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Paper · 42643637

Dioscin in liver diseases: pharmacological mechanisms, translational potential and future perspectives.

Liu J and 3 others. Front Pharmacol, 2026. Cited by 0 papers, by iCite.

11 sourcespapers-2026-11-12-0.1.0

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ConductScience Library. Dioscin in liver diseases: pharmacological mechanisms, translational potential and future perspectives. Papers, record 42643637. Release papers-2026-11-12-0.1.0, 12 Nov 2026. https://conductscience.org/library/papers/42643637

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Paper

PubMed's record of the paper, with iCite's citation figures. No abstract is shown.

Title
Dioscin in liver diseases: pharmacological mechanisms, translational potential and future perspectives.
PubMed
Journal
Front Pharmacol
PubMed
Year
2026
PubMed
Publication types
Journal Article, Review
PubMed
Citations, by iCite
0
iCite
Revised in PubMed
2026-08-27
PubMed

Authors4

The authors in the order PubMed's record lists them, with the ORCID iDs it states.

OrderAuthorForenameKindORCID iD
1Liu JJia
2Sun RRulan
3Huang JJie
4Wang CCheng

Source PubMed

Topics0

NLM's MeSH indexing of the paper, its major topics first.

None in this release.

Grants0

Each grant PubMed's record writes, matched to the NIH project NIH's grant and paper links name for this paper, and each of those links the record does not write.

None in this release.

Trials0

The trials whose ClinicalTrials.gov record lists the paper, and the trial numbers the paper's PubMed record names.

None in this release.

Comments and corrections0

PubMed's comments and corrections, NLM's own indexing: retractions first, then expressions of concern, errata and updates, and comments last.

None in this release.

References110

The papers the reference list in PubMed's record names, those in our copy of PubMed, in the list's order.

OrderPMIDTitleJournalYearIn PapersSource
129,574,225Dioscin alleviates non-alcoholic fatty liver disease through adjusting lipid metabolism via SIRT1/AMPK signaling pathway.Pharmacol Res2018https://conductscience.org/library/papers/29574225PubMed
228,337,145Dioscin Protects ANIT-Induced Intrahepatic Cholestasis Through Regulating Transporters, Apoptosis and Oxidative Stress.Front Pharmacol2017https://conductscience.org/library/papers/28337145PubMed
324,012,730Liquid chromatography tandem mass spectrometry in study of the pharmacokinetics of six steroidal saponins in rats.Steroids2013https://conductscience.org/library/papers/24012730PubMed
438,299,199Mitochondrial dysfunction: A promising therapeutic target for liver diseases.Genes Dis2024https://conductscience.org/library/papers/38299199PubMed
519,513,766Hemolytic mechanism of dioscin proposed by molecular dynamics simulations.J Mol Model2010https://conductscience.org/library/papers/19513766PubMed
639,749,193Apigenin as an emerging hepatoprotective agent: current status and future perspectives.Front Pharmacol2024https://conductscience.org/library/papers/39749193PubMed
739,342,780Dioscin improves fatty liver hemorrhagic syndrome by promoting ERα-AMPK mediated mitophagy in laying hens.Phytomedicine2024https://conductscience.org/library/papers/39342780PubMed
839,996,739Mechanosignaling via Integrins: Pivotal Players in Liver Fibrosis Progression and Therapy.Cells2025https://conductscience.org/library/papers/39996739PubMed
925,853,178Potent effects of dioscin against liver fibrosis.Sci Rep2015https://conductscience.org/library/papers/25853178PubMed
1024,385,684The effect of inflammatory cytokines in alcoholic liver disease.Mediators Inflamm2013https://conductscience.org/library/papers/24385684PubMed

Cited by1

The papers in our copy of PubMed whose reference list names this one, newest first. iCite, which reads more sources, counts 0 citations.

PMIDTitleJournalYearIn Papers
42,796,621Saponins Derived from Chinese Medicinal Materials in Hyperuricemia and Gout: Therapeutic Mechanisms and Emerging Perspectives.Molecules2026https://conductscience.org/library/papers/42796621

Source PubMed

Genes12

The genes the Gene search release ties to the paper: Swiss-Prot's references first, then PubTator's text mining.

GeneNCBI Gene IDLinked byIn GenesSource
AKT1207PubTator's text mininghttps://conductscience.org/library/genes/207NCBI Gene
CTNNB11,499PubTator's text mininghttps://conductscience.org/library/genes/1499NCBI Gene
GPX42,879PubTator's text mininghttps://conductscience.org/library/genes/2879NCBI Gene
KEAP19,817PubTator's text mininghttps://conductscience.org/library/genes/9817NCBI Gene
MTOR2,475PubTator's text mininghttps://conductscience.org/library/genes/2475NCBI Gene
MYD884,615PubTator's text mininghttps://conductscience.org/library/genes/4615NCBI Gene
NFE2L24,780PubTator's text mininghttps://conductscience.org/library/genes/4780NCBI Gene
NFKB14,790PubTator's text mininghttps://conductscience.org/library/genes/4790NCBI Gene
PIK3CB5,291PubTator's text mininghttps://conductscience.org/library/genes/5291NCBI Gene
SLC7A1123,657PubTator's text mininghttps://conductscience.org/library/genes/23657NCBI Gene

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Drugs0

The drugs whose trials, in the Pipelines release, list the paper.

None in this release.

Data0

The data bank entries PubMed's record names. Trial numbers are under Trials.

None in this release.

Data dates

The date of each source this release read.

PubMed
2026-11-06
PubMed
iCite
2026-11-05
iCite
PMC's ID file
2026-11-05
PMC's ID file
NIH RePORTER's grant and paper links
2026-11-01
NIH RePORTER's grant and paper links
Trials
2026-11-12
Gene search
2026-11-11
Pipelines
2026-09-18

Sources 11

  • ChEMBL, through Open TargetsCC BY-SA 3.0
  • ClinicalTrials.govNo license stated
  • HGNCCC0
  • iCiteCC BY 4.0
  • NCBI GenePublic domain (US government)
  • NIH RePORTER's grant and paper linksPublic domain
  • Open TargetsCC0 1.0
  • PMC's ID filePublic domain (US government)
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0
Each source's terms
  • ChEMBL, through Open TargetsCC BY-SA 3.0, for the parts from ChEMBL. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • ClinicalTrials.govnone stated
  • HGNCCC0: https://www.genenames.org/about/license/
  • iCiteCC BY 4.0
  • NCBI GeneNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • NIH RePORTER's grant and paper linksPublic domain (NIH RePORTER)
  • Open TargetsCC0 1.0. Open Targets asks that users cite its latest paper: Buniello et al. 2025, Open Targets Platform: facilitating therapeutic hypotheses building in drug discovery, Nucleic Acids Research 53(D1):D1467, PMID 39657122. Read on 8 Nov 2026 at https://platform-docs.opentargets.org/licence
  • PMC's ID fileNone named for the file. NCBI says information created by or for the US government on its site is in the public domain, and asks that NLM be acknowledged: https://www.ncbi.nlm.nih.gov/home/about/policies/
  • PubMedNLM's terms
  • PubTator 3Public domain
  • UniProtCC BY 4.0. UniProt's licence file: "All databases and documents in the UniProt FTP directory may be copied and redistributed freely, without advance permission, provided that this copyright statement is reproduced with each copy." UniProt asks that users cite its latest paper: The UniProt Consortium, UniProt: the Universal Protein Knowledgebase in 2025, Nucleic Acids Research 53:D609-D617, https://doi.org/10.1093/nar/gkae1010. Read on 9 Nov 2026 at https://www.uniprot.org/help/license

ClinicalTrials.gov's data carry an international copyright outside the United States: https://clinicaltrials.gov/about-site/terms-conditions.

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