0
iCiteCC BY 4.0
NCBI GenePublic domain (US government)
NIH RePORTER's grant and paper linksPublic domain
Open TargetsCC0 1.0
PMC's ID filePublic domain (US government)
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0

Each source's terms

ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
ClinicalTrials.govnone stated
HGNCCC0: https://www.genenames.org/about/license/
iCiteCC BY 4.0
NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
PubMedNLM's terms
PubTator 3Public domain
UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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