Paper · 36098804
A dominant negative mutation uncovers cooperative control of caudal Wolffian duct development by Sprouty genes.
Altés G and 12 others. Cell Mol Life Sci, 2022. Cited by 4 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. A dominant negative mutation uncovers cooperative control of caudal Wolffian duct development by Sprouty genes. Papers, record 36098804. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/36098804
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- A dominant negative mutation uncovers cooperative control of caudal Wolffian duct development by Sprouty genes.
- PubMed
Topics11
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Organogenesis | Major topic |
| Wolffian Ducts | Major topic |
| Animals | |
| Female | |
| Male | |
| Mammals | |
| Mice | |
| Mice, Knockout | |
| Mutation | |
| Signal Transduction | |
| Tyrosine |
Source PubMed
Grants2
Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.
| Grant, as the record writes it | Agency | Country | NIH project | Stated by |
|---|---|---|---|---|
| PID2020- 114947GB-I00 | Ministerio de Ciencia, Innovación y Universidades | PubMed | ||
| BFU2017-83646-P | Ministerio de Economía y Competitividad | PubMed |
Source PubMed
Trials0
The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.
None in this release.
Comments and corrections0
PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.
None in this release.
References36
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 20,084,103 | Kidney development in the absence of Gdnf and Spry1 requires Fgf10. | PLoS Genet | 2010 | https://conductscience.org/library/papers/20084103 | PubMed |
| 2 | 17,202,188 | The mouse seminal vesicle shape mutation is allelic with Fgfr2. | Development | 2007 | https://conductscience.org/library/papers/17202188 | PubMed |
| 3 | 12,593,796 | hSpry2 is targeted to the ubiquitin-dependent proteasome pathway by c-Cbl. | Curr Biol | 2003 | https://conductscience.org/library/papers/12593796 | PubMed |
| 4 | 25,376,241 | β-CateninC429S mice exhibit sterility consequent to spatiotemporally sustained Wnt signalling in the internal genitalia. | Sci Rep | 2014 | https://conductscience.org/library/papers/25376241 | PubMed |
| 5 | 24,361,260 | Coordinated activity of Spry1 and Spry2 is required for normal development of the external genitalia. | Dev Biol | 2014 | https://conductscience.org/library/papers/24361260 | PubMed |
| 6 | 33,247,285 | Antigen retrieval and clearing for whole-organ immunofluorescence by FLASH. | Nat Protoc | 2021 | https://conductscience.org/library/papers/33247285 | PubMed |
| 7 | 29,079,424 | Feedback regulation of RTK signaling in development. | Dev Biol | 2019 | https://conductscience.org/library/papers/29079424 | PubMed |
| 8 | 15,937,482 | Loss of mammalian Sprouty2 leads to enteric neuronal hyperplasia and esophageal achalasia. | Nat Neurosci | 2005 | https://conductscience.org/library/papers/15937482 | PubMed |
| 9 | 22,627,285 | Novel mechanisms of early upper and lower urinary tract patterning regulated by RetY1015 docking tyrosine in mice. | Development | 2012 | https://conductscience.org/library/papers/22627285 | PubMed |
| 10 | 24,238,962 | Programmed cell senescence during mammalian embryonic development. | Cell | 2013 | https://conductscience.org/library/papers/24238962 | PubMed |
1 to 10 of 36
See all 36 referencesCited by4
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 4 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 41,923,047 | Zinner syndrome associated with concealed penis and azoospermia in an adolescent: a case report and literature review. | BMC Urol | 2026 | https://conductscience.org/library/papers/41923047 | PubMed |
| 39,174,793 | Multiple endocrine defects in adult-onset Sprouty1/2/4 triple knockout mice. | Sci Rep | 2024 | https://conductscience.org/library/papers/39174793 | PubMed |
| 38,670,941 | Sprouty1 is a broad mediator of cellular senescence. | Cell Death Dis | 2024 | https://conductscience.org/library/papers/38670941 | PubMed |
| 37,507,768 | Loss of SPRY2 contributes to cancer-associated fibroblasts activation and promotes breast cancer development. | Breast Cancer Res | 2023 | https://conductscience.org/library/papers/37507768 | PubMed |
Genes3
The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.
| Gene | NCBI Gene ID | Linked by | In Genes | Source |
|---|---|---|---|---|
| CBL | 867 | PubTator's text mining | https://conductscience.org/library/genes/867 | NCBI Gene |
| SPRY1 | 10,252 | PubTator's text mining | https://conductscience.org/library/genes/10252 | NCBI Gene |
| SPRY2 | 10,253 | PubTator's text mining | https://conductscience.org/library/genes/10253 | NCBI Gene |
Drugs0
The drugs whose trials, in the Pipelines release, list the paper.
None in this release.
Data0
The data bank entries PubMed's record names. Trial numbers are under Trials.
None in this release.
Data dates
The date of each source this release read.
- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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