Paper · 37465670
MDSCs in breast cancer: an important enabler of tumor progression and an emerging therapeutic target.
Liu H and 3 others. Front Immunol, 2023. Cited by 51 papers, by iCite.
11 sourcespapers-2026-11-12-0.1.0
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ConductScience Library. MDSCs in breast cancer: an important enabler of tumor progression and an emerging therapeutic target. Papers, record 37465670. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/37465670
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Paper
PubMed's record of the paper, with iCite's citation figures. No abstract is shown.
- Title
- MDSCs in breast cancer: an important enabler of tumor progression and an emerging therapeutic target.
- PubMed
Topics7
NLM's MeSH indexing of the paper, its major topics first.
| MeSH term | Major |
|---|---|
| Breast Neoplasms | Major topic |
| Myeloid-Derived Suppressor Cells | Major topic |
| Humans | |
| Female | |
| Immunotherapy | |
| Immune System | |
| Tumor Microenvironment |
Source PubMed
Grants0
Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.
None in this release.
Trials0
The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.
None in this release.
Comments and corrections0
PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.
None in this release.
References153
The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.
| Order | PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|---|
| 1 | 33,456,560 | IL20RA signaling enhances stemness and promotes the formation of an immunosuppressive microenvironment in breast cancer. | Theranostics | 2021 | https://conductscience.org/library/papers/33456560 | PubMed |
| 2 | 33,777,750 | The Crosstalk Between Tumor Cells and the Immune Microenvironment in Breast Cancer: Implications for Immunotherapy. | Front Oncol | 2021 | https://conductscience.org/library/papers/33777750 | PubMed |
| 3 | 34,506,976 | Engineered exosome-like nanovesicles suppress tumor growth by reprogramming tumor microenvironment and promoting tumor ferroptosis. | Acta Biomater | 2021 | https://conductscience.org/library/papers/34506976 | PubMed |
| 4 | 31,110,935 | Tumor derived EDIL3 modulates the expansion and osteoclastogenesis of myeloid derived suppressor cells in murine breast cancer model. | J Bone Oncol | 2019 | https://conductscience.org/library/papers/31110935 | PubMed |
| 5 | 34,551,052 | DTX@VTX NPs synergy PD-L1 immune checkpoint nanoinhibitor to reshape immunosuppressive tumor microenvironment for enhancing chemo-immunotherapy. | J Mater Chem B | 2021 | https://conductscience.org/library/papers/34551052 | PubMed |
| 6 | 33,010,818 | Olaparib Suppresses MDSC Recruitment via SDF1α/CXCR4 Axis to Improve the Anti-tumor Efficacy of CAR-T Cells on Breast Cancer in Mice. | Mol Ther | 2021 | https://conductscience.org/library/papers/33010818 | PubMed |
| 7 | 32,103,175 | Epigenetic therapy inhibits metastases by disrupting premetastatic niches. | Nature | 2020 | https://conductscience.org/library/papers/32103175 | PubMed |
| 8 | 32,213,179 | XIAOPI formula inhibits the pre-metastatic niche formation in breast cancer via suppressing TAMs/CXCL1 signaling. | Cell Commun Signal | 2020 | https://conductscience.org/library/papers/32213179 | PubMed |
| 9 | 32,554,250 | Chinese herbal medicine Baoyuan Jiedu decoction inhibits the accumulation of myeloid derived suppressor cells in pre-metastatic niche of lung via TGF-β/CCL9 pathway. | Biomed Pharmacother | 2020 | https://conductscience.org/library/papers/32554250 | PubMed |
| 10 | 25,595,776 | Cancer-associated oxidoreductase ERO1-α drives the production of tumor-promoting myeloid-derived suppressor cells via oxidative protein folding. | J Immunol | 2015 | https://conductscience.org/library/papers/25595776 | PubMed |
1 to 10 of 153
See all 153 referencesCited by40
The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 51 citations.
| PMID | Title | Journal | Year | In Papers | Source |
|---|---|---|---|---|---|
| 42,960,449 | Emerging role of non‑coding RNAs in the progression of MASLD‑driven hepatocellular carcinoma (Review). | Int J Mol Med | 2026 | https://conductscience.org/library/papers/42960449 | PubMed |
| 42,953,055 | Epithelial-mesenchymal transition plasticity in breast cancer: regulatory mechanisms and therapeutic implications. | Front Oncol | 2026 | https://conductscience.org/library/papers/42953055 | PubMed |
| 42,720,907 | MYBL2 promotes malignant phenotypes and M2-like macrophage polarization through CCL2 in non-small cell lung cancer. | Mol Cell Biochem | 2026 | https://conductscience.org/library/papers/42720907 | PubMed |
| 42,608,394 | Harnessing macrophage signaling pathways and scalable engineering for next-generation immunotherapies. | Signal Transduct Target Ther | 2026 | https://conductscience.org/library/papers/42608394 | PubMed |
| 42,558,596 | Cell competition within breast tumor microenvironment: emerging role of cancer stem cells and immune cells in tumor progression and therapeutic implications. | Front Oncol | 2026 | https://conductscience.org/library/papers/42558596 | PubMed |
| 42,454,051 | Wnt/β-Catenin-mTOR-autophagy crosstalk in breast cancer: context-dependent control of tumor progression, immune suppression, and therapeutic resistance. | Front Immunol | 2026 | https://conductscience.org/library/papers/42454051 | PubMed |
| 42,223,750 | Morpho-functional plasticity of tumour-associated macrophages: linking cell shape, ultrastructure, and immune regulation in cancer. | Clin Exp Med | 2026 | https://conductscience.org/library/papers/42223750 | PubMed |
| 42,143,109 | Pre-treatment of cabozantinib prior to hypofractionated radiotherapy increases immunomodulatory effects and tumor size reduction in a 4T1 breast cancer murine model. | Sci Rep | 2026 | https://conductscience.org/library/papers/42143109 | PubMed |
| 42,079,576 | B cell-mediated immune surveillance defines the favorable prognosis of occult breast cancer: a multi-omics study. | Front Immunol | 2026 | https://conductscience.org/library/papers/42079576 | PubMed |
| 42,035,063 | Breast cancer immunotherapy: mechanisms of immune evasion, biomarkers, and emerging therapeutic strategies. | Mol Cancer | 2026 | https://conductscience.org/library/papers/42035063 | PubMed |
1 to 10 of 40
See all 40 cited byGenes38
The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.
| Gene | NCBI Gene ID | Linked by | In Genes | Source |
|---|---|---|---|---|
| ADAM17 | 6,868 | PubTator's text mining | https://conductscience.org/library/genes/6868 | NCBI Gene |
| AKT1 | 207 | PubTator's text mining | https://conductscience.org/library/genes/207 | NCBI Gene |
| ARG1 | 383 | PubTator's text mining | https://conductscience.org/library/genes/383 | NCBI Gene |
| CAMP | 820 | PubTator's text mining | https://conductscience.org/library/genes/820 | NCBI Gene |
| CCR4 | 1,233 | PubTator's text mining | https://conductscience.org/library/genes/1233 | NCBI Gene |
| CD14 | 929 | PubTator's text mining | https://conductscience.org/library/genes/929 | NCBI Gene |
| CD4 | 920 | PubTator's text mining | https://conductscience.org/library/genes/920 | NCBI Gene |
| CSF1R | 1,436 | PubTator's text mining | https://conductscience.org/library/genes/1436 | NCBI Gene |
| CSF2 | 1,437 | PubTator's text mining | https://conductscience.org/library/genes/1437 | NCBI Gene |
| CSF3 | 1,440 | PubTator's text mining | https://conductscience.org/library/genes/1440 | NCBI Gene |
1 to 10 of 38
See all 38 genesDrugs0
The drugs whose trials, in the Pipelines release, list the paper.
None in this release.
Data0
The data bank entries PubMed's record names. Trial numbers are under Trials.
None in this release.
Data dates
The date of each source this release read.
- Trials
- 2026-11-12
- Gene search
- 2026-11-11
- Pipelines
- 2026-09-18
Sources 11
- ChEMBL, through Open TargetsCC BY-SA 3.0
- ClinicalTrials.govNo license stated
- HGNCCC0
- iCiteCC BY 4.0
- NCBI GenePublic domain (US government)
- NIH RePORTER's grant and paper linksPublic domain
- Open TargetsCC0 1.0
- PMC's ID filePublic domain (US government)
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0
Each source's terms
- ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- ClinicalTrials.govnone stated
- HGNCCC0: https://www.genenames.org/about/license/
- iCiteCC BY 4.0
- NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
- Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
- PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
- PubMedNLM's terms
- PubTator 3Public domain
- UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license
ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.
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