Paper · 6603301
Human B cell growth factors overcome T cell-mediated inhibition of specific antibody production: a possible mechanism for the exacerbation of autoimmune disease.
Brenner MK and 1 other. Clin Exp Immunol, 1983. Cited by 4 papers, by iCite.
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ConductScience Library. Human B cell growth factors overcome T cell-mediated inhibition of specific antibody production: a possible mechanism for the exacerbation of autoimmune disease. Papers, record 6603301. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/6603301
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- Human B cell growth factors overcome T cell-mediated inhibition of specific antibody production: a possible mechanism for the exacerbation of autoimmune disease.
- PubMed
Topics12
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Antibody Formation | Major topic |
| Autoimmune Diseases | Major topic |
| Growth Substances | Major topic |
| T-Lymphocytes | Major topic |
| B-Lymphocytes | |
| Cells, Cultured | |
| Humans | |
| Immune Tolerance | |
| Immunoglobulin G | |
| Immunoglobulin M | |
| Interleukin-4 | |
| Lymphocyte Cooperation |
Source PubMed
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Trials0
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None in this release.
Comments and corrections0
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None in this release.
References17
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 70,359 | In vitro studies on H-2-linked unresponsiveness to synthetic polypeptides. III. Production of an antigen-specific T helper cell factor to (T,G)-A--L. | Eur J Immunol | 1977 | https://conductscience.org/library/papers/70359 | PubMed |
| 2 | 70,396 | Conditions determining the generation and expression of T helper cells. | Immunol Rev | 1977 | https://conductscience.org/library/papers/70396 | PubMed |
| 3 | 359,749 | Antigen-specific helper factor in man. | J Exp Med | 1978 | https://conductscience.org/library/papers/359749 | PubMed |
| 4 | 83,204 | Hypothesis: nonspecific polyclonal activation of memory B cells by antigen as a mechanism for the preservation of long term immunologic anamnesis. | Cell Immunol | 1978 | https://conductscience.org/library/papers/83204 | PubMed |
| 5 | 85,648 | Development of allergy in children. I. Association with virus infections. | J Allergy Clin Immunol | 1979 | https://conductscience.org/library/papers/85648 | PubMed |
| 6 | 312,259 | The production of a soluble human T-lymphocyte derived factor which substitutes for helper T lymphocytes in the in vitro production of immunoglobulin. | Immunology | 1979 | https://conductscience.org/library/papers/312259 | PubMed |
| 7 | 156,242 | Induction of polyclonal antibody synthesis by human allogeneic and autologous helper factors. | J Exp Med | 1979 | https://conductscience.org/library/papers/156242 | PubMed |
| 8 | 114,584 | T lymphocyte-mediated suppression of myeloma function in vitro. I. Suppression by allogeneically activated T lymphocytes. | J Immunol | 1979 | https://conductscience.org/library/papers/114584 | PubMed |
| 9 | 6,159,112 | Induction of nonspecific, proliferative dependent suppressor T lymphocytes in human mixed lymphocyte cultures. | Cell Immunol | 1980 | https://conductscience.org/library/papers/6159112 | PubMed |
| 10 | 6,449,702 | A human soluble suppressor factor affecting lymphocyte responses in vitro. | Proc Natl Acad Sci U S A | 1980 | https://conductscience.org/library/papers/6449702 | PubMed |
1 to 10 of 17
See all 17 referencesCited by3
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 4 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 6,235,068 | Enhanced T helper cell function for the spontaneous production of IgM rheumatoid factor in vitro in rheumatoid arthritis. | Clin Exp Immunol | 1984 | https://conductscience.org/library/papers/6235068 | PubMed |
| 3,456,356 | Cooperation between major histocompatibility complex mismatched mononuclear cells from a human chimera in the production of antigen-specific antibody. | J Clin Invest | 1986 | https://conductscience.org/library/papers/3456356 | PubMed |
| 2,959,412 | Rapid recovery of helper activity following T cell depleted allogeneic marrow transplant. | Clin Exp Immunol | 1987 | https://conductscience.org/library/papers/2959412 | PubMed |
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- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
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- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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