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Genomic analyses identify molecular subtypes of pancreatic cancer.
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Paper · 26909576

Cited by2,192

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 2,921 citations.

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PMIDTitleJournalYearIn PapersSource
42,979,207Liver-Lung Organotropism in Pancreatic Ductal Adenocarcinoma: A Five-Stage Framework.Cancer Manag Res2026https://conductscience.org/library/papers/42979207PubMed
42,978,795Machine learning models integrating 2D and 3D intratumoral heterogeneity features from multi-sequence MRI: accurate prediction of perineural invasion in pancreatic ductal adenocarcinoma.Front Artif Intell2026https://conductscience.org/library/papers/42978795PubMed
42,942,302BIMS-GA: Adaptive Multi-Objective Optimization for Network-Aware Pancreatic Ductal Adenocarcinoma Biomarker Discovery.Life (Basel)2026https://conductscience.org/library/papers/42942302PubMed
42,940,093Prognostic and Spatial Organization of Tertiary Lymphoid Structures in Pancreatic Ductal Adenocarcinoma.Biomedicines2026https://conductscience.org/library/papers/42940093PubMed
42,938,942Context-Dependent Spatial and Functional Roles of CD44 Across Pancreatic Ductal Adenocarcinoma States.Cancers (Basel)2026https://conductscience.org/library/papers/42938942PubMed
42,938,908The Evolution of Oligometastatic Pancreatic Ductal Adenocarcinoma: From Anatomic Description to Biologically Informed Patient Selection.Cancers (Basel)2026https://conductscience.org/library/papers/42938908PubMed
42,935,847Treating a systemic disease as a local one: failure points of current pancreatic cancer management and a multimodal, biology-guided framework for redesign.Front Oncol2026https://conductscience.org/library/papers/42935847PubMed
42,922,762Paternal regulation of H3K4 methylation supports tumor suppressor networks in mammals intergenerationally.bioRxiv2026https://conductscience.org/library/papers/42922762PubMed
42,898,383A MYC-LDHA-MCT4 expression program marks an SPP1/TREM2-centered macrophage state in pancreatic ductal adenocarcinoma.Front Immunol2026https://conductscience.org/library/papers/42898383PubMed
42,890,651Transcription factor-driven metabolic stratification reveals glycolytic and lipogenic subtypes with distinct prognostic and functional features in pancreatic cancer.Front Oncol2026https://conductscience.org/library/papers/42890651PubMed
42,870,132Driver mutation profiling by next-generation sequencing predicts survival and recurrence after curative-intent resection for pancreatic ductal adenocarcinoma: a genomic cohort study.Clin Transl Oncol2026https://conductscience.org/library/papers/42870132PubMed
42,869,988Clonal heterogeneity in human pancreatic ductal adenocarcinoma and its impact on tumor progression.Nat Genet2026https://conductscience.org/library/papers/42869988PubMed
42,866,405Beyond tumor biology: can host nutritional status refine prediction of early recurrence after pancreatic cancer surgery?Gland Surg2026https://conductscience.org/library/papers/42866405PubMed
42,858,298MVB Maturation Responsive to KRASG12D/TP53R175H Co-mutations Fuels Extracellular Vesicle-hypersecretion Phenotype to Aggravate Lymphangiogenesis and Lymphatic Metastasis.J Extracell Vesicles2026https://conductscience.org/library/papers/42858298PubMed
42,840,291Epigenetic dysregulation in pancreatic cancer: emerging biomarkers and clinical applications.Front Epigenet Epigenom2025https://conductscience.org/library/papers/42840291PubMed
42,836,735Pancreatic Cancer KRAS Breakthrough: Drug Graveyard No More.Clin Cancer Res2026https://conductscience.org/library/papers/42836735PubMed
42,832,147Radical Antegrade Modular Pancreatosplenectomy Versus Standard Resection in T3 Body and Tail Pancreatic Ductal Adenocarcinoma: A Multicenter Retrospective Cohort Study.Ann Surg Oncol2026https://conductscience.org/library/papers/42832147PubMed
42,826,199Tumor cell-intrinsic stress states drive sensitivity to CAR T cell therapy in pancreatic cancer.Sci Adv2026https://conductscience.org/library/papers/42826199PubMed
42,824,914ApoA2 isoforms as blood-based biomarkers reflecting pancreatic exocrine dysfunction for risk stratification and perioperative management of pancreatic cancer.Front Oncol2026https://conductscience.org/library/papers/42824914PubMed
42,807,818Engineering strategies to address immune and delivery barriers in pancreatic ductal adenocarcinoma: a barrier-matched translational framework.Front Immunol2026https://conductscience.org/library/papers/42807818PubMed

papers-2026-11-12-0.1.0

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  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
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  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

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