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Paper · 33529461

BAP1 promotes viability and migration of ECA109 cells through KLF5/CyclinD1/FGF-BP1.

Wang F and 3 others. FEBS Open Bio, 2021. Cited by 15 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. BAP1 promotes viability and migration of ECA109 cells through KLF5/CyclinD1/FGF-BP1. Papers, record 33529461. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/33529461

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
BAP1 promotes viability and migration of ECA109 cells through KLF5/CyclinD1/FGF-BP1.
PubMed
Journal
FEBS Open Bio
PubMed
Year
2021
PubMed
Publication types
Journal Article, Research Support, Non-U.S. Gov't
PubMed
Citations, by iCite
15
iCite
Relative citation ratio, by iCite
0.94
iCite
Revised in PubMed
2022-01-03
PubMed

Authors4

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Wang FFengyun
2Luo MMing
3Qu HHonglan
4Cheng YYufenghttps://orcid.org/0000-0003-1466-3115

Source PubMed

Topics14

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Esophageal NeoplasmsMajor topic
Tumor Suppressor ProteinsMajor topic
Ubiquitin ThiolesteraseMajor topic
Cell Line, Tumor
Cell Movement
Cell Proliferation
Cyclin D1
Gene Expression
Gene Expression Regulation, Neoplastic
Humans
Intercellular Signaling Peptides and Proteins
Kruppel-Like Transcription Factors
Prognosis
Transcription Factors

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections1

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

KindPMIDTitleIn Papers
Erratum in34,694,753Erratum to: BAP1 promotes viability and migration of ECA109 cells through KLF5/CyclinD1/FGF-BP1.https://conductscience.org/library/papers/34694753

Source PubMed

References17

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
127,042,227Esophageal cancer statistics in China, 2011: Estimates based on 177 cancer registries.Thorac Cancer2016https://conductscience.org/library/papers/27042227PubMed
221,874,000Germline BAP1 mutations predispose to malignant mesothelioma.Nat Genet2011https://conductscience.org/library/papers/21874000PubMed
326,808,342Cancer statistics in China, 2015.CA Cancer J Clin2016https://conductscience.org/library/papers/26808342PubMed
416,102,021Significance of the transcription factor KLF5 in cardiovascular remodeling.J Thromb Haemost2005https://conductscience.org/library/papers/16102021PubMed
516,034,054The ubiquitin-proteasome pathway and its role in cancer.J Clin Oncol2005https://conductscience.org/library/papers/16034054PubMed
622,406,828Epigenetic biomarkers in esophageal cancer.Cancer Lett2014https://conductscience.org/library/papers/22406828PubMed
728,781,790Somatic BRCA1-associated protein 1 (BAP1) loss is an early and rare event in esophageal adenocarcinoma.Mol Clin Oncol2017https://conductscience.org/library/papers/28781790PubMed
821,051,595Frequent mutation of BAP1 in metastasizing uveal melanomas.Science2010https://conductscience.org/library/papers/21051595PubMed
926,419,610BAP1 promotes breast cancer cell proliferation and metastasis by deubiquitinating KLF5.Nat Commun2015https://conductscience.org/library/papers/26419610PubMed
1019,815,555The deubiquitinating enzyme BAP1 regulates cell growth via interaction with HCF-1.J Biol Chem2009https://conductscience.org/library/papers/19815555PubMed

1 to 10 of 17

See all 17 references

Cited by16

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 15 citations.

PMIDTitleJournalYearIn PapersSource
42,977,970FGFBPs in health and tumors: emerging prognostic biomarkers and therapeutic implications.Biomark Res2026https://conductscience.org/library/papers/42977970PubMed
42,464,658The role of deubiquitinating enzymes and their inhibitors in esophageal carcinoma (Review).Int J Oncol2026https://conductscience.org/library/papers/42464658PubMed
41,178,930Abnormal expression and potential clinical value of oncogenic Krüppel-like factor-5 in lung squamous cell carcinoma.World J Clin Oncol2025https://conductscience.org/library/papers/41178930PubMed
40,535,101Single-cell RNA sequencing analysis of intrahepatic cholangiocarcinoma reveals SPP1 facilitates disease progression via interaction with CD4+ T cells.Oncol Lett2025https://conductscience.org/library/papers/40535101PubMed
40,427,333Decoding the Function of FGFBP1 in Sheep Adipocyte Proliferation and Differentiation.Animals (Basel)2025https://conductscience.org/library/papers/40427333PubMed
39,984,455Nuclear localization of BRCA1-associated protein 1 is important in suppressing hepatocellular carcinoma metastasis via CTCF and NRF1/OGT axis.Cell Death Dis2025https://conductscience.org/library/papers/39984455PubMed
39,918,680KLF5 promotes esophageal squamous cell carcinoma radioresistance by targeting the Keap1-Nrf2 pathway.Discov Oncol2025https://conductscience.org/library/papers/39918680PubMed
39,888,288Positive Feedback Regulation between KLF5 and XPO1 Promotes Cell Cycle Progression of Basal like Breast Cancer.Adv Sci (Weinh)2025https://conductscience.org/library/papers/39888288PubMed
39,379,684LN-439A, a novel BAP1 inhibitor, suppresses the growth of basal-like breast cancer by degrading KLF5.Acta Pharmacol Sin2025https://conductscience.org/library/papers/39379684PubMed
39,350,776Extraction and Prioritization of a Gene-Cancer-By-Survival Network Involved in Homeostasis of Intracellular Calcium Concentrations Using TCGA PANCAN Data.Bioelectricity2022https://conductscience.org/library/papers/39350776PubMed

1 to 10 of 16

See all 16 cited by

Genes8

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

GeneNCBI Gene IDLinked byIn GenesSource
BAP18,314PubTator's text mininghttps://conductscience.org/library/genes/8314NCBI Gene
BRAP8,315PubTator's text mininghttps://conductscience.org/library/genes/8315NCBI Gene
CCND1595PubTator's text mininghttps://conductscience.org/library/genes/595NCBI Gene
FGFBP19,982PubTator's text mininghttps://conductscience.org/library/genes/9982NCBI Gene
KLF5688PubTator's text mininghttps://conductscience.org/library/genes/688NCBI Gene
SERPINE15,054PubTator's text mininghttps://conductscience.org/library/genes/5054NCBI Gene
TGFB17,040PubTator's text mininghttps://conductscience.org/library/genes/7040NCBI Gene
UCHL17,345PubTator's text mininghttps://conductscience.org/library/genes/7345NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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