Paper · 40520105
The matricellular protein ADAMTS-like 2 regulates differentiation of skeletal muscle-resident fibro-adipogenic progenitor cells.
Redhead C and 7 others. iScience, 2025. Cited by 5 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. The matricellular protein ADAMTS-like 2 regulates differentiation of skeletal muscle-resident fibro-adipogenic progenitor cells. Papers, record 40520105. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/40520105
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- The matricellular protein ADAMTS-like 2 regulates differentiation of skeletal muscle-resident fibro-adipogenic progenitor cells.
- PubMed
Topics0
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Grants0
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Trials0
The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.
None in this release.
Comments and corrections0
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None in this release.
References66
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 25,520,397 | A Wnt-TGFβ2 axis induces a fibrogenic program in muscle stem cells from dystrophic mice. | Sci Transl Med | 2014 | https://conductscience.org/library/papers/25520397 | PubMed |
| 2 | 33,959,042 | Evolving Roles of Muscle-Resident Fibro-Adipogenic Progenitors in Health, Regeneration, Neuromuscular Disorders, and Aging. | Front Physiol | 2021 | https://conductscience.org/library/papers/33959042 | PubMed |
| 3 | 37,001,514 | WNT7A suppresses adipogenesis of skeletal muscle mesenchymal stem cells and fatty infiltration through the alternative Wnt-Rho-YAP/TAZ signaling axis. | Stem Cell Reports | 2023 | https://conductscience.org/library/papers/37001514 | PubMed |
| 4 | 32,456,017 | Mechanisms Regulating Muscle Regeneration: Insights into the Interrelated and Time-Dependent Phases of Tissue Healing. | Cells | 2020 | https://conductscience.org/library/papers/32456017 | PubMed |
| 5 | 20,081,841 | Muscle injury activates resident fibro/adipogenic progenitors that facilitate myogenesis. | Nat Cell Biol | 2010 | https://conductscience.org/library/papers/20081841 | PubMed |
| 6 | 27,825,160 | Pathophysiology of Volumetric Muscle Loss Injury. | Cells Tissues Organs | 2016 | https://conductscience.org/library/papers/27825160 | PubMed |
| 7 | 30,463,013 | AMPK Activation Regulates LTBP4-Dependent TGF-β1 Secretion by Pro-inflammatory Macrophages and Controls Fibrosis in Duchenne Muscular Dystrophy. | Cell Rep | 2018 | https://conductscience.org/library/papers/30463013 | PubMed |
| 8 | 30,197,239 | Myomaker and Myomerger Work Independently to Control Distinct Steps of Membrane Remodeling during Myoblast Fusion. | Dev Cell | 2018 | https://conductscience.org/library/papers/30197239 | PubMed |
| 9 | 26,742,767 | Connective tissue cells expressing fibro/adipogenic progenitor markers increase under chronic damage: relevance in fibroblast-myofibroblast differentiation and skeletal muscle fibrosis. | Cell Tissue Res | 2016 | https://conductscience.org/library/papers/26742767 | PubMed |
| 10 | 25,981,413 | Fibrogenic Cell Plasticity Blunts Tissue Regeneration and Aggravates Muscular Dystrophy. | Stem Cell Reports | 2015 | https://conductscience.org/library/papers/25981413 | PubMed |
1 to 10 of 66
See all 66 referencesCited by4
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 5 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 42,681,291 | Phase-transition failure in aged skeletal muscle regeneration. | Biogerontology | 2026 | https://conductscience.org/library/papers/42681291 | PubMed |
| 42,332,008 | Losartan shows limited benefit in preclinical models of Geleophysic dysplasia. | Sci Rep | 2026 | https://conductscience.org/library/papers/42332008 | PubMed |
| 42,067,923 | Stimulation of EGFR signaling in fibro-adipogenic precursors decreases adipogenesis in Duchenne muscular dystrophy. | Skelet Muscle | 2026 | https://conductscience.org/library/papers/42067923 | PubMed |
| 41,864,337 | The Pathogenic ADAMTSL2 D167N Variant Causes Geleophysic Dysplasia-Like Connective Tissue Changes in Mice. | Am J Pathol | 2026 | https://conductscience.org/library/papers/41864337 | PubMed |
Genes3
The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.
| Gene | NCBI Gene ID | Linked by | In Genes | Source |
|---|---|---|---|---|
| ADAMTSL2 | 9,719 | PubTator's text mining | https://conductscience.org/library/genes/9719 | NCBI Gene |
| FAP | 2,191 | PubTator's text mining | https://conductscience.org/library/genes/2191 | NCBI Gene |
| TGFB1 | 7,040 | PubTator's text mining | https://conductscience.org/library/genes/7040 | NCBI Gene |
Drugs0
The drugs whose trials, in the Pipelines release, list the paper.
None in this release.
Data0
The data bank entries PubMed's record names. Trial numbers are under Trials.
None in this release.
Data dates
The date of each source this release read.
- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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