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Paper · 42094430

Fibroblast Growth Factor Receptor Signaling in Maturing Osteoblasts Controls Cell-Matrix Interactions Critical for Osteocyte Survival.

Patra D and 8 others. bioRxiv, 2026. Cited by 0 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Fibroblast Growth Factor Receptor Signaling in Maturing Osteoblasts Controls Cell-Matrix Interactions Critical for Osteocyte Survival. Papers, record 42094430. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/42094430

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Fibroblast Growth Factor Receptor Signaling in Maturing Osteoblasts Controls Cell-Matrix Interactions Critical for Osteocyte Survival.
PubMed
Journal
bioRxiv
PubMed
Year
2026
PubMed
Publication types
Journal Article, Preprint
PubMed
Citations, by iCite
0
iCite
Revised in PubMed
2026-09-21
PubMed

Authors9

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Patra DDebabratahttps://orcid.org/0000-0001-8606-1573
2Smith CCraig
3Wei CChenming
4Mazur CMCourtney Mhttps://orcid.org/0000-0003-1143-5483
5Ameadaji IIlemona
6Li TTiandao
7Wein MNMarc Nhttps://orcid.org/0000-0002-6015-8147
8Silva MJMathew J
9Ornitz DMDavid M

Source PubMed

Topics0

NLM's MeSH indexing of the paper, its major topics first.

None in this release.

Grants7

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

Grant, as the record writes itAgencyCountryNIH projectStated bySource
R01 AR066590NIAMS NIH HHSUnited StatesR01AR066590PubMedNIH RePORTER
R01 DK116716NIDDK NIH HHSUnited StatesR01DK116716PubMedNIH RePORTER
P30 AR074992NIAMS NIH HHSUnited StatesP30AR074992PubMedNIH RePORTER
F32 AR081660NIAMS NIH HHSUnited StatesF32AR081660PubMedNIH RePORTER
R01 AR047867NIAMS NIH HHSUnited StatesR01AR047867PubMedNIH RePORTER
T32 DK007028NIDDK NIH HHSUnited StatesT32DK007028PubMedNIH RePORTER
R01 AR079246NIAMS NIH HHSUnited StatesR01AR079246PubMedNIH RePORTER

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections1

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

KindPMIDTitleIn Papers
Updated in42,706,256Fibroblast growth factor receptor signaling in maturing osteoblasts controls cell-matrix interactions critical for osteocyte survival.https://conductscience.org/library/papers/42706256

Source PubMed

References54

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
19,566,981Bone matrix proteins: isolation and characterization of a novel cell-binding keratan sulfate proteoglycan (osteoadherin) from bovine bone.J Cell Biol1998https://conductscience.org/library/papers/9566981PubMed
211,032,854Osteocyte and osteoblast apoptosis and excessive bone deposition accompany failure of collagenase cleavage of collagen.J Clin Invest2000https://conductscience.org/library/papers/11032854PubMed
332,040,934The Osteocyte: New Insights.Annu Rev Physiol2020https://conductscience.org/library/papers/32040934PubMed
433,437,628A modified tape transfer approach for rapidly preparing high-quality cryosections of undecalcified adult rodent bones.J Orthop Translat2021https://conductscience.org/library/papers/33437628PubMed
512,756,187Conditional inactivation of FGF receptor 2 reveals an essential role for FGF signaling in the regulation of osteoblast function and bone growth.Development2003https://conductscience.org/library/papers/12756187PubMed
623,994,943Osteocyte differentiation is regulated by extracellular matrix stiffness and intercellular separation.J Mech Behav Biomed Mater2013https://conductscience.org/library/papers/23994943PubMed
721,312,267Sclerostin is a locally acting regulator of late-osteoblast/preosteocyte differentiation and regulates mineralization through a MEPE-ASARM-dependent mechanism.J Bone Miner Res2011https://conductscience.org/library/papers/21312267PubMed
811,276,432Fibroblast growth factors.Genome Biol2001https://conductscience.org/library/papers/11276432PubMed
910,470,495Expression of mRNAs for type-I collagen, bone sialoprotein, osteocalcin, and osteopontin at different stages of osteoblastic differentiation and their regulation by 1,25 dihydroxyvitamin D3.Cell Tissue Res1999https://conductscience.org/library/papers/10470495PubMed
1026,235,988FGF23 Regulates Bone Mineralization in a 1,25(OH)2 D3 and Klotho-Independent Manner.J Bone Miner Res2016https://conductscience.org/library/papers/26235988PubMed

1 to 10 of 54

See all 54 references

Cited by0

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 0 citations.

None in this release.

Genes4

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

GeneNCBI Gene IDLinked byIn GenesSource
BGLAP632PubTator's text mininghttps://conductscience.org/library/genes/632NCBI Gene
FGFR12,260PubTator's text mininghttps://conductscience.org/library/genes/2260NCBI Gene
PDPN10,630PubTator's text mininghttps://conductscience.org/library/genes/10630NCBI Gene
SOST50,964PubTator's text mininghttps://conductscience.org/library/genes/50964NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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