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Paper · 36980201

Cellular Senescence, a Novel Area of Investigation for Metastatic Diseases.

Faggioli F and 2 others. Cells, 2023. Cited by 10 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Cellular Senescence, a Novel Area of Investigation for Metastatic Diseases. Papers, record 36980201. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/36980201

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Cellular Senescence, a Novel Area of Investigation for Metastatic Diseases.
PubMed
Journal
Cells
PubMed
Year
2023
PubMed
Publication types
Journal Article, Review, Research Support, Non-U.S. Gov't
PubMed
Citations, by iCite
10
iCite
Relative citation ratio, by iCite
0.83
iCite
Revised in PubMed
2023-04-26
PubMed

Authors3

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Faggioli FFrancescahttps://orcid.org/0000-0001-8030-3674
2Velarde MCMichael Chttps://orcid.org/0000-0003-2384-5404
3Wiley CDChristopher D

Source PubMed

Topics3

NLM's MeSH indexing of the paper, its major topics first.

MeSH termMajor
Cellular SenescenceMajor topic
NeoplasmsMajor topic
Humans

Source PubMed

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References161

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
127,706,102The Redox Status of Cancer Cells Supports Mechanisms behind the Warburg Effect.Metabolites2016https://conductscience.org/library/papers/27706102PubMed
219,053,174Senescence-associated secretory phenotypes reveal cell-nonautonomous functions of oncogenic RAS and the p53 tumor suppressor.PLoS Biol2008https://conductscience.org/library/papers/19053174PubMed
328,827,372Monocarboxylate Transporter MCT1 Promotes Tumor Metastasis Independently of Its Activity as a Lactate Transporter.Cancer Res2017https://conductscience.org/library/papers/28827372PubMed
432,078,450Polyploidy in Cardiomyocytes: Roadblock to Heart Regeneration?Circ Res2020https://conductscience.org/library/papers/32078450PubMed
522,266,115Inhibition of fatty acid synthase attenuates CD44-associated signaling and reduces metastasis in colorectal cancer.Cancer Res2012https://conductscience.org/library/papers/22266115PubMed
624,232,458Ionizing radiation-induced responses in human cells with differing TP53 status.Int J Mol Sci2013https://conductscience.org/library/papers/24232458PubMed
715,657,080Stromal-epithelial interactions in aging and cancer: senescent fibroblasts alter epithelial cell differentiation.J Cell Sci2005https://conductscience.org/library/papers/15657080PubMed
825,035,397Osteopontin shapes immunosuppression in the metastatic niche.Cancer Res2014https://conductscience.org/library/papers/25035397PubMed
929,946,781The Intratumoral Heterogeneity of Cancer Metabolism.Adv Exp Med Biol2018https://conductscience.org/library/papers/29946781PubMed
1026,147,250MTOR regulates the pro-tumorigenic senescence-associated secretory phenotype by promoting IL1A translation.Nat Cell Biol2015https://conductscience.org/library/papers/26147250PubMed

Cited by8

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 10 citations.

PMIDTitleJournalYearIn PapersSource
42,811,753
4-Octyl Itaconate Attenuates Age-Induced Parotid Gland Dysfunction Through the Suppression of Fibrosis and Lipid Deposition by AMPKα/PPARα and the Inhibition of Oxidative Stress via the AMPKα/Nrf2 Signaling Pathway.
FASEB J2026https://conductscience.org/library/papers/42811753PubMed
42,089,920Integrated biomarker mapping reveals differential expression of senescence profiles in IDH-wild-type glioblastoma recurrent versus primary tumors.Virchows Arch2026https://conductscience.org/library/papers/42089920PubMed
41,142,786Identification and characterization of senescent macrophages in renal allograft rejection: a cross-species MultiOmics study.Front Immunol2025https://conductscience.org/library/papers/41142786PubMed
40,744,643Activation of a Src-JNK pathway in unscheduled endocycling cells of the Drosophila wing disc induces a chronic wounding response.Genetics2025https://conductscience.org/library/papers/40744643PubMed
40,434,135Circadian Gene BMAL1 Regulation of Cellular Senescence in Thyroid Aging.Aging Cell2025https://conductscience.org/library/papers/40434135PubMed
38,831,121SenNet recommendations for detecting senescent cells in different tissues.Nat Rev Mol Cell Biol2024https://conductscience.org/library/papers/38831121PubMed
38,139,316CDKN1A/p21 in Breast Cancer: Part of the Problem, or Part of the Solution?Int J Mol Sci2023https://conductscience.org/library/papers/38139316PubMed
37,157,887Spatial resolution of cellular senescence dynamics in human colorectal liver metastasis.Aging Cell2023https://conductscience.org/library/papers/37157887PubMed

Genes43

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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