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Paper · 42199525

Cellular signaling crosstalk between osteoporosis and diabetes: Common mechanisms and therapeutic targets (Review).

Cheng Y and 6 others. Biomed Rep, 2026. Cited by 0 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Cellular signaling crosstalk between osteoporosis and diabetes: Common mechanisms and therapeutic targets (Review). Papers, record 42199525. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/42199525

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Cellular signaling crosstalk between osteoporosis and diabetes: Common mechanisms and therapeutic targets (Review).
PubMed
Journal
Biomed Rep
PubMed
Year
2026
PubMed
Publication types
Journal Article, Review
PubMed
Citations, by iCite
0
iCite
Revised in PubMed
2026-08-13
PubMed

Authors7

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Cheng YYangyi
2Gao JJingyuan
3Yang YYuyang
4Xing LLei
5Hou XXiaoli
6Zhang LLiu
7Tian FFaming

Source PubMed

Topics0

NLM's MeSH indexing of the paper, its major topics first.

None in this release.

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References256

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
140,552,618Rosmarinic Acid Ameliorates Type 2 Diabetic Osteoporosis by Reducing NLRP3 Expression and Alleviating Osteoblast Pyroptosis via the FOXO1/TXNIP Signaling Pathway.J Agric Food Chem2025https://conductscience.org/library/papers/40552618PubMed
232,845,528Sclerostin-Neutralizing Antibody Treatment Rescues Negative Effects of Rosiglitazone on Mouse Bone Parameters.J Bone Miner Res2021https://conductscience.org/library/papers/32845528PubMed
322,135,675Thiazolidinediones on PPARγ: The Roles in Bone Remodeling.PPAR Res2011https://conductscience.org/library/papers/22135675PubMed
441,597,184Studies of Foxo1 over the Past 25 Years: Mechanisms of Insulin Resistance and Glucose Dysregulation.Cells2026https://conductscience.org/library/papers/41597184PubMed
523,401,682Prevalence of Osteoporosis and Its Associated Factors among Older Men with Type 2 Diabetes.Int J Endocrinol2013https://conductscience.org/library/papers/23401682PubMed
638,311,970Piperlongumine, a Piper longum-derived amide alkaloid, protects mice from ovariectomy-induced osteoporosis by inhibiting osteoclastogenesis via suppression of p38 and JNK signaling.Food Funct2024https://conductscience.org/library/papers/38311970PubMed
737,871,879Intermittent fasting favorably modulates adipokines and potentially attenuates atherosclerosis.Biochem Pharmacol2023https://conductscience.org/library/papers/37871879PubMed
837,308,719Bavachinin selectively modulates PPAR γ and maintains bone homeostasis in Type 2 Diabetes.Phytother Res2023https://conductscience.org/library/papers/37308719PubMed
940,327,708Fibroblast activation protein-α interacts with CXCL12 to inactivate canonical Wnt signaling and regulate osteoblast differentiation.Stem Cells2025https://conductscience.org/library/papers/40327708PubMed
1039,760,914ALG5 downregulation inhibits osteogenesis and promotes adipogenesis by regulating the N-glycosylation of SLC6A9 in osteoporosis.Cell Mol Life Sci2025https://conductscience.org/library/papers/39760914PubMed

1 to 10 of 256

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Cited by0

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 0 citations.

None in this release.

Genes7

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

GeneNCBI Gene IDLinked byIn GenesSource
AGER177PubTator's text mininghttps://conductscience.org/library/genes/177NCBI Gene
CTNNB11,499PubTator's text mininghttps://conductscience.org/library/genes/1499NCBI Gene
GLP1R2,740PubTator's text mininghttps://conductscience.org/library/genes/2740NCBI Gene
INS3,630PubTator's text mininghttps://conductscience.org/library/genes/3630NCBI Gene
RENBP5,973PubTator's text mininghttps://conductscience.org/library/genes/5973NCBI Gene
TNFRSF11B4,982PubTator's text mininghttps://conductscience.org/library/genes/4982NCBI Gene
TNFSF118,600PubTator's text mininghttps://conductscience.org/library/genes/8600NCBI Gene

Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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