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Paper · 41206509

The Glyco-Switch of life: O-GlcNAcylation in cell fate decision.

Wang A and 2 others. Glycobiology, 2025. Cited by 0 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. The Glyco-Switch of life: O-GlcNAcylation in cell fate decision. Papers, record 41206509. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/41206509

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
The Glyco-Switch of life: O-GlcNAcylation in cell fate decision.
PubMed
Journal
Glycobiology
PubMed
Year
2025
PubMed
Publication types
Journal Article, Review, Research Support, N.I.H., Extramural
PubMed
Citations, by iCite
0
iCite
Revised in PubMed
2026-05-11
PubMed

Authors3

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Wang AAo
2Young MMatthew
3Jiang JJiaoyanghttps://orcid.org/0000-0001-8445-9924

Source PubMed

Topics9

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
AcetylglucosamineMajor topic
Humans
Signal Transduction
Glycosylation
Animals
NF-kappa B
Cell Differentiation
Transforming Growth Factor beta
Receptors, Notch

Source PubMed

Grants4

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes itAgencyCountryNIH projectStated bySource
R01 GM121718NIGMS NIH HHSUnited StatesR01GM121718PubMed and NIHNIH RePORTER
R01 GM152998NIGMS NIH HHSUnited StatesR01GM152998PubMed and NIHNIH RePORTER
R01GM121718NIH HHSUnited StatesR01GM121718PubMed and NIHNIH RePORTER
R01GM152998NIH HHSUnited StatesR01GM152998PubMed and NIHNIH RePORTER

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References145

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
130,664,665The lineage stability and suppressive program of regulatory T cells require protein O-GlcNAcylation.Nat Commun2019https://conductscience.org/library/papers/30664665PubMed
238,942,028O-GlcNAcase regulates pluripotency states of human embryonic stem cells.Stem Cell Reports2024https://conductscience.org/library/papers/38942028PubMed
332,472,127Multifaceted regulation of Notch signaling by glycosylation.Glycobiology2021https://conductscience.org/library/papers/32472127PubMed
410,521,409IkappaB kinases phosphorylate NF-kappaB p65 subunit on serine 536 in the transactivation domain.J Biol Chem1999https://conductscience.org/library/papers/10521409PubMed
532,896,043The landscape of GPCR signaling in the regulation of epidermal stem cell fate and skin homeostasis.Stem Cells2020https://conductscience.org/library/papers/32896043PubMed
636,104,770O-GlcNAcylation: an important post-translational modification and a potential therapeutic target for cancer therapy.Mol Med2022https://conductscience.org/library/papers/36104770PubMed
712,244,103Characterization of the Ikappa B-kinase NEMO binding domain.J Biol Chem2002https://conductscience.org/library/papers/12244103PubMed
839,399,480G protein coupled receptor in apoptosis and apoptotic cell clearance.FASEB Bioadv2024https://conductscience.org/library/papers/39399480PubMed
933,515,675SHCBP1 interacting with EOGT enhances O-GlcNAcylation of NOTCH1 and promotes the development of pancreatic cancer.Genomics2021https://conductscience.org/library/papers/33515675PubMed
1022,158,438O-linked-N-acetylglucosamine on extracellular protein domains mediates epithelial cell-matrix interactions.Nat Commun2011https://conductscience.org/library/papers/22158438PubMed

1 to 10 of 145

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Cited by1

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 0 citations.

PMIDTitleJournalYearIn Papers
42,471,548OGT as a metabolic-epigenetic integrator in cancer: context-dependent mechanisms and therapeutic vulnerabilities.Mol Med2026https://conductscience.org/library/papers/42471548

Source PubMed

Genes4

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

GeneNCBI Gene IDLinked byIn GenesSource
CXCR610,663PubTator's text mininghttps://conductscience.org/library/genes/10663NCBI Gene
NFKB14,790PubTator's text mininghttps://conductscience.org/library/genes/4790NCBI Gene
OGT8,473PubTator's text mininghttps://conductscience.org/library/genes/8473NCBI Gene
TGFB17,040PubTator's text mininghttps://conductscience.org/library/genes/7040NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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